Search PubMed⌕ Search

SEARCH · Search PubMed

Results for “MOUTH ABNORMALITIES”

Search indexed PubMed citations on genomics, clinical trials, systematic reviews and public health. Explore titles, authors and supplied subject terms, then open the PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 847 records · Page 47Linked to original sources

[Population and familial incidence of orofacial anomalies considered microforms of cleft lip and palate].

This paper presents the results and analysis of the distribution frequencies of orofacial features in a population and among the relatives of 325 probands with cleft lip and palate. Our results indicate that some of the features (diastema, nostril asymmetry and others) are more frequent in the relatives of children with clefting than in a general population. These studies suggest that several minor clinical conditions in parents may be important for medico-genetical consultations.

Adolescent↗

Chromosomal considerations--application of modern cytogenetics.

Attention is directed towards the use of new techniques in cytogenetics. C-, G-, Q-, and R-banding methods are briefly discussed and illustrated. Methods of sister chromatid differential staining and silver staining for demonstration of nucleolus organizing regions are reviewed and their application in medical cytogenetics is pointed out. Frequency of chromosomal aberration and the importance of the orofacial area in diagnosis of chromosomal syndromes are the bases for the argument for the usefulness of cytogenetics in the field of stomatology-dentistry.

Bromodeoxyuridine↗

Origin of hydrocortisone induced orofacial clefts in the chick embryo.

The growth and fusion of facial processes were studied in White Leghorn chicken embryos treated with hydrocortisone. This treatment results in complete bilateral cleft beak in 80-100% of cases. The frontonasal complex of the experimental embryos exhibited marked hypoplasia prior to cleft manifestation. During the critical period for cleft beak formation, the maximum mitotic activity was concentrated in the medial nasal processes, which thus became vulnerable to the mitosis inhibiting effect of corticoids. Primary hypoplasia of the facial processes was documented as a causative factor of corticoid-induced cleft beak which is analogous to mammalian cleft lip/palate.

Animals↗