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PubMedDate not supplied

The antimicrobial effect of narrow-band UVB (313 nm) and UVA1 (345-440 nm) radiation in vitro.

Two new UV lamps were investigated with respect to their antibacterial effectiveness in vitro. Propioni (n = 20 strains) and Micrococcaceae (n = 16 strains) bacteria extracted from acne patients were applied to RCM and sheep blood agar plates and irradiated with a narrow-band UVB lamp (TL 100W/10R) at a wavelength of 313 nm and a UVA1 lamp (TL 01) at a wavelength of 345-440 nm. The precisely defined energy levels were, in the case of narrow-band UVB, 0.00, 0.30, 0.50, 1.00, 2.00 and 3.00 J/cm2 and, in the case of UVA1, 0.00, 2.50, 5.00, 7.50, 10.00 and 20.00 J/cm2. UVA1 inhibited neither the growth of Propioni nor Micrococcaceae bacteria. In contrast, the growth of Micrococcaceae was inhibited already at a dosage of 0.30 J/cm2 of narrow-band UVB (P < 0.05), highly significant from 0.50 J/cm2 (P < 0.01) and to a maximum of 2.2 powers of 10 at 3.00 J/cm2 compared with non-radiated control plates. Propioni bacteria were significantly inhibited at the minimum dosage of 0.30 J/cm2 of narrow-band UVB (P < 0.01) and to a maximum of 2.8 powers of 10 at 3.00 J/cm2.

Acne Vulgaris↗
PubMed1981

Antagonism within populations of micro-organisms from normal human skin.

The skin flora of 20 normal subjects was sampled from 7-cm2 sites on the volar forearm, forehead and ball of the foot. Interactions between all aerobic and anaerobic strains isolated from the same site were examined by a deferred-antagonism technique. Seventeen subjects possessed inhibitory strains on at least one skin site. Of the Micrococcaceae tested, 20.6% were inhibitory. Of the aerobic diphtheroids, more strains were sensitive (19.2%) than inhibitory (5.1%). Inhibitory Micrococcaceae and aerobic diphtheroids revealed intergeneric and intrageneric inhibition. Antagonistic propionibacteria were found (7.4%) but no sensitive strains. Several Micrococcaceae were specifically antagonistic towards only one strain isolated from the same skin site; no sensitive strains from other skin sites or other subjects were found. Quantitative sampling showed that only 17.5% of the inhibitory strains clearly dominated their skin sites numerically.

Adult↗
PubMed1976

The relevance of growth rates in urine to the pathogenesis of urinary-tract infections due to Micrococcus subgroup 3 (Staphylococcus saprophyticus biotype 3).

A novobiocin-resistant "biotype" of Micrococcus subgroup 3 (Staphylococcus saprophyticus) is known to be a primary pathogen of the female urinary tract and to cause infections as severe as those produced by Escherichia coli. The growth characteristics of this virulent biotype were compared in vitro with those of other Micrococcaceae and of E. coli to determine whether rapid growth explains the virulence of the biotype. Nutrient broth was shown to have growth-supporting qualities that differed from those of urine and it was therefore unsuitable for these studies. In urine, the virulent biotype grew more slowly, had a longer lag period, and reached much lower final viable counts than did Escherichia coli. Surprisingly, the virulent biotype also grew more slowly and reached a lower final viable count than did several other Micrococcaceae isolated from the urinary tract of healthy women. Urine from women who had suffered a recent infection with the virulent biotype had growth-supporting properties similar to those of urine from healthy women. Experiments with filtered forestream urine suggested that urethral secretions do not contain a factor determining growth rates of this organism in urine. The possibility that virulent strains adapt to rapid growth in urine was excluded. No evidence was obtained that the virulent biotype inhibits the growth of other Micrococcaceae in urine. Ability to grow rapidly in urine does not therefore explain the virulence of novobiocin-resistant strains of subgroup-3 micrococci.

Adult↗