Search PubMed⌕ Search

SEARCH · Search PubMed

Results for “RINDERPEST”

Search indexed PubMed citations on genomics, clinical trials, systematic reviews and public health. Explore titles, authors and supplied subject terms, then open the PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 847 records · Page 47Linked to original sources
PubMed1990

Genetic comparison of ovine and bovine pestiviruses.

Viral RNA oligonucleotide fingerprinting was used to compare genetic relationship among pestiviruses originating from ovine or bovine host species. Ovine pestiviruses, including reference border disease virus and 2 border disease isolates originating from natural pestivirus infections of sheep, appeared to have a more distant genetic relationship among themselves than with certain bovine pestiviruses. A closer genetic relatedness was evident between border disease virus and 3 noncytopathic bovine pestiviruses, including Draper bovine viral diarrhea virus (BVDV), a BVDV isolate that originated from aborted bovine fetuses, and a virus that was isolated from the serum of a calf that had a chronic BVDV infection. Four noncytopathic bovine viruses, including Draper BVDV and 3 field isolates, were closely related. Reference Oregon C24V BVDV, a cytopathic virus, was closely related to only 1 of the 7 noncytopathic viruses in this study.

Animals↗
PubMed1991

Aerobic bacteria from nasal discharge of goats suffering from clinical PPR: isolation and identification.

Bacteriological isolation of aerobic bacteria from nasal discharge of achondroplastic West African dwarf goats showing clinical signs of peste des petits ruminants (PPR), and the laboratory procedures carried out for the identification of the isolates are described. From a total of forty nasal swabs obtained seventy five isolates were recovered. The percentage frequency of isolation of different organisms in descending order of occurrence is as follows: Staphylococcus 44.0%; Streptococcus 22.67%; Neisseria 12.00%; Pasteurella 10.67%; Pseudomonas 4.00%; Proteus 4.00% and Corynebacterium 1.33%. A further break-down using biochemical tests revealed that Staphylococcus aureus accounted for 30.67% of the total bacteria isolates; Staphylococcus epidermidis 13.33%; Streptococcus viridans 18.67%; Streptococcus pyogenes 4.00%; Neisseria catarrhalis 12.00%; Proteus mirabilis 2.67%; Proteus rettgeri 1.33%; Pseudomonas aeruginosa 5.33%; Pasteurella multocida 4.00%; Pasteurella haemolytica 6.67% and Corynebacterium pyogenes 1.33%.

Animals↗
PubMed1990

Pathogenicity of attenuated peste des petits ruminants virus in sheep and goats.

Progressive loss of virulence for goat kids was noticed when peste des petits ruminants (PPR) virus was passaged in Vero cells. While goats inoculated with the 60th passage suffered from the clinical PPR disease and mortality, goats inoculated with the 80th passage did not show any sign of the disease. If the progressive loss of virulence of the virus with passage continues, it will not be long before a homologous PPR vaccine will be obtained at the National Veterinary Institute, Vom.

Animals↗
PubMed1989

Management of clinical cases of peste des petits ruminants (PPR) disease in goats.

Clinical cases of Peste des petits ruminants (PPR) disease in goats were treated symptomatically. Broad spectrum antibiotics, intestinal sedatives and fluid therapy were used for the treatment of pneumonia, diarrhoea and the restoration of the body fluid ionic balance. Good feeding and nursing in warm, draught-free pens are necessary. Lemon fruit and Citrus aurantium proved to be effective for the treatment of the orf-like labial scabs. This treatment raised the survival rate of goats by 13.3%.

Animals↗
PubMed1989

Correlation of humoral immune response with clinical presentation, pulmonary lesions and mortality patterns of goats experimentally infected with peste des petits ruminants virus.

The patterns of humoral immune response, measured by enzyme-linked immunosorbent assay and the development of clinical signs, pulmonary lesions and mortality patterns were studied in goats experimentally infected with peste des petits ruminants virus (PPRV) or Kata virus. It was possible to classify the animals into high and low responders according to the pattern of humoral immune response. The high responders had a prolonged, chronic infection characterized by a giant cell pneumonia which was sometimes complicated by bronchopneumonia. Such animals survived beyond 1 month. The low immune responders had a subacute or acute clinical presentation. In the former case, the animals developed an uncomplicated giant cell pneumonia and died within 1 month of infection. Those with the acute symptoms developed pulmonary congestion and oedema and succumbed to the infection in 1 week. The clinical and pathological response of goats to PPRV infection, and ultimately the outcome of the infection correlated well with the ability of the goats to mount specific antibodies to PPRV.

Animals↗
PubMed1989

Fluid, electrolyte and haematological changes in experimental peste des petits ruminants virus infection in West African dwarf goats.

The changes in plasma volume, blood volume, erythrocyte indices, leucocyte indices and serum biochemical values during experimental pestes des petits ruminants (PPR) virus infection of West African dwarf goats (Fouta djallon) were investigated. There was a progressive decline of the plasma volume and serum sodium and potassium values in all the infected goats as from day 5 post-infection and this became quite marked between days 10 and 23 post-infection. The fall was severest in those animals which had diarrhoea. There were no significant changes in the chloride, bicarbonate, phosphate, urea, total protein and albumin values of the goats during the course of the diseases. Haemoconcentration and apparent polycythaemia in the early and terminal stages of the disease were associated with severe dehydration which characterized the infection. There was initial neutrophilic leucocytosis during the phase of fever followed by marked lymphopaenic leucopaenia which progressed terminally in most of the infected goats. The destructive effect of the virus on lymphocytes is suggested as a cause of lymphopaenia.

Animals↗