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A latent trait approach to the development of persistent stuttering.

The premise that stuttering disorders develop according to the orthogenetic principle, preceding in a continuous, unilinear fashion from a state of relative lack of differentiation to a state of increasing differentiation and hierarchic integration, was examined. Responses to Woolf's Perceptions of Stuttering Inventory of 87 individuals who stutter were analyzed using a Rasch 1980 latent trait model for dichotomously scored data. Analyses of responses indicated struggle, avoidance, and expectation through the development of stuttering that became increasingly articulated, integrated, stable, and yet responsive to environmental changes. Four stages of development were noted: Stage I was characterized by the expectation of interruptions in the flow of speech, the addition of unnecessary sounds, and general body tension. Stage II was typified by distinctions between troublesome and not so troublesome words and sounds and between the speaker and various audiences and contexts for speaking. In Stage III, speech control decreased despite more focused and complex efforts to control the environment and the speech apparatus. Stage IV was characterized by automatic scanning of all speech, increasingly uncontrolled body movements, and attempts to produce fluent speech by way of comprehensive changes to sound, rhythm, and pitch. Follow-up of 29 respondents suggested the latent struggle was generally stable over time.

Adolescent↗

Personal constructs of fluency: a study comparing stutterers and nonstutterers.

The personal construct systems relating to fluency of a group of five stutterers and a group of five nonstutterers were examined using the repertory grid technique. The results support findings that stereotypical notions about stuttering were characteristic of both stutterers and nonstutterers and did not support Fransella's 1972 assumption that constructs relating to fluency differ in the communication subsystems of stutterers and nonstutterers.

Adult↗

Middle ear muscle activity during vocalization in normal speakers and stutterers.

Although it has been established that the middle ear muscles contract prior to vocalization, it is not known which muscle is mainly responsible for this activity. Also, there have been contradictory reports about whether this activity in stutterers differs from that of normal speakers. To tackle these questions, extratympanic pressure measurements prior to vocalization are reported for normal speakers and stutterers. This measure allows activity deriving from the two middle ear muscles to be differentiated and for the temporal course to be followed more accurately than by impedance measurement. To establish which muscle is primarily responsible for the pre-vocalization activity, the measured pressure changes prior to vocalization are compared with activity measured in tasks known to involve the stapedius or tensor tympani alone. These data show that in normal speakers and stutterers, the activity that is measured prior to vocalization resembles that of the tensor tympani. Contrary to other reports, there is no difference between normal speakers and stutterers in the time course of this activity.

Humans↗

An electropalatographic analysis of stutterers' speech.

Electropalatography (EPG) was used to investigate tongue-palate contacts from three adult stutterers during fluent and dysfluent speech and the results compared with normative data. Recordings were made of 22 read sentences, a reading passage and a conversational sample. The results showed that the lingual/palatal contacts of some phonemes during periods of stutterers' fluent speech consistently differed from that of the non-stutterers. Furthermore, stutterers produced greater intrasubject articulatory variability. Finally, during successive repetitions, the EPG patterns often became less similar to the target (or 'normal') configuration.

Adult↗

Stuttering amelioration at various auditory feedback delays and speech rates.

The primary purpose of this study was to determine if the finding of Kalinowski et al. (1993) of dramatic reductions in stuttering under delayed auditory feedback (DAF) at normal and fast speech rates could be replicated. The second purpose was to determine if stuttering frequency is differentially affected by various delays in an attempt to identify the optimal delay for fluency enhancement for both normal and fast speech rates. Fourteen adult stutterers read eight different passages at either a normal or fast speech rate under non-altered auditory feedback (NAF) and DAF with delays of 25, 50 and 75 ms. Results showed that significant fluency enhancement occurred under DAF at both normal and fast speech rates at all DAF settings (p < 0.05). This finding corroborates the notion that a slowed rate of speech is not a necessary antecedent for fluency improvement under conditions of altered auditory feedback. In addition, the results indicated that 50 ms appears to be the shortest delay producing the maximum reduction in stuttering frequency.

Adolescent↗

An electropalatographic (EPG) study of the speech of two stuttering subjects.

Two stuttering subjects and two control subjects were used in a comparative pilot study using EPG. The subjects read twenty sentences and repeated three words, 'a deer', 'a clock' and 'a kitkat', ten times. These data allowed three main methods of EPG analysis: variability measures on the three words in isolation; qualitative comparison between stuttering and control speakers' production of perceptually fluent speech and analysis of specific disfluencies. Results showed that compared to control speakers the stuttering subjects displayed increased contacts for fluent alveolar and velar plosives and fricative undershoot. They also showed less temporal variability for velar stop articulations. The results suggest that EPG is a useful technique in the analysis of articulatory characteristics of stuttering behaviour.

Adult↗

Oral kinesthetic deficit in adults who stutter: a target-accuracy study.

