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Results for “Neural Conduction”

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At least 919 records · Page 51Linked to original sources
PubMed1971

Classification of neuromuscular disorders.

Anew type of classification of neuromuscular diseases is presented. It is based on etiology; when this is not possible, on pathogenesis; if neither is possible, on distinctive features, biochemical greater than morphologic greater than clinical-genetic ones. The initial categorization designates which cell type, lower motor neuron or myofiber, is considered responsible for the major abnormality. This classification is designed to promote understanding of the pathogenesis and etiology of neuromuscular diseases in general and be applicable to a particular patient when the diagnostic studies are completed. It is not arranged as an approach to the initial differential diagnosis of a given patient.

Amyloidosis↗
PubMedDate not supplied

[Space-time organization of recruitment in rabbit cerebral cortex].

The spatio-temporal organization of the recruiting responses in the cerebral cortex was studied in an acute experiment on alert rabbits. Local low-frequency stimulation of the thalamic central medial nucleus leads to the appearance of both "generalized" and "local" spindles. Temporal shifts of 0.1--2 sec. occur in the appearance of the "generalized" spindles. The performed analysis permits the assumption that under local stimulation the spatio-temporal dynamics of the recruiting response is connected with the appearance of several spindle pacemakers which differ in their characteristics, e. g. in the speed of the excitability recovery and in their interrelations.

Animals↗
PubMed1979

A study of synaptic plasticity in hippocampal slices.

Long-lasting potentiation in the hippocampal pathways is used at present as a model for long-term plasticity in the nervous system. In this study post-tetanic potentiation was investigated in the dentate gyrus-area CA3 pathway by extra- and intracellular recordings from transverse slices of the mouse hippocampus. Tetanization of the dentate gyrus led to a reduction in the latency of action potentials (APs) and EPSPs recorded from area CA3, to an increase in the amplitude of EPSPs and in the steepness of their ascending slope, and to an augmented probability of APs. These changes persisted for a period of several seconds to 30 min after tetanization. Of special interest were records from cells responding with EPSPs: of a short latency (2-3 ms) which was not changed by an increase in the frequency and strength of stimulation. We assume that such EPSPs are monosynaptic. Our results suggest that monosynaptic EPSPs can undergo long-lasting (up to 30 min) post-tetanic potentiation.

Action Potentials↗