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At least 109 records · Page 6Linked to original sources
PubMed2006

Parkin blushed by PINK1.

Mutations in the PTEN-induced putative kinase 1 (PINK1) are a common cause of autosomal recessive Parkinson's disease. In a recent issue of Nature, two independent reports by and show that loss of Drosophila PINK1 leads to defects in mitochondrial function resulting in male sterility, apoptotic muscle degeneration, and minor loss of dopamine neurons that is rescued by overexpression of the ubiquitin E3 ligase, parkin. Thus, PINK1 and parkin appear to function in a common pathway suggesting a convergence of the two genes most commonly associated with autosomal recessive PD.

Animals↗
PubMed1990

At first blush.

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Antigens, Tumor-Associated, Carbohydrate↗
PubMed2005

Why blue blushed.

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Brain Abscess↗
PubMed1990

At first blush.

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Arousal↗
PubMed1998

Uterine blush mimicking a hypervascular tumour in the pelvis as demonstrated by a triple-phase bone scan.

Transient visualization of the uterus, mimicking a hypervascular soft tissue tumour in the pelvic region, in the blood flow and blood pool phases of a triple-phase bone scan, was seen in 78 of 91 females of reproductive age. It was noted in all cases of menstrual (n = 15), early proliferative (n = 9) and secretory (n = 42) phases of the cycle, but in only 12 of 25 subjects studied in the late proliferative phase. This observation differs from previous reports of uterine uptake only being seen in the menstrual and secretory phases, or in all menstrual cycle phases. None of the 58 control patients (20 males, 16 pre-menarcheal females and 22 post-menopausal females) had such uterine uptake in either the blood flow or blood pool phase in the pelvic region. We suggest that this normal physiological phenomenon, mimicking a hypervascular soft tissue tumour in the pelvis, occurs in the menstrual, early proliferative and secretory phases of pre-menopausal females and in all phases of the cycle in younger females of reproductive age.

Adolescent↗
PubMedDate not supplied

Patients with erythrophobia (fear of blushing) show abnormal autonomic regulation in mental stress conditions.

OBJECTIVE: The objective of this study was to analyze the autonomic functions of patients with erythrophobia. METHODS: Forty patients with a diagnosis of erythrophobia (female/male ratio 18/22) without any other organic lesions and 20 healthy volunteers (female/male ratio 10/10) were assessed. Clinical evaluation was performed using a modified version of semistructured interviews. Autonomic testing was performed by means of spectral analysis of heart rate and continuous blood pressure by sparse discrete Fourier transformation at rest and under mental stress. RESULTS: There were no significant difference between the two samples in age, sex distribution, BMI, resting systolic, or diastolic blood pressure, nor was there a difference in autonomic baseline functioning between the 40 patients with erythrophobia and the control subjects. On the other hand, patients with erythrophobia consistently showed higher pulse rates (88 +/- 20 vs. 78 +/- 9 bpm, p <.05), higher total heart rate power values (8.40 +/- 0.63 vs. 8.07 +/- 1.02 p <.05), higher midfrequency spectral values (7.38 +/- 0.66 vs. 7.02 +/- 1.18, p <.01), higher high-frequency spectral values (6.89 +/- 0.86 vs. 6.48 +/- 1.44, p <.05), and lower baroreceptor sensitivity (8.62 +/- 8.16 vs. 11.65 +/- 4.42, p <.005) than the healthy subjects. ANOVA showed a significant group interaction (p <.0001) between the samples. CONCLUSIONS: This study provides evidence for abnormal autonomic functioning in patients with erythrophobia when under mental stress.

Adult↗