[Oxidation-reduction potential of soft tissues of the fetal head in labor complicated by intrauterine hypoxia and late toxicosis of pregnancy].
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Female↗
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Results for “OXIDATION REDUCTION”
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On the inter-relationship between glucagon action, the oxidation-reduction state of pyridine nucleotides, and calcium retention by rat liver mitochondria.
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Animals↗
An investigation of the oxidation-reduction potential and of the effect of oxygen on the germination and outgrowth of Clostridium butyricum spores, using platinum electrodes.
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Carbon Dioxide↗
[Condition of oxidation-reduction processes in patients with chronic osteomyelitis of the jaw and phlegmons of the maxillofacial area in connection with surgical intervention and anesthesia].
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Adult↗
[Role of oxidative-reductive processes in recovery of the functional activity of the thyroid gland in prolonged crush syndrome].
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Acute Kidney Injury↗
Studies on the oxidation-reduction potentials of heme proteins. VI. Human hemoglobin treated with various sulfhydryl reagents.
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Amides↗
[Glycolytic and oxidation-reduction metabolism in rat liver in chronic ethanol intoxication. Effect of the administration of nucleotide coenzymes].
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Animals↗
Oxidation-reduction potential dependence of the interaction of cytochromes, bacteriochlorophyll and carotenoids at 77 degrees K in chromatophores of Chromatium D and Rhodopseudomonas gelatinosa.
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Bacterial Chromatophores↗
Determination of the oxidation-reduction potential of the bound iron-sulphur proteins of the primary electron acceptor complex of photosystem I in spinach chloroplasts.
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Chlorophyll↗
Oxidation-reduction potentials of molybdenum, flavin, and iron-sulfur centers in milk xanthine oxidase: variation with pH.
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Animals↗
Anaerobic oxidation-reduction titrations of human ceruloplasmin. Evidence for diamagnetic electron-acceptors in the protein.
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Anaerobiosis↗
Electron-paramagnetic-resonance studies on nitrogenase. Investigation of the oxidation-reduction behaviour of azoferredoxin and molybdoferredoxin with potentiometric and rapid-freeze techniques.
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Adenosine Triphosphate↗
[Stimulation of the oxidative-reductive enzymes of the corneal system in traumatic lesions (an experimental study)].
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Animals↗
[The status of ascorbate oxidation-reduction system of blood in patients with chronic pancreatitis throughout parenteral nutrition].
There was conducted operative treatment of patients with complicated chronic pancreatitis. Intravenous infusion of aminoacid cocktail with enhanced content of glutamine, methionine with the selenium addition was applied in the complex of therapy. Under the influence of modified cocktail during 3 days there was observed more complete restoration of the ascorbic acid (AA) level and lowering of her oxidated forms content. This effect became more potent in addition of AA (0.75 per one infusion).
Adult↗
Energy dependence of oxidation-reduction potentials of the b and c cytochromes in beef heart submitochondrial particles.
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Adenosine Triphosphate↗
Binding and oxidation-reduction of monoamine oxidase-type 8alpha-(S-peptidyl) flavins with Azotobacter (Shethna) flavodoxin.
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Azotobacter↗
[Rhythms of slow-wave sleep and wakefulness in fluctuations of the potential of the oxidative-reductive status of the cerebral cortex].
A method of recording slow ROSP's changes of the brain in chronic animals was used. It has been shown that wakefulness was accompanied by quasisinusoidal oscillations of ROSP (periods--several seconds). During slow sleep, oscillations of ROSP became more complicated and their periods were longer. It is suggested that transitions from sleep to wakefulness and vice versa are connected with removal of maximums of oxidative metabolism tension between biochemical systems characterized by different rhythms of self-regulation. Rhythmic oscillations of ROSP reveal the possibility of separate functional system of the ROSP of the brain cortex to synchronize their oscillations in the brain tissue (biochemical synergism).
Animals↗
[Intracellular oxidative-reductive processes in tissues in hyperthermia].
Experiments were conducted on male albino rats to study some intracellular metabolites (lactate, pyruvate, malate, glutamate, alpha-ketoglutarate, ammonia) and the redox process in the tissues of the liver, kidneys, myocardium, and skeletal muscles during hyperthermia (40 degrees C for one hour and 45 degrees C for one hour). Changes of the metabolite content and shifts in the redox process in the direction of oxidation in the liver and kidneys at both levels of hyperthermia are evidence of the development of tissue hypoxia of circulatory character in these organs. The mitochondrial NAD/NADH ratio in the myocardium reduced in moderate hyperthermia and increased during a heat stroke. There were no signs of cellular hypoxia in the skeletal muscles. It is concluded on basis of the results that changes of the blood flow in the organs play the leading role in the origin of thermal hypoxia.
Animals↗