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Results for “Color Vision Defects”

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At least 109 records · Page 6Linked to original sources
PubMed1995

Association of acquired color vision defects in blue cone monochromatism.

The retinal functions of a 43-year-old man with diabetic retinopathy in a family affected by blue cone monochromatism were studied. While changes in visual acuity and color vision were similar to those seen in patients affected by rod monochromatism, and his spectral sensitivities on high intensity background and his threshold versus intensity curve indicated only suggestive blue cone function, mass electroretinograms (ERG) findings demonstrated well preserved blue cone function. The disparity in blue cone function between psychophysical and mass electroretinographical evaluation may be caused by the difference in how much the fovea influenced each testing. Diabetic retinopathy most likely caused significant reduction of the psychophysical blue cone function and resulted in clinical findings similar to rod monochromatism. The mass blue cone ERG was useful to differentiate blue cone monochromatism from rod monochromatism in this diabetic patient.

Adult↗
PubMedDate not supplied

Multidimensional scaling of D15 caps: color-vision defects among tobacco smokers?

Tobacco smoke contains a range of toxins including carbon monoxide and cyanide. With specialized cells and high metabolic demands, the optic nerve and retina are vulnerable to toxic exposure. We examined the possible effects of smoking on color vision: specifically, whether smokers perceive a different pattern of suprathreshold color dissimilarities from nonsmokers. It is already known that smokers differ in threshold color discrimination, with elevated scores on the Roth 28-Hue Desaturated panel test. Groups of smokers and nonsmokers, matched for sex and age, followed a triadic procedure to compare dissimilarities among 32 pigmented stimuli (the caps of the saturated and desaturated versions of the D15 panel test). Multidimensional scaling was applied to quantify individual variations in the salience of the axes of color space. Despite the briefness, simplicity, and "low-tech" nature of the procedure, subtle but statistically significant differences did emerge: on average the smoking group were significantly less sensitive to red-green differences. This is consistent with some form of injury to the optic nerve.

Color Perception↗
PubMed1990

[Color vision defects in chronic open angle glaucoma].

Glaucomatous optic nerve atrophy is associated with morphological and psychophysical changes. Using Roth's Besancon anomalometer, the Farnsworth 100 hue test and Nagel's anomaloscope, we examined color vision in 86 eyes of 51 patients suffering from chronic open-angle glaucoma and 57 eyes of 41 normal subjects. In the normal control group, blue und green sensitivity decreased and, accordingly, the anomaly quotient tested with Nagel's anomaloscope increased significantly (p less than 0.00001) with age. If the glaucoma and control groups were matched for age, refractive error and central visual acuity, decreasing blue sensitivity significantly (p less than 0.05) correlated with diminished visibility of the retinal nerve fiber bundles, a higher morphological glaucoma stage and larger perimetric defects. The presence and depth of localized defects of the retinal nerve fiber layer were not significantly different in glaucoma subgroups with lower and higher blue sensitivity, respectively, when the subgroups were matched for age, refractive error and visual acuity. No papillomorphologic marker for the cyanodyschromatopsia was detected. Red-green color vision was not significantly different between the normal and glaucoma eyes. Testing of blue color vision as an additional method is useful in the differential diagnosis of beginning glaucomatous optic nerve damage in patients with clear optic media and lack of macular changes.

Adult↗