Search PubMed⌕ Search

SEARCH · Search PubMed

Results for “Autonomic Pathways”

Search indexed PubMed citations on genomics, clinical trials, systematic reviews and public health. Explore titles, authors and supplied subject terms, then open the PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 991 records · Page 55Linked to original sources
PubMedDate not supplied

Further observations on the sensitive innervation of some bird's proctodeum.

The AA. studied the autonomic and sensitive somatic innervation of some female bird's proctodeum, through the properly modified Ruffini's gold chloride method. The vegetative component was constituted by ganglion cells of different size, isolated or grouped to form ganglia, found along the course of nerve trunks or in the concurrent point of different nerve bundles. The sensitive somatic innervation was represented by free and encapsulated endings differently distributed in the thickness of the wall. The former were composed of thin networks, while the latter, located more frequently in the muscular tunica and in the subadventitial connective, were composed of encapsulated receptors classified as Pacini, Pacini-like and Herbst corpuscles. The morphology of these receptors was described and hypotheses were brought up about their probable functional role. The AA, also found, even if very rarely, helicoidal collagen fibres around nerve fascicles.

Animals↗
PubMed2003

[Pathophysiology of cluster headache].

The aetiology of cluster headache (CH) is unknown and an unifying pathophysiologic explanation is not available. Based on the clinical features of the disorder, three areas appear to be involved in the pathogenesis and the expression of cluster headache: the trigeminal nociceptive pathways, the autonomic system and the hypothalamus. A brain stem connection may exist between the trigeminal nerve and the cranial parasympathetics. This would explain the reflex trigeminal-autonomic activation but a central nervous system dysfunction located in posterior hypothalamic gray matter is probably pivotal in the process. Such a dysfunction is supported by both posterior hypothalamic activation shown during CH attacks by positron emission tomography and increase of posterior hypothalamic volume shown in magnetic resonance imaging using voxel morphometry. Moreover, such a neurovascular model could be more precise considering some recent experimental and clinical research data as serotoninergic neuromediation involvement, nitric oxide one and a possible genetic predisposition.

Autonomic Nervous System↗