[Man as a source of zoonotic microsporosis caused by Microsporum canis].
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Animals↗
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Results for “MICROSPORUM”
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[Zooanthroponotic microsporosis caused by Microsporum distortum].
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Child↗
[Mixed dermatophytic infection of the scalp with Trichophyton schoenleinii and Microsporum rivalieri].
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Child↗
On a peculiar dermatophytosis caused by Microsporum gypseum.
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Adult↗
[Study of human hair infected with Microsporum canis].
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ABO Blood-Group System↗
[Case of tinea barbae profunda caused by Microsporum nanum].
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Aged↗
Isolation of saprophytic Microsporum praecox Rivalier from sites associated with horses.
Several M. praecox isolates of saprophytic origin were obtained in Belgium from horses and their surroundings. Visualization of macroconidia in dust collected in stables proved its saprophytic origin. A few strains were obtained from human cases of tinea corporis.
Animals↗
In vivo studies on phospholipid biosynthesis in Microsporum gypseum.
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Choline↗
Tinea faciei caused by Microsporum canis. A case report.
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Adult↗
[Mechanism of transmission of Microsporum canis, and the characteristics of clinical course and diagnosis of zooanthroponotic microsporosis].
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Animals↗
[The production of antibodies to an extracellular keratinase of microsporum canis (author's transl)].
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Antibodies, Fungal↗
[Microsporum canis outbreak caused by pedigree cats in the Karl-Marx city area].
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Adolescent↗
[Epidemiology and clinical manifestations of zooanthroponotic Microsporum infection].
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Animals↗
[Microsporum canis infections in Hungary].
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Adolescent↗
[First isolation of Microsporum ferrugineum (Ota 1921) in Munich].
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Child↗
[Microsporum canis infection in man. A local epidemic in Oslo].
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Adolescent↗
[Report on a group infection by Microsporum canis in the Magdeburg district].
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Adolescent↗
[Treatment of tinea microsporum. Metabolic interference between phenobarbital and griseofulvin].
Two epileptic children treated with phenobarbital and suffering from tinea capitis, not responding to a long lasting treatment with Griseofulvin, healed only after phenobarbital has been discontinued. We ascribe our therapeutic problems to the metabolic interference of Griseofulvin with phenobarbital. We have measured the blood concentration of Griseofulvin several times in one of our 2 children and in a control of the same age living in the same dietary conditions and in the same conditions of Griseofulvin intake; these measurements confirm these data. The point of interest of this report is to draw attention on: -the pharmacologic hypotheses put forward to explain this drug interference, -the other factors of a bad therapeutic response to a treatment with Griseofulvin.
Child↗