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Results for “Erythema Multiforme”

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PubMed1978

[Yersiniosis].

A survey was undertaken about bacteriologic, epidemiologic and clinical features of infections caused by Yersinia. Yersinia enterocolitica and Yersinia pseudotuberculosis are gram negative rods of the enterobacteriaceae family. The number of cases of these infections has increased considerably in the past few years. Erythema nodosum often occurs after an enteritic infection by Yersinia. Other skin manifestations are erythema exsudativum multiforme, erythema figuratum and "drug-eruption-like exanthema". Moreover, Yersinia-arthritis may occur. In one particular case a Reiter-syndrome was described. The mechanism whereby Yersinia induce skin lesions is unknown. Prognosis is good. Therapy of choice for infections caused by Yersinia are tetracyclines.

Adult↗
PubMed1984

[HLA typing of patients with herpes simplex recidivans and postherpetic erythema exsudativum multiforme].

62 patients suffering from recurrent herpes simplex, with ten out of them showing postherpetic erythema multiforme, have been investigated with special reference to histocompatibility standardisation. In comparison with a control group (n = 170), our patients revealed a high and statistically significant level of HLA-B5 (p less than 0,01). Postherpetic erythema multiforme seems to be associated with HLA-A9.

Adult↗
PubMed1987

[Erythema exsudativum multiforme in rickettsiosis].

A patient with Mediterranean spotted fever with a typical exanthema acquired in South Africa developed erythema multiforme 12 days after the beginning of the rickettsial infection. The exanthema caused by the rickettsial infection as well as the erythema multiforme were effectively treated with tetracyclines.

Bites and Stings↗
PubMed1998

Depot sulfonamid associated linear IgA bullous dermatosis with erythema multiforme-like clinical features.

A case of erythema multiforme-like reaction, following therapy with sulfadimethoxynum is reported in a 19-year-old male patient. Histological examination demonstrated a subepidermal bulla and direct immunofluorescence revealed linear deposition of IgA at the dermoepidermal junction. These observations illustrate that linear IgA bullous dermatosis can mimic the clinical features of erythema multiforme and suggest the possibility of drug-induced pathogenesis.

Adult↗