Effects of ACTH4--10 on self-stimulation behavior in the rat.
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Adrenocorticotropic Hormone↗
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Results for “Self Stimulation”
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Brain monoamines in the modulation of self-stimulation, feeding, and body weight.
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Animals↗
A portrait of the substrate for self-stimulation.
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Animals↗
Basal tegmental self-stimulation after septal ablation in rats.
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Animals↗
Effects of apomorphine on self-stimulation behavior.
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Animals↗
Concurrent circadian periodicities: gross activity and self stimulation with light.
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Animals↗
GABA antagonism lowers self-stimulation thresholds in the ventral tegmental area.
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Animals↗
Self-stimulation, salicylate and ambient temperature.
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Animals↗
Self-stimulation of hippocampus in rats.
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Animals↗
Community-referenced research on self-stimulation.
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Autistic Disorder↗
[Effect of ambient temperature on self-stimulation frequency and seizure development in the white rat].
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Animals↗
[A quantitative assessment and analysis of the temporal structure of the self stimulation reaction].
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Animals↗
[A neuropharmacologic analysis of the cholinergic mechanisms of self stimulation].
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Acetylcholine↗
Blood pressure, heart rate and respiratory responses from hypothalamic self-stimulation in the cat.
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Animals↗
Influence of monoamine precursors on the brain electrical self-stimulation in the rat.
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Acetylcholine↗
Self-stimulation enhancement in diabetic rats.
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Animals↗
Self-stimulation from dopaminergic terminal fields.
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Animals↗
Effect of pimozide on self-stimulation threshold under a continuous and fixed-interval schedule of reinforcement.
Rats implanted with a monopolar electrode in the medial mesencephalon and trained to press a lever to self-administer trains of stimulating pulses were tested under a continuous (CRF) and a fixed interval (FI-1s) schedule of reinforcement before and after systemic pimozide injection (0.35 mg/kg). As previously shown, this dose of pimozide caused reductions in maximal response rates and in the rewarding effectiveness of the stimulation. The magnitude of the attenuation in rewarding effectiveness was significantly greater under CRF than under FI conditions, an effect that can be attributed to the decrease in the number of earned rewards concomitant to a decrease in response rates. The present results show that the tighter control over reward density provided by the use of a FI schedule of reinforcement reduces the probability of artifactually measuring increases in reward thresholds following treatments that suppress response rates.
Animals↗