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Results for “Consciousness Disorders”

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At least 109 records · Page 6Linked to original sources
PubMed1989

[Hyperkinetic mutism within the scope of consciousness disorder in a case of systemic lupus erythematosus].

A thirty-year-old housewife, who had been diagnosed as SLE, had been hyperkinetic and mute for 13 months after reiteration of manic-depressive episodes. In this condition she walked around without aim, ate and drank only with nursing help, and could not understand the circumstances around her; she was incontinent of urine and feces. Serological examination did not show any abnormal results during psychic exacerbation. She recovered dramatically by electroconvulsive therapy, and was completely amnestic about the past 15 months. Because her mental state was considered to be limited consciousness, it might be possible that her hyperkinetic and mute state is situated between twilight state and akinetic mutism.

Adult↗
PubMed1991

[Twilight consciousness disorder caused by overdose of indomethacin].

A case pertaining to legal psychiatric practice is described: the development of dusk clouding of consciousness in a patient with chronic alcoholism after the intake of indomethacin in high doses, foregoing insomnia and episodic alcohol abuse. Possible casuistry of the case forms no basis for ignoring the fact that in legal psychiatry, analogous episodes because of the intake of the high doses of certain drugs including indomethacin may be repeated.

Back Pain↗
PubMedDate not supplied

[Paradoxical vestibular reaction in patients with consciousness disorders (nystagmographic recording)].

In 32 patients with coma of varying intensity and aetiology the effect of light on caloric nystagmus was studied. During consciousness disturbances a light stimulus provoked or increased the intensity of induced caloric nystagmus. This reaction was thus a reverse (paradoxical reaction) of that observed in waking state. Photic stimulation may provoke the rapid phase of nystagmus through facilitating impulses coursing from the subcortical or cortical visual centres to Cajal's nuclei and then through the reticular formation to the vestibulo-oculomotor system. In waking state these impulses are inhibited while in precomatic state they may become prevalent. The paradoxical reaction disappears in deep coma together with all vestibular reactions.

Cognition Disorders↗
PubMed1996

[Severe metabolic alkalosis with a consciousness disorder].

HISTORY AND CLINICAL FINDINGS: A 47-year-old man in a reduced general condition, presumed to be a chronic alcoholic, was hospitalised in a sleepy state and impaired level of consciousness (Glasgow Coma Scale 8). There were no focal neurological deficits, but all proprioceptor reflexes were weak. Body temperature was 36.8 degrees C, blood pressure 90/60 mm Hg, and heart rate 80/min. INVESTIGATIONS: Biochemical tests showed sodium concentration reduced to 121 mmol/l, potassium to 1.83 mmol/l, chloride to 55 mmol/l and, on the next day, phosphate to 0.11 mmol/l. Blood gas analysis demonstrated a noncompensated respiratory alkalosis (pH 7.69, bicarbonate 39.5 mmol/l and a base excess of 20 mmol/l. TREATMENT AND COURSE: The impaired consciousness was thought to be due to the marked alkalosis in combination with hypophosphataemia. The alkalosis was completely removed within 48 hours by administration of Ringer's solution and potassium chloride concentrate, without sodium chloride Phosphate deficit was neutralised with KH2PO4 infusion. Normal consciousness was restored. CONCLUSIONS: Even severe hypochloraemic alkalosis can be quickly reversed with infusion of chloride without sodium Successful treatment with chloride alone excludes alkalosis induced by mineralocorticoids.

Acute Disease↗
PubMed1994

[Differential diagnosis of acute consciousness disorders].

The most important causes of syncope and coma are discussed. Syncope may be due to cardiac, vascular or cerebral disease. A cause of syncope is not established in up to 50% of cases. Where a diagnosis is possible the patient's history, physical examination, ECG and prolonged ECG monitoring serve to establish the underlying disease in most cases. Additional diagnostic tests should only be performed in patients with possible cardiac syncope, since these cases show a higher mortality rate than patients with non-cardiac syncope. Coma may be caused by metabolic disorders, intoxication or cerebral diseases, which are easily identified by history, physical examination and simple laboratory tests in most cases. Further evaluation of patients with unknown cause of coma depends on whether focal neurologic signs, meningeal irritation and fever are present.

Acute Disease↗