[Use of immunosuppressors in chronic inflammatory intestinal diseases].
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A study was made of 256 young children suffering from gastroenterocolitis, dyspepsia, food poisoning for the presence of conditionally pathogenic representatives of the Enterobacteriaceae family. Klebsiellae were isolated in 39.8% of the patients and in 22.3% of 67 healthy children. A study was made of morphological, cultural-biochemical properties and the antigenic structure in 308 strains of the Klebsiella denus. Capsules were detected by microscopic examination. Biochemical properties were determined after Kaufman, and some of the cultures proved to differ from the typical biochemical pattern described by this author. K-antigen was found in 185 klebsiella strains; of this number 99 strains belonged to Kl. pneumoniae and 86--to Kl. aerogenes. Fimbria were revealed in some of the Kl. pneumoniae and Kl. aerogenes strains. Bacteriocines were detected in 103 of 206 strains (by Abbot and Shennon's method).
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Surveillance of general outbreaks of infectious gastroenteritis was introduced in 1998 by the Food Safety Authority of Ireland (FSAI), in co-operation with the eight health boards. A total of 67 general outbreaks of gastroenteritis in Ireland were reported to the FSAI in 1998 and 1999. Over 1900 people were ill as a result of these outbreaks. Four percent required hospitalisation and there were two deaths. The duration of the outbreaks varied between one day and 38 days. Salmonellae (44%) and small round structured viruses (SRSV) (12%) were the most commonly reported pathogens. In 25% of the outbreaks the aetiology was unknown. The commonest settings were restaurants, hotels and take-aways, which accounted for 45% (30/67) of all outbreaks. Sixteen percent of all outbreaks occurred in hospitals and residential institutions. Over half of the outbreaks were reported to be foodborne, 63% of which were due to various serotypes of Salmonella enterica. Eggs were implicated as the vehicle of infection in 13% of all outbreaks. An infected food handler was identified in almost one third of outbreaks, although it could not be established if this had contributed directly to the outbreak.
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