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PubMed1966

[A case of heart injury].

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Adult↗

PubMed1968

[The formation of an interventricular shunt as late as complication of a heart injury].

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Adult↗

PubMed1963

[ON THE PATHOLOGICAL ANATOMY OF HEART INJURIES].

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Azerbaijan↗

PubMedDate not supplied

The abdominal compartment syndrome as a consequence of penetrating heart injury.

The abdominal compartment syndrome results from gradual increase of intra-abdominal pressure and affects many bodily systems. It is usually associated with abdominal trauma or surgery. We report the first case of this syndrome developing after a penetrating wound to the chest and heart.

Abdomen↗

PubMedDate not supplied

[Heart injuries involving 3 chambers].

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Adult↗

PubMed1986

[18 years after a penetrating heart injury: follow-up].

This report presents a case of penetrating chest trauma leading to pericardial tamponade and ventricular septum defect successfully resuscitated by surgery in 1968 in an 11-year-old boy. 10 years later a first-size calcified hematoma was removed. However the 30% left to right intracardiac shunt flow as established by complete heart catheterization prior to surgery was not corrected. Non-invasive methods were used to assess cardiac function 18 years after the initial event including physical examination, chest X-ray, thoracic computer tomography, ECG, 24 h Holter monitoring and exercise testing, combined with myocardial scintigraphy and radionuclide angiography and echocardiographic techniques using the transthoracic and the transoesophageal approach. The diagnostic value of echocardiographic examinations is emphasized with special reference to contrast- and Doppler-echocardiography including the color-coded Doppler-flow-imaging technique.

Adult↗

PubMed2006

[Grafting of aortic valve and ascending aorta in the patient after heart injury and aortal commissurotomy].

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Aneurysm, Ruptured↗

PubMed2006

[Cellular mechanism of heart injury in the early stage of crush injury in rats].

OBJECTIVE: To study cellular mechanism of cardiomyocytes injury in the early stage of crush injury by observing some effects of crush injury rat sera on cultured neonatal rat cardiomyocytes. METHODS: One to three days old neonatal rat cardiomyocytes were cultured in vitro and some effects of crush injury rat sera on beating rate, cell surface area, total protein content, 3H-Leu incorporation, intracellular calcium concentration ([Ca2+]i) and Fos protein expression were observed in cultured rat cardiomyocytes. RESULTS: Compared with normal rat serum group, crush injury rat sera decreased beating rate(beats/min) of cardiomyocytes from 88.3 to 26.4, cell surface area, total protein content, 3H-Leu incorporation, [Ca2+]i (nmol/L) and PI of Fos protein expression were increased. CONCLUSION: Crush injury rat sera suppress cell beating, increase intracellular calcium, induce Fos protein synthesis and cause cell hypertrophy, which may cause cardiac injury in the early stage of rush injury.

Animals↗

PubMedDate not supplied