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At least 937 records · Page 52Linked to original sources
PubMed2004

Molecular epidemiology of porcine H3N2 influenza A viruses isolated in Germany between 1982 and 2001.

We examined influenza virus strains of the subtype H3N2 from outbreaks of respiratory diseases in swine herds in Germany. Four different clusters can be distinguished when comparing parts of the HA1 gene from porcine H3N2 isolates analyzed between 1982 and 2001. Comparison between these clusters reveals a bp homology of less than 90%. In contrast, the homology within the clusters is between 93.7 and 100%. Each of these clusters was confined to a specific time period. For the NA gene an additional cluster is formed by the porcine H1N2 isolate. The findings that different subtypes and drift variants are circulating in the German pig population explain the emergence of new influenza virus variants and the need for continued surveillance of swine.

Animals↗
PubMed1977

Neurologic disease in naturally occurring Salmonella choleraesuis infection in pigs.

A granulomatous meningoencephalomyelitis was seen in 17 feeder pigs submitted for diagnostic evaluation from eight farms. Vasculitis and destructive microgranulomas were widespread in both brain and spinal cord. Occasionally there were microabscesses. Salmonella choleraesuis var. kunzendorf was isolated from six of the eight groups, and there were lesions of acute salmonellosis in all animals examined. In three of the affected herds the principal disease manifestation was neurologic.

Animals↗
PubMed1979

Pathology of proliferative haemorrhagic enteropathy in pigs.

Examination of the small intestine of pigs with proliferative haemorrhagic enteropathy showed changes consistent with defects in vascular permeability. Early in the disease there were many eosinophils and distension of lacteals and intercellular spaces with proteinaceous material. Later the predominant features were red blood cells and exudate in tissue spaces. This was most severe and extensive at the tips of villi which were covered by a cast of cells and fibrinous exudate. Adenomatous intestinal mucosal cells contained organisms that were free within the apical cytoplasm and were morphologically identical with those seen in the related disease, porcine intestinal adenomatosis. Also these bacteria were seen free in the subepithelial mucosal area, in blood vessels and within membrane-bound vesicles in phagocytic cells in the mucosa and its blood vessels. Mast cells were prominent in some areas as were thrombosed vessels.

Animals↗
PubMed1980

Intestinal atresia and stenosis in animals: a report of 34 cases.

Intestinal atresia was found in 29 animals and stenosis in five. Atresia was found in the duodenum in one pup; in the jejunum in nine calves, two lambs and one piglet; in the ileum in one pup, one lamb and one piglet; and in the colon in one foal, seven calves, one lamb, one piglet and three kittens. Stenosis was found in the duodenum of a foal, in the jejunum in two calves and one pup, and in both the ileum and the colon of a kitten. One lamb showed ileal atresia as well as ileal stenosis. We classified the atresia as type 1, membrane atresia (four cases); type 2, cord atresia (six cases); and type 3, blind-end atresia (19 cases). In the literature and our own study, atresia was found only in the small intestine in puppies, but in foals and kittens it was found only in the colon. Atresia of the colon is more common in animals than in man, but multiple atresia, frequent in man, is rare in animals.

Animals↗
PubMed1981

Focal mineralization and nonspecific granulomatous inflammation of respiratory mucous membranes in pigs.

Discrete mineralized foci and granulomatous inflammation occurred in the lamina propria mucosae of respiratory mucous membranes of adult pigs. Lesions were present in clinically healthy pigs of both sexes, including castrated males, fed various pelleted or non-pelleted diets. They were mainly in longitudinally corrugated mucosae of the dorsal wall of the trachea. Identical mineralization and inflammation occurred in the nasal cavity and, in decreasing frequency and intensity, in the thoracic trachea and bronchi. The lesions in respiratory mucous membranes occurred in pigs with and without mineralization in other organs. The distribution of lesions in the respiratory tract, and the higher frequency in pigs fed non-pelleted dusty feeds, suggest that focal mineralization was caused by inhaled particles of calcium salts.

Animal Feed↗
PubMed1984

Ocular and orbital cysticercosis in hogs.

The prevalence of cysticercosis in hog muscles was correlated with oculo-orbital prevalence, and histologic characteristics of changes in the parasite and hog were observed. The orbital contents, globe, and eyelids of 39 hogs affected with Cysticercus cellulosae were examined. Thirty-seven parasites were found: 35 within the orbit and two intraocular. Twenty parasites with adjacent host tissue were studied histologically. Inflammatory infiltrates ranging from diffuse lymphoplasmacytic to dense granulomas were seen in 16 cysticerci. The scolex was unaltered in all specimens.

Animals↗
PubMed1984

Pathomorphology and pathogenesis of bacterial meningoventriculitis of neonatal ungulates.

