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PubMed1992

[Histogenesis of isthmic nodular salpingitis].

Nodular salpingitis of the isthmus is a clinicopathological entity related to what is classically referred to as interstitial adenomyosis. These two lesions are due to the same aetiopathogenic process and result from inflammatory proliferation of the tubal epithelium with formation of pseudoglandular ducts. The only difference between the two is that one is ensheathed by a fine network of mesonephric muscle fibres and the other is surrounded by a thick layer of myometrial muscle fibres. These two lesions should be grouped under the same heading: nodular salpingitis. The term adenomyosis is incorrect due to the absence of endometrial glandular ducts and stroma in these lesions. Nodular salpingitis of the isthmus should be distinguished from tubal endometriosis, which is part of the clinical expression of extrauterine endometriosis. In particular, this lesion must be distinguished from interstitial endometriosis which, for some authors, corresponds to colonisation of the tubal mucosa by endometrium and, for others, to metaplasia of the tubal mucosa.

Diagnosis, Differential↗
PubMed1985

[Celioscopy in the early follow-up of salpingitis].

The authors report their findings in 34 cases of salpingitis that were diagnosed and controlled laparoscopically one month after treatment had been finished. Four different elements came up: persistence of inflammation, adhesions, tubal patency and the prognosis for future fertility. It would seem that these last three criteria depend greatly on the seriousness at first of the salpingitis, as opposed to the degree of persistent inflammation. The authors show how valuable laparoscopic drainage of a pyosalpinx is and how valuable laparoscopic control after salpingitis is. Having been given these features they can visualise a new approach to the diagnosis and treatment of pelvic infection.

Adolescent↗
PubMed1987

Acute salpingitis in sterilized women.

Previously sterilized women are generally considered protected against salpingitis. However, when we diagnosed salpingitis by laparoscopy in 48 patients during a 16-month period, we found four (8%) who had been previously sterilized. Therefore, contrary to generally stated belief, salpingitis may occur after interruption of tubal continuity.

Acute Disease↗
PubMed1987

[Diagnostic value of the study of anti-Chlamidia antibodies in salpingitis. 379 cases].

Antibodies to Chlamydia were assayed by complement fixation (CF) and inclusion indirect immunofluorescence (IFI) in sera collected from 379 patients with salpingitis: 30.3% of the patients had total and IgM antibodies at IFI and CF antibodies (profile I); 26.6% of the patients had total and IgM antibodies at IFI without CF antibodies (profile II); 31.6% of the patients had only total antibodies at IFI without specific IgM and without CF antibodies (profile III); 11.3% of the patients were Chlamydia antibody negative (profile IV). In the control group of 50 pregnant women apparently non infected, the profile distribution was 2% profile I, 8% profile II, 38% profile III, and 54% profile IV. Detection of IgM antibodies to Chlamydia trachomatis in 57% of patients with salpingitis, taking only one specimen, suggested recent or active chlamydial infection. CF antibodies indicated diffuse infection. Total antibodies correlated well with IgG antibodies detected by ELISA. Their finding was by no means diagnosis for Chlamydia being the cause of tubal infection, although titers observed in salpingitis patients were higher than in controls.

Adolescent↗
PubMed1988

[Treatment of acute non-chlamydial salpingitis. Study of the efficacy and tolerance of a single-therapy antibiotic: Augmentin].

An open randomised comparative trial of the efficacy and safety of Augmentin as against the triple therapy of penicillin-gentamicin-metronidazole in acute salpingitis was conducted in forty women admitted to hospital. Laparoscopy was performed routinely to confirm the diagnosis. The two groups of patients were comparable as to age and clinical and biological symptoms and the severity of the salpingitis (grade I to IV, with the presence or absence of Fitz-Hugh-Curtis syndrome). Treatment was started immediately after the laparoscopy, first by parental route until the patient had been apyrexic for 48 hours. Oral follow-up was then commenced. Twenty women received Augmentin and twenty the triple antibiotic therapy. Specimens for bacteriological study were obtained before treatment (culture of the urine, culture from the IUD and from the cervix, and swabs were taken laparoscopically). This made it possible to identify aerobic and anaerobic organisms. (The gonococcus was found more often in the group treated with triple antibiotics). On discharge, cures had been obtained in 12 women and 6 more were responding out of the Augmentin group. Out of the triple therapy group 8 were cured and 10 were responding. There was one failure in each group (persisting fever). Long-term assessment was carried out in the out-patients three weeks after discharge. 11 out of the 13 reviewed in the Augmentin group and 8 out of the 14 in the triple therapy group were considered as definitely cured. The clinical safety of both treatments was good. These results demonstrate that Augmentin is as effective as the combined therapy in treating acute salpingitis and with the added advantage of its easy use and lower cost.

