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PubMed1994

The inhibitory action of lead on mechanical responses of the proventricular smooth muscle in the chick.

The purpose of the present experiments was to examine the mechanism of the proventricular dilatation caused by lead in the isolated vagus nerve-proventricular smooth muscle preparation of the chick. Lead caused dose- and time-dependent inhibition of contractions induced by vagal stimulation, transmural stimulation and externally applied acetylcholine (ACh). Vagally evoked contraction was much more sensitive to the inhibitory action of lead than the contractile response to ACh. The lower the frequency of transmural stimulation, or the lower the concentration of ACh was applied, the greater the inhibitory action of lead on the evoked smooth muscle contraction. The results suggest that proventricular impaction occurring in lead poisoning results from the pre- and post-synaptic inhibition of the vagus nerve-smooth muscle transmission.

Animals↗
PubMedDate not supplied

Proventriculitis and ventriculitis associated with zygomycosis in ostrich chicks.

This report describes eight cases of proventriculitis and ventriculitis in ostrich chicks less than 2 months old. Clinical signs included acute onset of lethargy and anorexia in three cases, and chronic weight loss with lethargy and anorexia in four cases; no history was available in one case. There was limited antibiotic therapy in two cases; a third case was treated for giardiasis. Concurrent bacterial, yeast, and viral infections were common. Lymphoid depletion and/or necrosis of bursa, thymus, and spleen suggested severe immune challenge or immunosuppression in many cases. Histologically, there was severe ulcerative proventriculitis and ventriculitis with intralesional fungal hyphae. In two chicks with granulomatous pneumonia, similar fungal hyphae were also observed in the lung. Fungal hyphae were rarely septate, with irregular, non-parallel walls, and ranged in diameter from 7 to 20 microns. Occasional globoid distentions of the hyphae were present. Fungi were identified morphologically as species in the Zygomycetes class; in one case a Mucor sp. was cultured. Zygomycetes appear to be potentially serious opportunistic pathogens of ostrich chicks.

Animals↗
PubMed1993

Adverse drug reaction to metoclopramide hydrochloride in a macaw with proventricular dilatation syndrome.

A 4-year-old female blue and gold macaw (Ara ararauna) with a history of chronic vomiting was treated with metoclopramide hydrochloride. After the second treatment, ataxia, torticollis, and opisthotonos became evident. These signs resolved with the administration of diphenhydramine hydrochloride. Despite supportive care, the bird died several days later. Histologic lesions were suggestive of proventricular dilatation syndrome.

Animals↗
PubMedDate not supplied

Multiple intussusceptions in a juvenile rhea (Rhea americana) with proventricular impaction.

Multiple intussusceptions of the small intestine were identified in a 4-mo-old rhea (Rhea americana) that died acutely after chronically poor growth. The chick was one of a group of 12 chicks that exhibited musculoskeletal deformities, poor growth, and subsequent death. Gross necropsy findings of this chick revealed proventricular impaction by sticks and stones accompanied by multiple intussusceptions of the small intestine. This finding identifies a disease process that may afflict ratites when raised for production and that may complicate treatment of proventricular impaction.

Abnormalities, Multiple↗
PubMed1997

[Proventriculitis in domestic fowl (Gallus gallus var. dom. L., 1758) caused by megabacteria].

Three cases of a disease similar to going light syndrome in budgerigars in a flock of young laying hens in Turkey are reported. In all cases megabacteria in association with E. coli could be isolated. The causing agent appeared to be large, rod-shaped, gram-positive or gram-labil, and catalase test positive bacteria with cytochromoxydase activity. Subcultivation of these bacteria could not be achieved.

Animals↗
PubMedDate not supplied

Effects of biogenic amines in broiler chickens.

Biogenic amines in spoiled animal by-product feeds have been implicated in causing poor performance and intestinal lesions in broilers. This study was designed to determine if biogenic amines, at the concentrations found in animal by-product meals, would reduce performance in broilers or cause lesions. Twelve treatments were used in a 2 x 6 factorial arrangement with the main effects being either a corn-soybean meal diet or a corn-soybean meal diet with 10% animal by-products added and either no amines added or added levels of phenylethylamine (4.8 mg/kg), putrescine (49 mg/kg), cadaverine (107 mg/kg), histamine (131 mg/kg), or a combination of all these amines. Levels of biogenic amines used in this study simulated those found in areas with reported problems attributed to biogenic amines. Broilers were monitored for performance, gross lesions, and histologic evidence of lesions at 2, 4, and 6 wk. No consistent effects were observed on performance, and by the conclusion of the trial, no statistical differences were noted in the performance of any of the treatments. No gross lesions were observed on a consistent basis in any of the treatments. Histopathology was likewise unremarkable. On the basis of this study, it would appear that these four biogenic amines, at levels detected in the United States, do not pose a serious health concern for the broiler industry.

Animal Feed↗