The current study was based on the hypothesis that chronic developmental stuttering in adults involves a deficiency in oral kinesthesia. The authors used a target-accuracy task to compare oral kinesthesia in adults who stutter (n = 17) and in normal speakers (n = 17). During the task, participants were instructed to make accurate jaw-opening movements in visual and nonvisual feedback conditions. The authors further contrasted oral movement control in a normal response time condition with that in a reaction time condition. Overall, the adults who stutter consistently made significantly less accurate and more variable movements than the control participants in the nonvisual condition, but particularly in the reaction time condition. In general, the present findings suggest that chronic developmental stuttering involves an oral kinesthetic deficiency, although without direct measures of somatosensory function, one cannot exclude a motor deficit interpretation.

Adolescent↗

Stuttering: an update for physicians.

Stuttering is a disturbance in the normal fluency and time patterning of speech. Developmental stuttering (DS), with or without associated psychiatric illness, is the most common form and includes all cases with gradual onset in childhood that are not the result of acquired brain damage. Persistent developmental stuttering (PDS) is DS that has not undergone spontaneous or speech-therapy-induced remission. Organic models of DS focus on incomplete lateralization or abnormal cerebral dominance. There is also evidence that DS has a significant genetic component to its cause. Neuroimaging research data and the effectiveness of dopamine receptor antagonists in DS seem to support the theory of a hyperdopaminergic origin. Speech therapy remains the main treatment for DS; however, antidepressants can be useful in selected cases. Risperidone, a serotonin-dopamine antagonist, has been shown to be more effective than placebo in decreasing the severity of stuttering. The long-term efficacy and safety of serotonin-dopamine antagonists in DS deserve further study.

Adult↗

Decreased copper level in the blood serum of male stutterers and the occurrence of the vibratio brevis phenomenon.

The blood serum copper level and the phonation start were investigated in sixteen male subjects with developmental stuttering (mean age = 25.8 years). A statistically significant decrease in blood serum copper level was revealed in the stutterers under study. The differences between their mode and mean values and that of the control group were 4.6 mumol/l and 3.1 mumol/l, respectively. The negative linear regression between the copper level in serum and the vibratio brevis phenomenon was registered in the stutterers under study. A decreased copper level can be involved in the biochemical abnormalities of male developmental stutterers and it can be included among the factors participating in pathophysiology of an uncertain phonation start.

Adolescent↗

[Stuttering as the only manifestation of a cerebral infarct].

A 53-year-old right-handed man acutely developed stuttering. On his examination there was blocks and repetitions exclusively on first syllables of words in conversational speech, improving in automatic tasks. There was neither aphasia, nor other neurological deficits. An MR imaging of the brain showed a circumscribed cortical infarct on the left precentral circunvolution. Cerebral angiography was consistent with atherosclerotic narrowing of the intracavernous segment of left internal carotid artery. Artery-to-artery embolism was the suggested mechanism for this stroke and the patient was treated with oral anticoagulants for six months. Stuttering improved progressively and the patient became asymptomatic a month after the stroke. Ictal acquired stuttering symptomatic of ischemic stroke without aphasia or other neurological deficit is exceptional. An small infarct on the left motor area as cause of isolated acquired stuttering adds new information about neural circuits involved in this phenomenon.

Cerebral Angiography↗

Genesis of self-identity as disother: life histories of people who stutter.

This paper explores the processes shaping self-identity formation as DisOther and the actions of participants who stutter. It illuminates the experiences of adults who stutter using a biographical, narrative, life history methodology. The participants were seven South African adults of diverse racial, social and economic backgrounds from KwaZulu Natal, South Africa. Five males and two females were invited to participate via purposive and convenience sampling processes. Their stories of living with stuttering in their life worlds over time were constructed via biographical interviews using personal, social and temporal lenses typical of life history methodology. The interviews were audio-recorded and transcribed. The data were analysed at two levels using a combination of strategies. The first level entailed a narrative analysis that was represented as research stories for each participant. The cross-case and thematic analysis of research stories constituted the second level analysis of narratives. The findings explain the complex and interrelated personal and social processes over time which contribute to the genesis of self-identity formation as DisOther Social inscriptions of difference occurred in immediate home, school and work contexts over time via multiple processes such as labelling, norming, judging and teasing. Personal processes included discoveries of difference via critical events, repeated reinforcement of difference, self-judgement and temporal burdening. Furthermore, the actions participants took in negotiating stuttering were examined. The implications of the findings and limitations of the study are presented.

Adaptation, Psychological↗

[Stuttering in children as a problem of phoniatric outpatient clinics].

Stuttering in young children is an extremely complex phenomenon. On all levels of intervention such as evaluation, differential diagnosis between stuttering and normal disfluency, diagnostic guidance of therapy process or direct and indirect treatment a number of questions arise for which answers have not been yet found. But realizing that the disorder is most difficulty to cure in adults and adolescents prevention or early treatment is essential. So diagnosticians and therapists need a high level of knowledge about the onset, the maintenance and the development of the disorder in every individual case of stuttering in young children. Decisions concerning the indications for diagnostic and therapeutic interventions must not be made by intuition but by professionally balancing the influencing factors such as psycho-social variables or marked deficits in mental, motor or sensory areas of the child's development. We want to demonstrate the different dimensions of the diagnostic and therapeutic process and the amount of professional time and effort which is necessary for the handling the stuttering child and his family.

