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At least 73 records · Page 4Linked to original sources
PubMed1977

Use of 1alpha-hydroxycholecalciferol in the prevention of bovine parturient paresis.

A synthetic biologically active derivative of vitamin D (350 microgram of 1alpha-hydroxycholecalciferol [1alpha(OH)CC]) was injected into 2 nonlactating 7-year-old Israeli-Friesian cows. Plasma calcium values increased after 24 hours, peaked at 48 hours, and returned to base-line values 120 hours after injection. An injection of 350 microgram of 1alpha(OH)CC was given to 23 parturient-paresis-prone Israeli-Friesian cows from 7 days to 6 hours prepartum; 13 cows were injected once, 6 were injected twice, and 4 were injected 3 times, all at 48-hour intervals. Parturient-paresis-prone cows (n = 23) of the same breed were used as controls. Within 0 to 36 hours postpartum, plasma calcium concentrations were found to be higher in cows injected with 1alpha(OH)CC than in the control cows. The increase was highly significant (P less than 0.01) in cows injected at least twice. None of the cows injected with 1alpha(OH)CC, within 72 to 24 hours prior to calving developed parturient paresis; but 9 of 23 control cows developed parturient paresis. Prior to calving, none of the injected cows developed hypercalcemia and there was no local or systemic clinically detectable signs of toxiosis. When given at the right time prepartum, 1alpha(OH)CC is considered to be an improvement over previous methods of preventing bovine parturient paresis.

Animals↗
PubMedDate not supplied

[Differential diagnosis of unilateral paresis and paralysis of the larynx].

Differential diagnosis of laryngeal paresis and paralysis using electron glottography (EGG) was tried in 57 patients and 30 healthy controls. Measurements were made of speed quotient (SQ) and open quotient (OQ) at piano and forte. It was found that in patients and controls SQ and OQ were not similar. SQ and OQ differed also in paralysis and paresis. Tonicity of the inner laryngeal muscles seems to decline more in paralysis than in paresis. Aerodynamic properties of the larynx are damaged more in paralysis and remain unchanged in paresis. Reduced SQ and high OQ at forte indicate unilateral laryngeal paralysis. OQ should be measured both at piano and forte. Thus, EGG is an effective procedure for differential diagnosis of laryngeal paralysis and paresis. It is especially convenient when laryngostroboscopy is problematic.

Diagnosis, Differential↗
PubMed1999

Passive dynamics of the knee joint in healthy children and children affected by spastic paresis.

OBJECTIVE: The purposes of this study were (1) to evaluate how changes in biomechanical parameters affect segment dynamics in children and (2) to determine whether the biomechanical parameters were changed in children with spastic paresis. DESIGN: In vivo measurements were collected of knee viscoelastic properties. Background. It is unknown if the inertial and viscoelastic properties of a human growing limb should be considered in motor performance. Also unclear are whether changes in passive dynamics might be responsible for abnormal control in human spastic paresis. METHOD: Small oscillation techniques were used to measure moment of inertia of lower leg, stiffness and viscous damping of the knee joint. Eighty seven healthy children and 32 children with spastic paresis participated. RESULTS: Moment of inertia, stiffness and the damping changed with the fifth power of child's height. Dynamic equation of motion parameters were a constant, independent of the child's height. Passive viscoelastic parameters were not changed in spastic patients. CONCLUSIONS: Inertial and viscoelastic properties of a growing limb segment should not be considered in motor performance. Passive viscoelastic properties were not changed in patients with spastic paresis and, therefore, cannot be responsible for abnormal control in human spastic paresis. RELEVANCE: There is no need to adapt control patterns in children (ages 6-18). Passive viscoelastic parameters cannot be used as a descriptor of spasticity.

Adolescent↗
PubMed2007

[Recurrent facial paresis with facial neurinoma].

