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Results for “RADIATION SICKNESS”

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PubMed1976

[Shifts in several aspects of phospholipid metabolism in the brains of white rats with radiation sickness].

Sublethal (400) and lethal (1600) doses of roentgen decrease the level of neutral phospholipids of rat brain and increase that of acid phospholipids. With the development of radiation disease the content of free glycerine and glycerophosphate increases and the glycerokinase and glycolytic pathways of glycerophosphate biosynthesis are activated together with the intensification of the direct and reverse reactions catalyzed by glycerophosphate dehydrogenase. The permeability of the artificial bimolecular phospholipid membranes prepared from rat brain phospholipid extracts is significantly increased at the end of the radiation.

Animals↗
PubMed1990

[The immunological monitoring of patients with acute radiation sickness 12 to 36 months after the accident at the Chernobyl Atomic Electric Power Station].

Immunoassays of the 1st and 2nd levels were investigated in patients with acute radiation disease 12-36 mos. after irradiation. A decrease in the level of helper/inducer T-lymphocytes, a tendency to a decrease in the number of suppressors and an increase in the number of theophylline-stimulated T-cells in patients with acute radiation disease of the 3rd degree were observed. Control of the immune status of this group of patients is recommended.

Accidents↗
PubMed1998

[The clinico-hematological picture of acute radiation sickness in the initial period after the Chernobyl catastrophe].

The time and degree pattern of the primary response manifestations did not in all instances correlate with subsequent severity of radiation pathologies, which fact may be explained by different levels of bodily exposure to alpha and beta-emitting radionuclides, products of combustion of different substances, psychological status of the people. There is a wave-like change in the parameters characterizing the cellular composition of blood.

Acute Disease↗