Acute Disease↗
PubMed1986

Salpingitis in mice induced by human strains of Chlamydia trachomatis.

Human strains of Chlamydia trachomatis were inoculated unilaterally into the genital tracts of female TO, CBA, CBA/nu and C3H mice via the intrauterine route or under the ovarian bursa. Inflammatory changes were not seen in the oviducts or uterus of mice given two laboratory-adapted LGV serovars (L1 and L2), although chlamydiae were recovered from the lower genital tract. However, salpingitis and endometritis occurred after each of three chlamydial strains (serovars D and E) had been inoculated. Oviduct inflammation was seen for up to 6 weeks after inoculation but reached maximum severity usually after about 2 weeks, the lumen sometimes being occluded by exudate and necrotic debris. Pathological changes were seen often in both oviducts indicating canalicular spread of the organisms through the uterus. Pre-treatment of the mice with progesterone had an enhancing effect in that the lesions developed more rapidly; such treatment, in halting the oestrous cycle, probably made a larger number of target cells available for more efficient infection. Involvement of the oviduct on the uninoculated side occurred more rapidly in T-cell impaired nude mice than in immunologically normal mice, although there was little or no effect on the severity of the oviductal changes. There was evidence that the susceptibility of different strains of mice to chlamydial salpingitis varied. Thus, inflammatory changes in C3H mice were more severe than in TO mice and the changes in C3H and CBA strains were longer lasting than those in TO mice. This suggests that a possible genetic predisposition in the human situation should not be ignored. Finally, one chlamydial strain of low passage produced more severe salpingitis in mice than another strain of similar passage. By analogy different chlamydial strains may not be of equal pathogenicity in the human situation.

Animals↗
PubMed1979

Nodular salpingitis and tubal endometriosis. I. Comparative clinical study.

Nodular salpingitis and tubal endometriosis have been referred to in succession as salpingiosis, diverticulosis, adenomyosis of the oviduct, endometrioid conditions, etc. This varied terminology underscores their etiopathogenetic and morphological substratum which is different from that of non specific tubal inflammation, but at the same time this variety of terms has always created confusion in interpretation and diagnosis. We have considered it necessary to carry out a comparative study of nodular salpingitis and tubal endometriosis in 42 cases of sterility operated during the last two years for tubal obstruction and in which histological examination has yielded evidence for the lesions of nodular salpingitis (NS) or endometriosis (EM) in at least one of the oviducts. As for the etiology of the two diseases, we have discussed the role plaid by inflammatory conditions, uterine trauma (curettage) and dystrophic disorders, as well as the importance of hyperandrogenism in NS. Taking into account the diffuse sclerogenic tendency of the tubal wall in NS and the concomitant inflammatory and dystrophic lesions in the peritubal tissues in EM, the postoperative outlook depends on early surgery, to be performed before tubal anatomy has been completely altered.

Adult↗
PubMed1984

The development of infections of the genitourinary tract in the wives of infertile males and the possible role of spermatozoa in the development of salpingitis.

Genitourinary tract infections developing in wives during marital life in 1,350 infertile couples were examined. Women married to men with a history of genital tract infection had a significantly higher incidence of vaginitis, urinary tract infection, salpingitis and genital herpes when compared with women whose husbands did not have genital tract infections before the marriage. Of the variables examined, the two factors that showed the most significant association with the tendency to develop salpingitis were the sperm count and the length of time the couple had been trying to achieve a pregnancy. The wives of azoospermic males did not have pelvic inflammatory disease develop but had the same incidence of infection of the lower part of the genital tract as the other studied groups. It is suggested that the bacterial flora of the seminal fluid can play a role in developing salpingitis in the female and that spermatozoa may be involved in delivering bacteria to the higher genital tract structures.

Adult↗
PubMed1984

[Ciliary activity of cells of the fallopian tubes (apropos of the sequellae of salpingitis)].