PATIENT: A clinical case of a 49-year-old man is described who presented with a right facial paresis for 4 weeks. The patient had undergone a decompression of facial nerve and a radical mastoidectomy elsewhere in 1998 because of a facial paresis and acute mastoiditis. In the following years a complete right facial paresis occurred several times improving with prednisolone. In addition to topognostic examinations a high resolution computed tomography of the temporal bone was made. CT showed a mass in the right tympanic cavity with close contact to the ossicular chain. Diagnostic tympanotomy disclosed a tumor of the tympanic segment of the facial nerve. Pathological examination indicated a diagnosis of neurinoma with an Antoni B architecture. In a second operation the tumor was resected totally and a facial nerve reconstruction was performed by a greater auricular nerve interposition graft. Seven month postoperative beginning nerve reinnervation was seen proceeding continually until the control examination after 1 1/2 year. DISCUSSION: Facial neurinoma are a rare course of facial paresis. There are no specific symptoms. That's why the diagnosis is difficult. But it is necessary to think of with differential diagnosis of facial paresis.

Cranial Nerve Neoplasms↗
PubMed1992

Transcranial magnetic stimulation in pontine infarction: correlation to degree of paresis.

Transcranial magnetic stimulation was performed in 20 patients with pontine infarction who had initially some degree of hemiparesis. Only patients with a well defined lesion on magnetic resonance imaging that was appropriate for the neurological signs were included. Recordings were made from the abductor pollicis brevis muscle (APB) bilaterally. The degree of hand paresis was estimated clinically and related to the following parameters: central motor conduction time (CMCT), interside latency difference of total latency, and amplitude ratio of affected to unaffected side. Increasing degree of paresis was associated with increasing latency parameters and decreasing amplitude ratio. In the four patients with severe paresis a low amplitude response could be evoked and CMCT was delayed by up to 10 ms. When the paresis had resolved at the time of transcranial magnetic stimulation CMCT was normal. However, amplitude ratio was less than 100% in all but one patient, with most of the values ranging between 40% and 60%, which indicates a subclinical pyramidal tract lesion. Median nerve sensory evoked potentials (SEP) and related interside latency difference to amplitude ratio N20/P25 were also recorded. In contrast to TCMS, decreased amplitude ratio of SEP was not associated with delayed latency. Clinically, the mild degree of and good recovery from paresis in ventral pontine infarction was remarkable.

Adult↗
PubMed2005

Isolated dysarthria-facial paresis syndrome: a rare clinical entity which is usually overlooked.

BACKGROUND AND AIMS: The aim of the study is to search the lesion localization of the pure isolated facial paresis-dysarthria syndrome in patients who were admitted to our neurology clinic in a prospective study. METHODS: Over a period of six years, the patients who had no prominent sensorimotor dysfunction were examined by neurologists and underwent computerized tomography (CT) and/or magnetic resonance imaging (MRI). RESULTS: Eleven patients out of more than 2000 had the aforementioned clinical picture. Lacunar infarctions were identified at the corona radiata in nine patients, and at the internal capsule in two patients. As reported previously, facial paresis was usually mild and temporary. Six of our eleven patients were seen at the outpatient clinic one month later. Four of them had completely recovered and the other two had mild dsyarthria without any facial paresis. The other five could not be reached after leaving the hospital. CONCLUSIONS: Dysarthria-facial paresis is a rare clinical entity and possibly a variation of dysarthria-clumsy hand syndrome, and we suggest that pure facial paresis (FP) and pure dysarthria should be considered as very extreme examples of this syndrome.

Aged↗
PubMed2001

[Physical therapy in peripheral facial paresis].

Peripheral facial paresis is a serious clinical and social problem. The effect of physical therapy in 44 patients aged 13-80 was studied. Idiopathic peripheral facial paresis was the cause of mimic muscles dysfunction in 75% of our patients. Medical treatment was conducted according to the established program, consisting in applying combined physical, thermal, electrotherapeutic procedures, exercises and massage. All the patients were examined before physical therapy and after I and II series of procedures, based on Pietruski's evolution table of paresis scored in point and percent scale. Patients between age 13-44 demonstrated 47-50% mimic facial muscles efficiency before the treatment, after I series of procedure it amounted to 80-90% and after the II series to 90-100%, so all the symptoms of paresis disappeared. Similar values were scored by patients aged 45-64 years. Minimal percentage of improvement of mimic facial muscles (57%) was noticed in patients aged 65-80 years The increase of the score was evaluated in the studied group after two stages of treatment. It seems to be best in the younger patients. In the examined group the best improvement evaluated as regression of the symptoms was found in patients with idiopathic peripheral facial paresis. The process of improvement was very slow and not full among patients who additionally suffered from co-existing diseases. Patients with diabetes improved by 57%, with arterial hypertension by 53%, the patients with obesity only by 43% after two cycles of treatment.

