SEARCH · Search PubMed
Results for “ADIE SYNDROME”
Search indexed PubMed citations on genomics, clinical trials, systematic reviews and public health. Explore titles, authors and supplied subject terms, then open the PubMed record.
Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.
[Etiology and localisation of pupillotonia and of Adie's syndrome].
Explore the source record for details and available documents.
A search for serum antibodies in Adie's syndrome.
Serum specimens from a large number of patients with Adie's syndrome were checked for virus antibody levels: Measles hemagglutination inhibition (HI), parainfluenza types I, II and III (HI), Epstein-Barr immunofluorescence, Mumps complement fixation (CF), Adenovirus (CF), Varicella-Zoster (CF), Herpes simplex (CF), Cytomegalovirus (CF), Mycoplasma hominis (CF), Toxoplasma gondii passive hemagglutination. These antibody levels were compared with specimens from a control group of similar age distribution and sex ratio. No statistically significant differences between the patients with Adie's syndrome and the control group could be demonstrated. This observation offers some indirect support to Harriman's idea that Adie's syndrome is not due to a viral ciliary ganglionitis but rather to an indolent neuronal degeneration in the ciliary and dorsal root ganglia.
Adie's syndrome: a medical treatment for symptomatic patients.
A symptomatic patient with Adie's syndrome is described as successfully managed with pilocarpine 0.125% applied topically to the involved eye three times daily. This is the first report of such a patient responding to direct parasympathomimetic therapy. Alternate approaches to the therapy of Adie's syndrome are mentioned. An approach to the management of Adie's syndrome is proposed.
The therapy of Adie's syndrome with dilute pilocarpine hydrochloride solutions.
Impressed by the benefit two patients with Adie's Syndrome derived from topical 0.1% pilocarpine therapy, we performed the present study to examine the measurable effects of 0.1% pilocarpine in several patients with this syndrome. Our study suggests those patients with Adie's Syndrome who lack stereoacuity may benefit from long term therapy with dilute pilocarpine if their stereoacuity may benefit from long term therapy with dilute pilocarpine if their stereoacuity improves following a single drop of 0.1% pilocarpine. Therefore, we recommend measuring stereoacuity during pupil diagnostic testing with 0.1% pilocarpine hydrochloride. Potential problems concerning medical therapy of Adie's Syndrome with parasympathomimetics are discussed.
[The significance of certain clinical aspects of Adie's syndrome].
Explore the source record for details and available documents.
[Tonic pupil and Adie's syndrome].
Explore the source record for details and available documents.
[Pathogenetic and clinical considerations on the association of Adie's syndrome and the Klippel-Feil congenital malformation].
Explore the source record for details and available documents.
[Adie's syndrome from a neurological standpoint (author's transl)].
The findings in 22 examinations of tonic pupils, 15 of which presented Adie's syndrome, are described. The neurological and chemical as well as liquor analyses are normal except for the well known tendon reflex anomalies. Neurophysiologically, in 7 of 10 examinations mild disruptions in the sensory nerve conduction velocity were observed. The literature of the last 10 years, where it is of interest from a neurological standpoint and where it relates to the site of damage or to the origin of the tendon reflex abnormalities in Adie's syndrome, is discussed. Adie's syndrome is interpreted as damage to the peripheral nervous system in a polyneuropathic form.
[OBSERVATIONS ON THE COURSE OF ADIE'S SYNDROME].
Explore the source record for details and available documents.
[Pupillographic findings in Adie's syndrome].
Explore the source record for details and available documents.
[Tonic pupils in Sjögren's syndrome].
INTRODUCTION: Adie's syndrome is usually a disease of unknown origin. We report two cases secondary to Sjögren syndrome. CASE REPORTS: A 26-year-old man developed in few months a sensitive neuropathy with a bilateral tonic pupil. A 50-year-old woman complained of sensitive signs probably related to a ganglionopathy and dysautonomic disorders affecting sudomotor and vasomotor functions. Adie syndrome had been diagnosed three years earlier. In both patients, the systemic signs and the results of the complementary tests led to the diagnosis of Sjögren's syndrome. Corticosteroids had limited effects on the sensitive signs and no influence on the tonic pupils. CONCLUSION: Adie syndrome, isolated or accompanied by other dysautonomic disorders, may reveal or precede the diagnosis of Sjögren's syndrome.
Segmental palsy of the iris sphincter in Adie's syndrome.
Explore the source record for details and available documents.