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At least 1,045 records · Page 58Linked to original sources
PubMed2005

[Treatment of condylar fracture by external pterygoid muscle-condyle anatomical reduction and internal rigid fixation with mini titanium plates].

PURPOSE: To investigate the surgical indications, procedures and treatment outcomes of condylar fracture by external pterygoid muscle-condyle anatomical reduction and internal rigid fixation with mini titanium plates. METHODS: Patients with dislocation of the condylar head, fracture of the condylar neck and subcondylar neck (the degree of displacement greater than 30 to 45) and decreased vertical height of the ramus (greater than 4 to 5mm) were included in the study. Modified aural-temporal stick-shaped incision or retromandibular approach was adopted in 23 cases with 28 condylar fractures. The fractures were treated by external pterygoid muscle-condyle anatomical reduction and internal rigid fixation with mini tension band titanium plates. All the patients were re-examined and evaluated 3 to 6 months after surgery in respects of facial symmetry, mouth opening, types of mouth opening, occlusion and mastication. RESULTS: All the patients had symmetrical face, mouth opening increased from 26.7+/-7.2 (before surgery) to 38.4+/-5.6 (3 months after surgery) and 37.2+/-4.5 mm(6 months after surgery). 2 patients with postoperative malocclusion had intermaxillary distraction for 1 week and their occlusion returned to normal. Radiographic check-up showed the condyle and titanium plates without displacement. No absorption was noted on the condylar surface. The patients had normal mastication. 2 patients with temporal facial paralysis recovered 3 months after conservative therapy. CONCLUSION: Treatment of condylar fracture by external pterygoid muscle-condyle anatomical reduction and internal rigid fixation with mini titanium plates in selected patients is an effective procedure for recovery of anatomical positions and masticatory function. Supported by Research Fund from Science and Technology Bureau of Jining city.

Bone Plates↗
PubMed1991

Familial autosomal recessive rigid spine syndrome with neurogenic facio-scapulo-peroneal muscle atrophy.

Two sisters and a first cousin presented with rigid spine and facio-scapulo-peroneal muscle atrophy. The patients belonged to a family with two first-cousin marriages. Electromyography, muscle and nerve biopsy showed neurogenic muscle atrophy without peripheral nerve involvement. Follow up did not show progression of the disease. This is the first observation of an association of neurogenic facio-scapulo-peroneal and rigid spine syndrome. The double first-cousin marriage suggests autosomal recessive inheritance.

Adolescent↗
PubMed1988

Relationship between perineal muscle contractions, penile tumescence, and penile rigidity during nocturnal erections.

The mechanism of penile erection and erectile dysfunction is still unclear and widely debated. The role of the perineal muscles in the erectile process, especially in changes in intracavernous pressure, is increasingly being studied on the hypothesis that perineal muscular contractions are essential to full penile rigidity. In a previous investigation we studied the correlation between voluntary perineal muscle contractions and intracavernous pressure during artificially induced erections. The purpose of the current study was to examine whether under normal conditions of nocturnal erection a similar relationship exists between the electromyographic activities of perineal muscles and changes in penile rigidity. Nocturnal penile recordings were made of seven volunteers with psychogenic erectile dysfunctions. During nocturnal erections simultaneous computerized recordings were made of penile tumescence, penile rigidity, and electromyographic activities of perineal muscles. The peaks for the three variables were reached simultaneously. The results of this study suggest the existence of two different physiologic phases: a vascular phase and a muscular phase. Furthermore, in cases of dysfunctions, specific diagnostic assessment and therapeutic management will be required for both penile tumescence and penile rigidity.

Adult↗
PubMed1975

Etonitazine-induced rigidity and its antagonism by centrally acting muscle relaxants.

Some narcotic drugs have been reported to produce increases in muscle tone in rats. In our laboratory we have found that etonitazine produces a 'lead-pipe' rigidity of the trunk and limb musculature. The reported studies were conducted to characterize etonitazine-induced rigidity more fully, to compare the degree of rigidity with that produced by morphine, codeine and methadone, and to assess the sensitivity of this rigidity to centrally acting muscle relaxants. Of the 4 narcotics tested, etonitazine was far more potent than methadone or morphine for producing rigidity; codeine did not produce peak rigidity comparable to the other 3 narcotics. Etonitazine-induced rigidity occurs at a supraspinal level since the effect was prevented by spinal transsection. Etonitazine-induced rigidity was prevented by the narcotic antagonists cyclazocine, pentazocine and naloxone but not by the serotonin depletor, p-chlorophenylalanine. Etonitazine-induced rigidity was antagonized by centrally acting muscle relaxants, including diazepam, methocarbamol, carisoprodol and zoxazolamine; in agreement with their relative clinical muscle relaxant potencies, diazepam is the most potent antagonist of etonitazine-induced rigidity.

Analgesics, Opioid↗