Adolescent↗
PubMed2003

Correlation of facial nerve paresis and histopathological type of vestibular schwannoma.

The objective of the study was to evaluate the dependence of facial nerve paresis, as a symptom of cerebellopontine angle tumour, on the histopathological subtype of vestibular schwannoma, diagnosed from a post-operative histopathological examination. We retrospectively analysed 91 surgically treated patients with vestibular schwannoma. We studied the histopathological subtype and the preoperative condition of the facial nerve. The following WHO 2000 subtypes were distinguished: neurinoma cellular (51 cases), neurinoma conventional (23 cases), neurinoma ancient (11 cases), (other types: 2 neurofibroma and 2 ganglioneuroma). We analysed the dependence of facial nerve paresis on the histological subtype of tumours and their sizes. The analysis was based on the traditional classification: Antoni A (11 cases), Antoni B (12 cases) Antoni A/B (23 cases) and Antoni B/A (40 cases). 30 patients (30%) in the analysed group had paresis of the facial nerve preoperatively. Preoperative facial paresis occurred frequently in subtypes "cellular" and Antoni B, and rarely in subtypes conventional and Antoni A and B/A. In the small tumour cases (up to 20 mm), facial nerve paresis occurred frequently in subtypes cellular and conventional, as well as in Antoni A and A/B.

Facial Nerve↗
PubMed1976

[Treatment of intestinal paresis in children].

In the paper, a clinical experience with treatment of intestinal paresis in 545 children, aged from 1 day to 13 years, including 225 children operated upon for peritonitis is summarized. The authors differentiate 3 stages in the development of intestinal paresis depending on the degree of intensity of systemic and local disturbances. The employed methods of treatment in intestinal paresis were classified by the principle of their effect as 3 groups. Application of some or other method of the paresis therapy is determined by the stage of its development. In treatment of postoperative intestinal paresis a continuous peridural blockade is considered to be the method of choice. Utilization of the latter enabled the authors to reveal a number of postoperative complications: mechanic intestinal obstruction, incompetent anastomosis, etc.

Adolescent↗
PubMed2006

Lateralised vestibular hypofunction: canal paresis and handedness.

In this retrospective study it was found that 549 out of 1175 patients seen for vestibular assessment within an 8-year period within a district hospital service showed lateralised canal paresis as determined by caloric testing. It was found that there was a tendency for canal paresis, indicative of vestibular hypofunction, to be on the side of handedness. This bias showed statistical significance, (chi-squared, p < .005), for the patients overall, as well as for the 65 left-handers considered separately, (p < .05). Surprisingly, this form of bias does not appear to have been reported previously. The bias was shown most clearly in the largest of the groups defined by sex and handedness, namely that of female right-handers. In this group the prevalence of associated hearing loss was similar for canal paresis on either side. This allowed speculation as to the particular type of disorder to which those cases of canal paresis that were associated with the bias might be due, from which it seemed that migraine, rather than Menière's disease or vestibular neuronitis, was likely.

Aged↗
PubMed1998

Paresis and unusual electrocardiographic signs in a severely hypomagnesaemic, hypocalcaemic lactating bitch.