Bacterial meningoventriculitis was studied in 26 neonatal ungulates. Preceded by a substantial bacteremia, usually due to Escherichia coli, the fibrinopurulent inflammation involved leptomeninges, choroid plexuses, and ventricle walls, but largely spared the neuraxial parenchyma. It is proposed that this surface-relatedness results from the transport of bacteria by monocytes of low bactericidal power, migrating by normal pathways to maintain significant surface populations of macrophages. The neuraxial parenchyma is spared because of its normal lack of a macrophage population. A similar pathogenesis would hold for the frequent concurrent appearance of serositis and synovitis.

Animals↗
PubMed1985

Enhanced myotoxicity and involvement of both type I and II fibers in monensin-tiamulin toxicosis in pigs.

Simultaneous administration of monensin and tiamulin to pigs resulted in enhanced myotoxicity. Skeletal muscles of tongue, diaphragm and legs were preferentially affected, whereas the masseter, longissimus thoracis and cardiac muscles, including the left auricle, were spared. Histochemical examination revealed an involvement of both type I and II fibers of skeletal muscles.

Animals↗
PubMed1986

Neuropathology of experimental 3-nitro-4-hydroxyphenylarsonic acid toxicosis in pigs.

Twenty pigs were fed a diet containing 187.5 mg kg-1 of 3-nitro-4-hydroxyphenylarsonic acid (3-nitro). Ten pigs were euthanized at intervals up to 29 days, 3-nitro was withdrawn from the diet of the remaining pigs on day 30, and these animals were subsequently euthanized at intervals up to 49 days after commencement of the experiment. A nervous syndrome characterized by clonic convulsive episodes inducible by exercise, developed at day 11. Paraparesis was apparent at day 22 progressing to paraplegia by day 33 (3 days after cessation of 3-nitro feeding). Histopathologic examination revealed myelin and axonal degeneration in the white matter of the spinal cord coincident with the onset of nervous signs. Marchi-positive degeneration was present in the dorsal funiculus at cervical level at day 22. Lesions intensified with increasing duration of toxicosis and while degenerate fibers were seen in all funiculi, there was preferential involvement of the fasciculi gracilis and cuneatus, the peripheral regions of the ventral and lateral funiculi, and a discrete area of the dorsal region of the lateral funiculus. Peripheral and optic neuropathies were evident from day 32 but were always mild and focal. The experiment establishes 3-nitro as a central-peripheral neurotoxicant of pigs.

Animals↗
PubMed1994

Specific in situ hybridization of the intracellular organism of porcine proliferative enteropathy.

The identity of the intracellular bacteria found in the enterocytes of pigs with proliferative enteropathy was investigated using specific DNA probes to various Campylobacter species and to a novel organism, ileal symbiont intracellularis. The ilea from pigs (Nos. 1-7) that were diagnosed by routine histopathology as having proliferative enteropathy were used. Diagnosis was made on the basis of proliferation of the enterocytes on hematoxylin and eosin-stained sections and the presence of large numbers of intracellular curved organisms on Warthin-Starry silver-stained sections. Four of these pigs (Nos. 1-4) had the chronic form of the disease, porcine intestinal adenomatosis, and three (Nos. 5-7) had the acute form, proliferative hemorrhagic enteropathy. An additional three normal pigs (Nos. 8-10) were obtained from three separate farms with no history of proliferative enteropathy. Frozen ileal sections were examined by in situ hybridization with DNA probes specific for ileal symbiont intracellularis and the three porcine intestinal Campylobacter species, C. coli, C. hyointestinalis, and C. mucosalis. In all seven pigs with either the intestinal adenomatosis or hemorrhagic enteropathy form of the disease, a DNA probe specific for ileal symbiont intracellularis hybridized to localized foci in the apical cytoplasm of ileal enterocytes. These hybridization sites corresponded to the location of intracellular bacteria in silver-stained sections of adjacent tissue. Sections from the three normal pigs tested with this probe and from all pigs tested with the Campylobacter species-specific DNA probes showed no specific hybridization reactions. The identity of the intracellular organism in these diseased pigs is ileal symbiont intracellularis.

Animals↗
PubMed1995

Disseminated adenovirus infection associated with cutaneous and visceral hemorrhages in a nursing piglet.

An unusual adenovirus infection in a conventionally raised nursing pig was characterized clinically by hemorrhagic diathesis of the skin and microscopically by disseminated viral inclusions in the endothelial, interstitial, and epithelial cells of the skin, kidney, spleen, liver, heart, and small and large intestines. This disease was confirmed ultrastructurally by demonstration of typical adenoviral particles in the nuclei of endothelial cells in the myocardium and of interstitial cells in the small intestine. This is the first report of disseminated adenovirus infection in a nursing pig in North America.

Adenoviridae Infections↗