A study was made of ciliary movement in tubal epithelium using microphotooscillography on the tubes of 91 patients who were operated on for tubal, ovarian or uterine pathology. Three groups were classified: 31 with healthy tubes, 47 with tubes that showed the consequences of salpingitis. These patients had been operated on for sterility after much treatment with antibiotics and corticoids. 13 sets of tubes from pregnant women (6 cases had intra-uterine pregnancies and underwent tubal ligation after Caesarean section or after a termination of pregnancy) and 7 cases had extra-uterine pregnancies with salpingectomy. This study has made it possible for us to make several observations: The ciliary activity was not altered by the time in the menstrual cycle. When the tubes were healthy tubal activity was hardly changed when a fibroid was present, nor when an ovarian cyst or endometriosis were present (550 beats a minute). In pregnant women ciliary activity is maintained until the pregnancy becomes intra-uterine. It is always abolished in cases of extra-uterine pregnancy, even if this is at some distance from the site of inflammatory reaction caused by implantation of the oocyte. In cases following salpingitis, ciliary activity in the tubes is often altered and sometimes nil. It therefore seems that if one excludes anatomical lesions such as tubal blocks and ampullary phimosis, salpingitis which is the cause of sterility changes tubal physiology greatly. This is true even at some distance from the site of the infective process as is shown by changes in ciliary activity.

Cilia↗
PubMedDate not supplied

Microscopic salpingitis is not an etiologic factor of tubal pregnancy with intrauterine devices.

OBJECTIVE: This prospective study was undertaken to test the hypothesis that microscopic chronic salpingitis is an etiologic factor in ectopic (tubal) pregnancy with an intrauterine device (IUD). PATIENTS AND METHODS: Fifty consecutive patients at a university hospital operated for tubal pregnancy fulfilled strict histological diagnostic criteria for tubal pregnancy. There were no statistically significant differences in prevalence of microscopic findings of chronic inflammation in patients who never used an IUD, had a history of IUD use, or had an IUD in situ at the time of laparotomy. Salpingitis isthmica nodosa was found in four patients (30.8%) without past or present history of IUD use as compared to two patients (5.4%) with past or present history of IUD (P < .05). CONCLUSION: The use of an IUD does not appear to cause tubal pregnancy by the mechanism of tubal inflammation or presence of salpingitis isthmica nodosa. Therefore, other mechanisms by which an IUD might cause ectopic pregnancy should be considered.

Adult↗
PubMed1993

Salpingitis or oophoritis: what causes fever following oocyte aspiration and embryo transfer?

BACKGROUND: Febrile morbidity following in vitro fertilization and embryo transfer (IVF-ET) is a rare but possibly serious complication. This report describes a case of salpingitis after IVF-ET and discusses the possible reasons for febrile morbidity following this common procedure. CASE: A 37-year-old woman undergoing IVF-ET for tubal factor infertility developed sudden, severe pelvic pain, fever, and leukocytosis 24 hours after ET. Laparoscopy revealed bilateral suppurative pyosalpinges with cystic, hemorrhagic ovaries. Pain, fever, and leukocytosis resolved with conservative surgery and intravenous antibiotic therapy. CONCLUSIONS: This case presents laparoscopic documentation of a rare complication of oocyte aspiration and/or ET, namely, salpingitis. Possibilities for the development of salpingitis following IVF-ET include activation of quiescent bacteria within the fallopian tubes from a previous pelvic infection, puncture of the bowel during oocyte aspiration, inoculation of the pelvis with cervicovaginal flora during oocyte aspiration, and introduction of bacteria-laden secretions or air into the fallopian tubes during ET. Although rare, the possibility of severe pelvic infection following IVF-ET warrants consideration of prophylactic antibiotic coverage.

Adult↗
PubMed1977

Morbidity of appendectomy in patients with acute salpingitis.

Diagnosis of right lower quadrant pain in a woman is frequently confusing. The course of 53 patients who had operation with a normal appendix and no other intra-abdominal pathology is compared with a group of 50 patients with a normal appendix and acute salpingitis. Appendectomies were performed in all cases and there was a lower mortality and morbidity rate in the group of patients with salpingitis. It is, therefore, concluded that appendectomy is a safe procedure in patients with acute salpingitis.

Acute Disease↗
PubMed1995

[Salpingitis and tubal pregnancy in materials from the Clinics of the Surgical Gynecological Academy of Medicine in Lublin in the years 1978-1988].