A severely hypocalcaemic, hypomagnesaemic lactating bitch exhibited clinical signs of pulmonary oedema, paresis, dementia, gastrointestinal ileus and urinary bladder atony. The total calcium, ionised calcium and magnesium levels were extremely low. The clinical picture was very different from the one typically encountered in canine lactation tetany, and instead resembled bovine postparturient paresis. Muscle tremors, rigidity and seizures were not part of the acute clinical picture, but rather atony, weakness and paresis. General muscle dysfunction probably resulted from the extremely low ionised calcium levels in combination with very low levels of magnesium and possibly potassium. Heart failure and atony of the urinary bladder and intestines were probably a result of the severe hypocalcaemia. The alteration in calcium to magnesium ratio may have depressed neuromuscular transmission, leading to paresis and atony. The unusual electrocardiogram possibly also resulted from abnormal magnesium and calcium cation levels.

Animals↗
PubMed2001

A diffusion-weighted MRI study of acute ischemic distal arm paresis.

OBJECTIVE: To study the site of the ischemic lesion, the underlying cause, and the prognosis of acute stroke with distal arm paresis. METHOD: The authors investigated 14 consecutive patients with acute distal arm paresis with a diagnostic stroke protocol and early MRI, including T2-weighted images, diffusion-weighted images (DWI), and perfusion-weighted images (PWI). Acute DWI lesions were shown on coregistered T2-weighted images for analysis of the exact anatomic lesion location. RESULTS: Patients showed a uniform (7/14), radial (3/14), or ulnar (4/14) distribution of hand paresis. In all cases, DWI identified small lesions located in the motor cortex. Topographic lesion analysis, which was correlated with the clinical deficit, showed lesions centered in the hand knob area (2/14), involving the lateral (6/14), medial (4/14), or both (2/14) borders of the hand knob. PWI (calculated time-to-peak maps) did not show a mismatch between the DWI lesion and the PWI lesion. In six patients, DWI and PWI lesions were identical in size and location; no definite perfusion deficit was seen in eight patients. In agreement with PWI, no patient showed clinical worsening, and six patients recovered completely within a week. Further investigations showed a potential source of embolus in 11 cases. CONCLUSIONS: Acute ischemic distal arm paresis is usually caused by a small cortical lesion in the motor hand cortex attributable to distal Rolandic artery obstruction without additional tissue at risk. These findings confirm the observed benign clinical course and its apparent main cause (artery-to-artery or cardiac embolism).

Acute Disease↗
PubMedDate not supplied

Head-shaking nystagmus: associations with canal paresis and hearing loss.

In 108 patients referred for caloric testing, head-shaking nystagmus (HSN) was noted in 37 electronystagmography (ENG) recordings. A canal paresis was found in 31 patients and a sensorineural or mixed hearing loss in one or both ears was found in 45 patients. Both HSN and canal paresis were insensitive predictors of either hearing loss or of each other. HSN was less sensitive as well as less specific than canal paresis for hearing loss. These data suggest that HSN is not as powerful a test as canal paresis in detecting lesions of the 8th nerve.

Adult↗
PubMed1990

Effect of subcutaneously released 24F-1,25-dihydroxyvitamin D3 on incidence of parturient paresis in dairy cows.

Twenty-one aged Jersey cows were fed a high calcium diet prior to parturition to predispose them to parturient paresis. Eleven cows were implanted subcutaneously with pellets containing 24F-1,25-dihydroxyvitamin D3 7 d before the expected date of parturition and thereafter at 7-d intervals until parturition. Ten cows were left untreated to serve as controls. Incidence of parturient paresis among control animals was 80% (8/10). Treatment reduced the incidence of parturient paresis to 9% (1/11). The mean observed plasma calcium concentration nadir of implanted cows was 6.61 +/- .40 mg/dl, which was significantly greater than the plasma calcium nadir of 4.45 +/- .39 mg/dl observed in the control cows. Two steers were implanted with and three nonpregnant, nonlactating cows received intramuscular injections of 24F-1,25 dihydroxyvitamin D3 to contrast circulating plasma concentrations achieved by the two routes. Intramuscular injection and implantation resulted in plasma 24F-1,25 dihydroxyvitamin D3 concentrations above 300 pg/ml for the first 48 h after administration. From d 4 until d 11 after administration, plasma concentration was maintained between 164 and 89 pg/ml in the implanted steers. Plasma concentration was undetectable 7 d after an intramuscular injection. These data indicate that, with refinement, sustained release of 24F-1,25 dihydroxyvitamin D3 can be successfully used to reduce the incidence of parturient paresis.

