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At least 1,009 records · Page 56Linked to original sources
PubMed1991

[Vibrio alginolyticus and swimmer's otitis externa. 2 cases and review of the literature].

We describe two patients with acute diffuse external otitis (swimmer's otitis) acquired in the Mediterranean shore, with Vibrio alginolyticus recovered from ear fluid. We describe the biochemical profile and sensitivity pattern (MIC) of both strains, comparing it to previously published data. A literature review was also performed, in which we found evidence for increasing concern of V. alginolyticus as an human pathogen. Also there is a need for considering halophilic vibrio as potential pathogens, specially if there is also epidemiologic support.

Adult↗
PubMedDate not supplied

Hyperbilirubinemia in infants with urinary tract infection.

In the light of a retrospective analysis of 213 case records of infants with urinary tract infections, hyperbilirubinemia was disclosed in 19 cases. In 7 cases jaundice was the main cause of admission of these children to the hospital. It was observed mainly in the youngest age group up to 3 months. Jaundice was hemolytic in some cases and hepatic in others. Urinary tract malformations had no effect on the frequency of jaundice in urinary tract infection or on its intensity.

Anemia, Hemolytic↗
PubMedDate not supplied

[Clinical and pathogenetic significance of Proteus mirabilis antibodies in uveitis associated with joint lesions in children and adolescents].

Immunological studies in 95 children with uveitis associated with juvenile chronic arthritis revealed infection with Proteus mirabilis in 14.7% cases, mainly in cases with reactive arthritis and juvenile rheumatoid arthritis (JRA). A clear-cut relationship between P. mirabilis infection and severity of the disease was detected. In 85% children infected with P. mirabilis uveitis was associated with a rapid decrease of ocular functions and development of complications, one of the most frequent of which was vitreous fibrosis, generally not typical of classical rheumatoid uveitis. Antibodies to P. mirabilis were detected in 72.2% children with positive antinuclear factor, a marker of ophthalmoarticular rheumatic diseases. The pathogenetic significance of P. mirabilis in uveitis associated with articular involvement deserves further investigation, but even now the detection of P. mirabilis can be used as a marker of uveitis exacerbations and severity.

Adolescent↗
PubMed1978

Protection against experimental Proteus mirabilis pyelonephritis in rats and significance of immunity.

Intraperitoneal immunization with formalin-killed bacteria as well as previous hematogenous infection with Proteus O3H1 showed a protective effect against hematogenous pyelonephritis in rats when the homologous strain was used. Transfer of hyperimmune antisera protected against hematogenously induced infection. Neither intravesical or intraperitoneal immunization with formalin-killed bacteria nor transfer of urines containing antibodies of the IgG class protected against ascending pyelonephritis when the O3H1 strain was used. Data are presented indicating that a rise of pH might decrease the biological effect of antibodies, suggesting that Proteus urease activity is a virulence factor of importance in this context.

Animals↗
PubMed1980

Experimental infection in rabbits evoked by Pseudomonas aeruginosa, Proteus mirabilis, Escherichia coli and Klebsiella pneumoniae treated with gentamicin, amikacin and sisomicin.

Rabbits were infected with freshly isolated strains of Ps. aeruginosa, P. mirabilis, E. coli or K. pneumoniae and treated with gentamicin, amikacin or sisomicin in a controlled study. Therapeutic results were evaluated by survival of animals and viable counts of etiologic bacteria in several tissues after sacrification of treated animals at the same time. Sisomicin was the drug of choice for Ps. aeruginosa and P. mirabilis infections, when compared with amikacin and gentamicin. Its action is practically equal to that of amikacin in K. pneumoniae infection. Sisomicin appeared less active in E. coli infections than amikacin, but was superior to gentamicin.

Amikacin↗
PubMed1991

[Frequency and antibiotic susceptibility of bacteria responsible for urinary infections in private practice from 1986 to 1990. Results of 5398 samples].

For each year from 1986 to 1990, fourteen private laboratories from all over France collected epidemiological data on germs discovered in urinary infections in France. This retrospective and comparative study covers 5,398 dossiers. The sensitivity of germs in private practice in urinary infections (ampicillin, AM-AC, cephalosporin, ceftriaxone, gentamicin, pipemidic acid, pefloxacin, cotrimoxazol) were analysed. Germ resistance tests carried out by the CMI study in the centre of reference (Pr Thabaut, HIA Bégin) showed variable divergences according to the different antibiogram techniques employed by the private laboratories.

Adolescent↗
PubMedDate not supplied

Anti-M in children with acute bacterial infections.

Four children, 7 to 24 months old, were found to have anti-M at the time of admission to the hospital for severe acute bacterial infections. All were M-N+. Two patients had meningitis, one had septic arthritis, and the fourth had a third-degree burn of the left hand. In follow-up studies the anti-M of patients No3 and No4 were no longer detectable after 12 and 11 months respectively. In all patients no demonstrable antibody was in either maternal or cord sera at time of birth. The clinical data and bacterial isolations lead us to postulate that bacterial infections account for the formation of naturally occurring anti-M in M-negative persons.

Acute Disease↗
PubMed1985

Aminophylline-induced suppression of pulmonary antibacterial defenses.

Respiratory infections are frequently observed in patients with chronic obstructive pulmonary disease, indicating that host defenses are compromised. Antibacterial defenses of the lung against such infections include the alveolar macrophage and polymorphonuclear leukocytes (PMN) that migrate into the lung to provide auxiliary phagocytic defenses. To test the hypothesis that aminophylline acutely impairs pulmonary antibacterial defenses, mice were challenged by aerosol inhalation with Staphylococcus aureus or Proteus mirabilis and injected intraperitoneally with aminophylline (20, 40, or 80 mg/kg). Pulmonary bactericidal activity and total lavaged lung cell and differential counts were determined 4 h after bacterial challenge. The highest dose of aminophylline suppressed the killing of S. aureus so that 55 +/- 5% of the initial viable bacteria remained as compared with 22 +/- 4% in the control animals. In contrast, there was a dose-related suppression of pulmonary antibacterial defenses against gram-negative bacteria. With doses of 40 and 80 mg/kg, lung defenses were ablated, allowing the proliferation of P. mirabilis to 115 +/- 9% and 253 +/- 9%, respectively, the control value being 26 +/- 3%. The number of PMN obtained by lavage after aerosol challenge with P. mirabilis was also inhibited by aminophylline in a dose-dependent manner. From the lungs of untreated animals 5.0 +/- 0.3 X 10(6) PMN were recovered as compared with 3.3 +/- 0.1 X 10(6), 2.5 +/- 0.2 X 10(6), and 1.8 +/- 0.1 X 10(6), respectively, with increasing doses of aminophylline. The bactericidal activity of lavaged PMN from the lungs of aminophylline-treated rats challenged with the gram-negative bacterium in vivo was significantly depressed when compared with that in control animals.(ABSTRACT TRUNCATED AT 250 WORDS)

Animals↗