Diagnosis, Differential↗

[Infanto-juvenile stuttering: a multidisciplinary approach].

The authors have conducted a clinical, psychopathological, exploratory research, with a multidisciplinary approach, applied to a sample of 52 children and adolescents stutterers, whose ages ranged between 3.6 and 19.8 years. They made a comparison of two groups, according to the moment in which they began stuttering and the level of development of their previous language. They emphasized the importance of studying the disorders of psychomotor and perceptive-motor functions; of learning disabilities, and the difficulties of interpersonal relationships in the family group and at school. They concluded that stuttering would be a symptom referred to a disorder in the rhythm of speaking, frequently associated to developmental deviations of different areas of language, psychomotor and/or perceptive-motor functions. Stuttering presents itself in normal as well as abnormal personality structures. Its beginning, persistence, and aggravation is frequently associated with unsatisfactory verbal and non verbal communication patterns.

Adolescent↗

Use of tiapride on stuttering in children and adolescents.

Reported here is the therapeutic efficacy of Tiapride with 10 10- to 17-yr.-old patients afflicted with severe stuttering problems. During the 20-wk. open, controlled study (baseline, low dose of Tiapride, high dose of Tiapride, follow-up observation without medication) the stuttering in various speech situations decreased markedly. No substantial change in different parameters of acoustic analysis and psychopathological characteristics accompanied the reduction in stuttering when group data were compared although positive effects concerning these variables could be seen in some cases. Several conclusions based on these results are presented. One assumed effect of the medication is improvement of central nervous regulatory mechanisms of speech motor coordination. Further, the value of the medication Tiapride in comprehensive stuttering therapy is discussed.

Adolescent↗

Indigenous healers and stuttering.

Traditional beliefs and attitudes of Black South Africans to stuttering were investigated. Four Indigenous Healers (IHs) from different ethnic groups were interviewed about their beliefs as to cause and management of stuttering, as well as the outcome of their treatment. The data reveals varying degrees of concern about stuttering. The traditional beliefs of cause and management of stuttering show some similarities to current beliefs held by speech pathologists. Implications in terms of direction in therapy, cooperation with IHs and future research in this field are discussed.

Adult↗

[Stuttering and neurosis].

Neurotic mechanisms have been found to be involved in the majority of cases of stuttering. Therefore, the treatment of this particular disorder should include different amounts of psychological treatment depending upon the role that such mechanisms are playing in the multifactorially determined etiopathogenesis of stuttering. The relationship of the symptomatology of stuttering to neurotic reactions and developments is described with reference to a typical clinical example. The description of the case, in which an attempt is made to determine the causes of the disorder, is followed by a discussion, relative to stuttering, of the classification of neuroses recommended in 1969 for use in the German Democratic Republic and taken as a base for this work.

Adult↗

[A case of acquired stuttering resulting from striatocapsular infarction].

Acquired stuttering resulting from a striatocapsular infarction is reported. A 54-year-old man was admitted to our hospital because of acute onset of stuttering and right facial palsy. The patient spoke very slowly. Speech was characterized by repetitions and prolongations. We considered that there was no aphasia, because comprehensive faculty was normal and neither paraphasia nor word-finding difficulty was recognized. Brain CTs and MRI revealed a striatocapsular infarction extending from the putamen to the caudate nucleus in the left hemisphere. Previously, there were reports of stuttering derived from parkinsonism and lesions in the supplementary motor area and thalamus. Therefore, we proposed that the stuttering arose from a collapse of basal ganglia circuits that connect the cerebral cortex and basal ganglia, including the extrapyramidal tract system.

Cerebral Infarction↗

Analysis of alpha-helical coiled coils with the program TWISTER reveals a structural mechanism for stutter compensation.

Alpha-helical coiled coils represent a widespread protein structure motif distinguished by a seven-residue periodicity of apolar residues in the primary sequence. A characteristic "knobs-into-holes" packing of these residues into a hydrophobic core results in a superhelical, usually left-handed, rope of two or more alpha-helices. Such a geometry can be parameterized. For this purpose, a new computer program, TWISTER, was developed. With the three-dimensional coordinates as input, TWISTER uses an original algorithm to determine the local coiled-coil parameters as a function of residue number. In addition, heptad positions are assigned based on structural criteria. It is known that frequently encountered discontinuities in the heptad repeat, such as stutters and skips, can be tolerated within a continuous coiled coil but result in a local distortion of its geometry. This was explored in detail with the help of TWISTER for several two- and three-stranded coiled coils. Depending on the particular protein, stutters were found to be compensated locally by an unwinding of the superhelix, alpha-helical unwinding, or both. In the first case, there is often a local switch from a left-handed to a right-handed superhelix. In general, the geometrical distortion is confined to about two alpha-helical turns at either side of the stutter. Furthermore, stutters result in a local increase of the coiled-coil radius.

Algorithms↗