Animals↗
PubMed1992

[A case of general paresis with marked improvement of cerebral blood flow after antiluetic therapy--case report].

Although the incidence of neurosyphilis has declined dramatically after the World War II because of the development of antibiotics, it is recently increasing a little bit. There has been few reports on the neuroimaging of general paresis. We studied a fresh case of general paresis by X-ray CT (XCT), MRI and PET scans, and report the changes of neuroimages before and after the therapy. A 38-year-old man was admitted with disorientation, thought disorder and personality change. He also had insomnia and megalomania. There were neither obvious neurological signs nor particular changes of the XCT scan abnormalities on admission. His clinical symptoms were gradually worsened within three months, when the XCT and MRI scans showed moderate brain atrophy especially in the frontal and temporal lobes with mild dilatation of the lateral ventricles. In the PET study, the cerebral blood flow was decreased in the entire brain, especially in the frontal lobes, although the thalamus and the basal ganglia had normal levels. By immunological procedures and the cerebrospinal fluid (CSF) studies, the diagnosis of general paresis was made. He was treated with penicillin G and other antibiotics. In one year after his first therapy, many symptoms vanished. Although the cerebral atrophy was a little advanced, the cerebral blood flow was dramatically increased. As far as we know, this is the first PET study of general paresis before and after penicillin treatments.

Adult↗
PubMed1989

Bilateral trochlear nerve paresis in hydrocephalus.

Three patients with nonneoplastic hydrocephalus had bilateral paresis of the trochlear nerves. Associated signs, including paresis of upgaze, light-near dissociation of the pupils, and convergence-retraction nystagmus, suggested rostral involvement of the mesencephalon. Trochlear nerve paresis and accompanying signs improved after revision of ventricular shunts in two patients. Bilateral trochlear nerve paresis may be a localizing sign of involvement of the superior medullary velum (the anatomic site of trochlear nerve decussation) by a dilated sylvian aqueduct and/or downward pressure from an enlarged III ventricle.

Adolescent↗
PubMed1998

[Left lateral gaze paresis due to subcortical hematoma in the right precentral gyrus].

We report a case of transient left lateral gaze paresis due to a hemorrhagic lesion restricted in the right precentral gyrus. A 74-year-old female experienced a sudden clumsiness of the left upper extremity. A neurological examination revealed a left central facial paresis, distal dominant muscle weakness in the left upper limb and left lateral gaze paresis. There were no other focal neurological signs. Laboratory data were all normal. Brain CTs and MRIs demonstrated a subcortical hematoma in the right precentral gyrus. The neurological symptoms and signs disappeared over seven days. A recent physiological study suggested that the human frontal eye field (FEF) is located in the posterior part of the middle frontal gyrus (Brodmann's area 8) and the precentral gyrus around the precentral sulcus. More recent studies stressed the role of the precentral sulcus and the precentral gyrus. Our case supports those physiological findings. The hematoma affected both the FEF and its underlying white matter in our case. We assume the lateral gaze paresis is attributable to the disruption of the fibers from the FEF. It is likely that fibers for motor control of the face, upper extremity, and lateral gaze lie adjacently in the subcortical area.

Aged↗
PubMed1983

Chronic isolated abducens paresis from tumors at the base of the brain.

A chronic sixth-nerve paresis, even if isolated, may be neither benign nor idiopathic. We describe five patients with tumors in the basisphenoid region manifested clinically by isolated, unilateral abducens paresis for five to 20 years. There were three meningiomas (one confirmed by biopsy, two presumed), a neurilemmoma, and a chondrosarcoma. Isolated abducens paresis may persist for years in both children and adults with tumors or aneurysms. All patients with chronic sixth-nerve paresis should undergo vigorous neuroradiologic investigations to rule out a petrous apex-cavernous sinus mass lesion.

Abducens Nerve↗