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At least 1,063 records · Page 59Linked to original sources
PubMed1979

Delirium tremens and related clinical states. Aetiology, pathophysiology and treatment.

Definitions of Delirium Tremens (DT) and related clinical states are discussed together with the concepts of aetiology and pathogenesis in relation to psychiatric disease. The withdrawal theory which considers reduction or cessation of alcohol intake as an important precipitating factor in DT is discussed; this theory is supported by experimental studies of ethanol withdrawal in man and by studies indicating cross dependence between ethanol and several other CNS depressors; the arguments in the literature for and against the withdrawal theory are discussed. Other possible aetiological factors such as type of liquor, hypovitaminosis, liver disease, dysfunction of the adrenals and fat emboli are reviewed, and it is concluded that these factors are unimportant as precipitating or specific aetiological factors in DT. Possible pathophysiological mechanisms are discussed, especially concerning electrolyte metabolism and cerebral blood flow and oxygen consumption. It is concluded that the pathophysiology of the disease is poorly understood, and some possibilities for future research are indicated. The discussion about treatment of DT is concentrated on drug treatment, and the literature concerning antipsychotics and sedatives is reviewed. It is concluded that barbital, a long-acting barbiturate, is the most effective treatment; diazepam can be recommended as an acceptable alternative. Finally, practical recommendations concerning treatment are given.

Adrenal Insufficiency↗
PubMed2006

Causative or precipitating aspects of burning mouth syndrome: a case-control study.

BACKGROUND: On causative or precipitating causes of burning mouth syndrome (BMS), there is a lack of consensus. In this prospective case-control study, we compared clinical features and laboratory aspects to evaluate the association of the proposed causative/precipitating factors of BMS. METHODS: A total of 61 BMS patients and 54 control subjects underwent several evaluations: rest and stimulated salivary flow rates measurements, laboratory tests, isolation of Candida species, assessment of parafunctional activities, detection of anxiety and depression by means of the Hospital Anxiety and Depression Scale. Odds ratio and 95% confidence interval were calculated to compare the variables. RESULTS: No statistically significant differences were found with regard to the tested variables except for anxiety and depression. CONCLUSIONS: The results of this study seem not to support a role for the usually reported causative or precipitating factors of BMS and efforts should be addressed towards different aetiologies including possible neuropathic mechanisms of BMS.

Anxiety↗
PubMed1982

Critical vitamins for low birthweight infants.

All vitamins are "critical" by definition for the growing infant. However, some of them are particularly relevant to the preterm or low birthweight (LBW) infant in whom a deficiency can occur more frequently than in a full-term newborn. In LBW infants vitamin deficiency may develop due to (1) low body stores at birth, (2) low intake, (3) limited absorption, (4) increased need or utilization, (5) presence of certain clinical conditions. The first reason concerns all lipid-soluble vitamins, and particularly vitamin E and K, which cross the placenta with some difficulty. Among hydrosoluble vitamins, cord-blood levels of vitamin B6 have been shown to be abnormally low in preterm infants. Low intake can occur because of low vitamin levels in milk or because of delayed and/or insufficient feeding. Limited intestinal absorption of vitamins in LBW infants has only been demonstrated for vitamin E and folic acid. The rapid post-natal growth may lead to increased vitamin utilization. In some clinical conditions particularly high intakes of certain vitamins are indicated. In our opinion, the really "critical" vitamins in LBW infants are vitamin D, E, K and folic acid, for which routine supplementation can be recommended, and possibly vitamins C and B6 under special circumstances.

Avitaminosis↗
PubMed1982

Do breast and bottle fed babies require vitamin supplements?

In the present stage of knowledge, the following approach to vitamin supplementation seems reasonable. Breast-fed full-term infants should be supplemented with vitamin D (400 I.U./day). Supplementation of bottle-fed infants will depend upon the vitamin content of the formula which is being used. Pre-term and LBW infants may need higher amounts of vitamin D (800-1 000 I.U./day) and should be supplemented with vitamin E (1-2 I.U./day), C (50 mg/day), B6 (30-60 mcg/day) and folic acid (50-100 mcg/day) when they are being fed formulas with high amounts of PUFA and proteins or with iron. Multivitamin supplementation appears to be acceptable for infants of very low birth weight (less than 1 500 g) until they reach a body weight of at least 2 000 grams or a caloric intake of 300 kcal/day. Finally, the present routine of administering 0.5-1.0 mg of vitamin K at birth to all infants should be continued.

Avitaminosis↗
PubMed1985

General aspects of nutrition, absorption and metabolism in patients with cystic fibrosis.

It is important to determine the energy losses and the possible deficiencies of vitamins and trace elements for the adequate treatment of the patient. The energy loss presumably can be assessed best by direct calorimetry, supplemented by the assay of faecal fat, nitrogen and of breath hydrogen. Restriction of energy losses is feasible by prescribing a high-protein diet with a normal fat content, preferably polyunsaturated fats, in combination with pancreatin. Deficiencies of vitamins and trace elements can be prevented by timely supplementation.

Avitaminosis↗
PubMed1975

The effect on the rat liver of long-term administration of different alcoholic beverages together with inadequate diets.

Rats were given 36 per cent of calories as ethanol, gin, brandy, whisky or red wine together with hypocaloric (25 per cent of normal), hypocaloric--low-protein--highfat, or hypocaloric--low vitamin diets for several months and compared with rats given isocaloric amounts of glucose instead of alcohol. In spite of high mortality rate no severe liver lesions occurred, especially no cirrhosis. Congeners present in different alcoholic beverages therefore seem to lack important hepatotoxic effects at least in the rat.

Alcoholic Beverages↗
PubMed1989

Nutritional status and psychometric test scores in cognitively impaired elders.

In a pilot study, we compared dietary and biochemical status for vitamin B1, B2, B12, and C and folate, iron, and protein in CI and CN elders grouped by MMSE scores. Biochemical evaluation of folate and B12 status was continued beyond the preliminary study. Intakes of all nutrients except folate exceeded 66% of the RDA. There were no significant differences among MMSE groups for dietary intake or biochemical values. Transketolase activity increased with degree of impairment, whereas measures for the other vitamins declined. Low or deficient LAA levels were found in 33% of all elders. Folate and B12 deficiencies, respectively, were found in 11% (12/109) and 4% (10/254) of the population. Very low values for serum B12 (less than 250 pg/ml) existed in 15% (37/254) of the subjects (31 CI; 7 CN). Moderately impaired elders (MMSE = 15-20) who used multivitamin supplements had RBC folate (p less than .003) and serum B12 (p less than .02) levels higher than those of subjects who did not use supplements.

Aged↗