This retrospective study was based on hospital records analysis. The incidence of tubal pregnancy and salpingitis were evaluated and possible correlation between these two diseases was estimated. Despite of many other authors reports about increase of tubal pregnancy incidence we observed decline of tubal pregnancy incidence in studied period. Increase of salpingitis incidence did not alter this trend. Authors conclude that recently reported increase of tubal pregnancy incidence in many countries is valid only in populations studied and can not be generalized. The increase of salpingitis incidence does not necessarily influence tubal pregnancy incidence.

Female↗
PubMed2005

Virulence characteristics of Escherichia coli isolates obtained from broiler breeders with salpingitis.

Thirty isolates of Escherichia coli from broiler breeders with salpingitis were studied. Using the slide agglutination test, the isolates were found to belong to serogroups O1, O2, O5, O36, O45, O53 and O78. Pathogenicity for day-old chicks was determined by air sac inoculation and isolates were categorized as having high, intermediate or low virulence. Growth on iron starvation medium was observed together with aerobactin production. Based on the results of in vitro adherence tests, attachment to oviduct epithelium from old birds was found to be superior to that observed using corresponding material from young birds. DNA hybridization testing for type 1, P, and S fimbriae revealed predominant expression of type 1, correlating with mannose-sensitive hemagglutination using guinea-pig erythrocytes. In this study, P and S fimbriae were not considered to be important adherence factors. Study findings would suggest that, as far as salpingitis is concerned, type 1 fimbriae can play an important role in E. coli infection in breeders. An interesting result to emerge from the study was the observation that E. coli isolates were completely resistant to serum from young breeders, whereas they were completely sensitive using serum from older breeders. Based on serogroups involved, pathogenicity for day-old chicks and virulence indicators, the salpingitis isolates were similar to those from cases of chronic respiratory disease.

Animals↗
PubMed1985

Type II collagen induced autoimmune salpingitis in the rat.

Previous studies showed type II collagen induced autoimmune salpingitis in Wistar rats. In this study we increased the number of animals and investigated whether or not type II collagen induced salpingitis is transferable by immune sera. Histopathological observations of salpingitis in the serum transferred rats were similar to those observed in the immunized rats but the degree of otopathological changes was less than that of immunized rats. Electronmicroscopic findings indicate an increased permeability in the capillaries and intercellular space and cellular infiltration in the submucosa of the immunized rats.

Animals↗
PubMed1987

Significance of antibodies to Mycoplasma genitalium in salpingitis.

Indirect haemagglutination and indirect immunofluorescence tests of sera from 95 patients with acute salpingitis failed to confirm the report of other investigators regarding serological evidence implicating Mycoplasma genitalium in pelvic inflammatory disease. The pathological role of Mycoplasma genitalium in salpingitis, non-gonococcal urethritis, and other diseases has not been proven.

Acute Disease↗
PubMed1985

Choice of antibiotics and length of therapy in the treatment of acute salpingitis.

This article reviews the rationale for the therapy of acute salpingitis and the conceptual basis for the length of therapy. The key to therapy of acute salpingitis is the need to accommodate polymicrobial etiology, polymicrobial bacterial superinfection, and the potential presence of penicillinase-producing strains of Neisseria gonorrhoeae into a therapeutic equation that has been determined by the appropriate staging of disease. The anticipated therapeutic response identified for monomicrobial disease due to Neisseria gonorrhoeae constitutes the end titration point for drug administration. Duration of continued therapy beyond this point is governed by the need to complete therapy for Chlamydia trachomatis or to assure resolution of advanced disease.

Acute Disease↗
PubMed1976

Amylases of the genital tract. II. Peritoneal fluid isoamylases in acute salpingitis.

The activities of the specific genital isoamylases in peritoneal fluid were diminished or absent in acute salpingitis. The decrease was proportional to the severity of the tubal inflammatory reactions but could not be related to a closure of the abdominal tubal ostia. In women with infections of the lower genital tract in whom the Fallopian tubes were normal at laparoscopy, the peritoneal fluid isoamylase activities were as high as in healthy women. Determination of the activities of specific genital isoamylases in peritoneal fluid obtained by cul-de-sac puncture would seem to constitute a specific diagnostic method for acute salpingitis.

Acute Disease↗