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At least 811 records · Page 45Linked to original sources
PubMed1976

Facial spasm.

There are many varieties of abnormal facial movement. This paper discusses hemifacial spasm and blepharospasm in particular. A technique found useful for the symptomatic relief of idiopathic hemifacial spasm is described, and experience with its use on 15 patients also described. Experiences in the operative treatment of 6 patients with essential blepharospasm are recounted.

Blepharospasm↗
PubMed2003

Cough gastric pressure and maximum expiratory mouth pressure in humans.

Maximal expiratory mouth pressure is a well established test that is used to assess expiratory muscle strength. However, low values are difficult to interpret, as they may result from technical difficulties in performing the test, particularly in patients with facial muscle weakness or bulbar dysfunction. We hypothesized that measuring the gastric pressure during a cough, a natural maneuver recruiting the expiratory muscles, might prove to be a useful additional test in the assessment of expiratory muscle function. Mouth expiratory and cough gastric pressures were measured in 99 healthy volunteers to obtain normal values and in 293 patients referred for respiratory muscle assessment to compare the two measurements. Between-occasion within-subject coefficient of variation, assessed in 24 healthy volunteers, was 10.3% for mouth pressure and 6.9% for cough. Mean +/- SD cough gastric pressure for normal males was 214.4 +/- 42.2 and 165.1 +/- 34.8 cm H2O for females. In 171 patients deemed weak by a low mouth expiratory pressure, 42% had a normal cough gastric pressure. In 105 patients deemed weak by a low cough gastric pressure, 5.7% had a normal expiratory mouth pressure. Low maximal expiratory mouth pressures do not always indicate expiratory muscle weakness. Cough gastric pressure provides a useful complementary test for the assessment of expiratory muscle strength.

Case-Control Studies↗
PubMed1975

[Microcoagulation in the treatment of facial spasm (author's transl)].

A method for the surgical treatment of facial spasm is described. The nerve trunk is exposed in the mastoid region and the sheath is opened. With a bipolar electrode, microspots are burnt spirally round the nerve trunk and also at a few places in the centre of the nerve after pulling the bundles apart. Three cases are reported and assessed in comparison with the methods of Miehlke, Fisch, Rosemann and Schaupp. The more rapid and easier delineation of the nerve trunk in the event of a recurrence are considered an advantage compared with the extratemporal methods.

Electrocoagulation↗
PubMed1989

[Myogenic facial pain].

Myogenic prosopalgia is one of the forms virtually unfamiliar to practitioners. In a majority of cases it belongs to the category of pain-related myofascial dysfunctions present in about 10% of patients with facial pains. This work deals with etiology, pathogenesis, clinical features, diagnosis and treatment of the disorder.

Diagnosis, Differential↗
PubMed1979

Progressive extrinsic ophthalmoplegia with peripheral neuropathy and storage of muscle glycogen.

A case of progressive extrinsic ophthalmoplegia associated with peripheral neuropathy and hypertriglyceridemia type IV is described. Motor and sensory conduction velocities of the spinal nerves were severely decreased, while the EMG of the facial muscles was more suggested of a myopathic disorder. Electron microscopic study of biopsies of biceps and peroneus brevis muscles disclosed many ragged red fibers, mainly type I, which contained typical abnormal mitochondria. Other fibers, all type II, contained increased amounts of glycogen between myofibrils or beneath the sarcolemmal membrane, but the mitochondria were normal. These fibers were more abundant in the peroneus brevis than in the biceps muscle. Nerve biopsy revealed marked loss of myelinated fibers, but neither mitochondrial changes nor glycogen storages were evident in Schwann's cells. Biochemical investigations confirmed the increased amount of glycogen in both muscle biopsies and revealed a decrease of guanilcyclase. Phosphorylase, phosphorylase b kinase, adenilcyclase, and carnitine concentrations were all normal. The pathogenesis of this syndrome is discussed and the relationship between mitochondrial abnormalities and glycogen accumulation in muscle tissue are considered.

