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Results for “FLUORIDE POISONING”

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PubMed1982

Evaluation of medicolegal investigators' suspicions and positive toxicology findings in 100 drug deaths.

The performance of trained medicolegal investigators was evaluated in 100 consecutive drug deaths, which occurred from January 1978 to May 1980 in St. Louis City and County. Carbon monoxide deaths were excluded from the study. The toxic agent responsible for death, as indicated by scene investigators and the decedent's drug history, was compared to the actual toxicology laboratory findings. In 84 of the cases, the toxicant was correctly indicated by the investigators. In the remaining 16 cases, 12 were suspected to be drug deaths but the major toxicant was not indicated, and in 4 cases no drugs were suspected. The manner of death had no influence on the investigators' performance. This study demonstrates the value of trained medicolegal investigators in providing helpful information to the pathologist and toxicologist before autopsy and laboratory analyses in cases of drug deaths.

Adult↗
PubMedDate not supplied

[Hydrotelluric fluorotic intoxication in North Yemen. First results (author's transl)].

The X Ray Department of the French medical mission of Taez receives patients coming from the whole Arab Yemen Republic (country situated in the south of the arab peninsula). Radiology has been a mean of detection and a mean of research of an important centre of osteofluorosis, a disease which had not been studied in this country until then. 47 cases have been collected throughout one year. The hydrotelluric origin of the intoxication has been proved by dosages of the spring waters used for drinking. The main usual clinical and radiological features of the disease are found in that population. The radiological classification proposed by ROHOLM has few clinical correlative interests, but seems available for epidemiological investigation. A first map of the North Yemen for chronic fluorisis has been drawn.

Adult↗
PubMed1981

Chemical profile of blood in fluoride toxicity. III. Plasma fibrinogen levels in rabbit.

Rabbits in two groups were given different doses of sodium fluoride i.e. low dose (10 mg NaF/kg body weight) and high dose (50 mg NaF/kg body weight). Blood was drawn from both groups on certain specific days after fluoride administration. The two different doses of sodium fluoride gave different response with regards to the fibrinogen levels in blood plasma. It is suggested that administration of low doses of sodium fluoride, induce new bone formation enhancing fibrinogen levels. But in high doses of sodium fluoride, moderate tissue damage results in high levels of plasma fibrinogen.

Animals↗
PubMed1981

Skeletal fluorosis from eating soil.

A woman with chronic pyelonephritis developed progressive muscular weakness and bone pain. For twenty years she had habitually ingested fluoride-rich soil. Osteosclerosis was found on x-ray examination, and fluorosis was confirmed by bone biopsy. Renal failure augmented skeletal retention of excessive fluoride intake which, in turn, appears to have intensified symptomatic renal osteodystrophy. Skeletal fluorosis from this unexpected source has not been previously described.

Adult↗
PubMedDate not supplied

[Effect of chorionic gonadotropin on estrogen excretion in healthy men and in fluorosis patients].

Estrogen excretion was studied by fractions in the urine of 10 healthy men and in 11 men suffering from fluorosis before and after the chorionic gonadotropin (CG) load. The response to the CG administration was expressed in increased urinary estrogens excretion in the healthy men and in the patients with fluorosis; the hyperexcretion intensity depended on the dose of the hormone administered. In comparison with healthy men, patients with fluorosis displayed specific temporary shifts in the maximal estrogen excretion peaks, and also a significant increase in estriol concentration in the urine. A conclusion was drawn that changes in the estrogen concentration in the urine of patients with fluorosis were connected with disturbed metabolism of these steroids, and not with elevation of their production in the testes.

Adult↗