Adult↗
PubMed2005

Facial expressions of emotion reveal neuroendocrine and cardiovascular stress responses.

BACKGROUND: The classic conception of stress involves undifferentiated negative affect and corresponding biological reactivity. The present study hypothesized a new conception that disaggregates stress into emotion-specific, contrasting patterns of biological response. METHODS: Ninety-two healthy adults engaged in stress-challenge tasks, during which cardiovascular responses, hypothalamic-pituitary-adrenocortical (HPA) axis responses (i.e., cortisol), emotional expressions (i.e., facial muscle movements), and subjective emotional experience (self-reported) were assessed. RESULTS: Pronounced individual differences emerged in specific emotional responses to the stressors. Analyses of facial expressions revealed that the more fear individuals displayed in response to the stressors, the higher their cardiovascular and cortisol responses to stress. By contrast, the more anger and disgust (indignation) individuals displayed in response to the same stressors, the lower their cortisol levels and cardiovascular responses. Individual differences in optimistic appraisals appeared to mediate these correlated patterns. CONCLUSIONS: Facial expressions of emotion signal biological responses to stress. Fear expressions signal elevated cortisol and cardiovascular reactivity; anger and disgust signal attenuated cortisol and cardiovascular reactivity, patterns that implicate individual differences in stress appraisals. Rather than conceptualizing stress as generalized negative affect, studies can be informed by this emotion-specific approach to stress responses.

Adult↗
PubMed2002

Testosterone treatment attenuates the effects of facial nerve transection on glial fibrillary acidic protein (GFAP) levels in the hamster facial motor nucleus.

Testosterone propionate (TP) administration coincident with facial nerve injury accelerates the recovery rate from facial muscle paralysis in the hamster. One mechanism by which TP could augment peripheral nerve regeneration is through glial fibrillary acidic protein (GFAP) regulation in the facial motor nucleus. In a previous study, axotomy alone induces increases in GFAP mRNA. with TP significantly attenuating the axotomy-induced increases in GFAP mRNA. In the present study, immunoblotting techniques were used to extend our previous GFAP mRNA studies to the protein level. Castrated male hamsters were subjected to a right facial nerve transection, with half of the animals receiving subcutaneous implants of 100% crystalline TP. The left facial motor nucleus of each animal served as an internal control. Postoperative survival times include Days 4, 7, and 14. In non-TP-treated animals, facial nerve transections alone increased GFAP levels at all time points, relative to internal controls. As previously observed at the mRNA level, TP treatment attenuated but did not eliminate the axotomy-induced increase in GFAP levels at all time points tested. These results suggest that the regulatory actions of gonadal steroids on GFAP expression manifested in parallel at the mRNA/protein levels.

Animals↗
PubMed1983

Unilateral scapulohumeral muscular atrophy.

A peculiar form of chronic spinal muscular atrophy is described, 5 unrelated young males developed muscle wasting of the left shoulder and upper arm region, mainly in the distribution of myotomes C5 and C6. This muscle atrophy remained confined to the primary site for 13-18 years, being followed in 4 cases by a mild muscle atrophy of the right upper arm or or the left forearm and hand and in 1 case by atrophy of the legs and the facial muscles. On EMG examination, muscle involvement was always found to be more widespread than on clinical examination. The repeated and extensive EMG examinations gave results typical of an anterior horn cell disease. Pyramidal signs were absent. During the observation period of 21-40 years the course was benign and at least partial working capacity was retained until retiring age.

Aged↗
PubMed1992

[Facial paralysis and chicken-pox].

Ten days after chicken-pox, a 24-year old man presented with a right facial palsy without meningitis. Albumino-cytologic dissociation in CSF--not observed in two other published cases--suggests an immune-mediated process. The patient was treated by prednisone and aciclovir. The upper facial muscles remained paretic 2 months after onset.

Acyclovir↗