Search PubMed⌕ Search

SEARCH · Search PubMed

Results for “SALTS”

Search indexed PubMed citations on genomics, clinical trials, systematic reviews and public health. Explore titles, authors and supplied subject terms, then open the PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 685 records · Page 38Linked to original sources
PubMed1967

Experimental microbiology of saturated salt solutions and other harsh environments. 3. Growth of salt-tolerant penicillium notatum in boron-rich media.

A stress-tolerant strain of Penicillium notatum, isolated by passage through a nutrient solution saturated with calcium acetate, was found to have a tolerance to boron in several states of oxidation. Growth in the presence of elementary boron, saturating amounts of boric acid, and with various concentrations of sodium borohydride was observed and mycelial mats were spectrographically analyzed for boron accumulation.

Boron↗
PubMed1991

Cytochemical Localization of ATPase Activity in Salt-Treated and Salt-Free Grown Lycopersicon esculentum Roots.

Adenosine-triphosphatase activity was localized by cytochemical methods in Lycopersicon esculentum Mill seedling roots. The identity of the enzyme was confirmed by its sensitivity to specific inhibitors. A differential distribution of ATPase activity was found depending on the region of the root. Under saline conditions, an increase of the tonoplast ATPase activity is observed, while the plasma membrane bound-ATPase activity decreases in the medial and basal regions of the root.

Journal Article↗
PubMed1999

Salt-Sensitive growth of Staphylococcus aureus: stimulation of salt-induced autolysis by multiple environmental factors.

The growth of Staphylococcus aureus 209P became extremely sensitive to a high NaCl concentration following lowered temperature, reduced air-supply, and decreased Ca2+ concentration in the medium. Cells in high-NaCl and low-Ca2+ concentration media either autolyzed or transformed into protoplast-like forms during growth when grown standing below 37 degrees C. The abnormal growth, however, was invariably avoided by preliminary supplementation with polyanetholesulfonate (autolysin inhibitor) in the growth media. These results suggested that the autolytic activity of this organism was precisely controlled by multiple environmental factors such as ionic strength, temperature, air supply, and the concentration of Ca2+.

Bacteriolysis↗
PubMed1976

Agonist and antagonist effects of Sar1-ala8--angiotensin II in salt-loaded and salt-depleted normal man.

1 Three normal subjects were infused with Sar1-ala8-angiotensin II (Saralasin, P113) whilst on a high sodium (200 mEq + normal diet) and a low sodium (10 mEq diet) intake. 2 On the high sodium intake when angiotensin II and plasma renin activity (PRA) were suppressed, P113 infusion (5-10 mug kg-1 min-1) caused a slight rise in BP and a marked drop in urine flow and sodium excretion, with a fall in glomerular filtration rate, and effective renal plasma flow. 3 On the low sodium intake, when angiotensin II and PRA were increased, P113 infusion (5-10 mugkg-1 min-1) caused no change in blood pressure, urine flow or sodium excretion. However, when P113 was infused at an incremental rate starting at 0.25 mug kg-1 min-1 there was a fall in standing BP, which was maximal at an infusion rate of 1 mug kg-1 min-1, and this fall in standing BP was largely abolished as the rate of infusion was increased to 10 mug kg-1 min -1. 4 These results show firstly that angiotension II is involved in maintaning standing blood pressure during dietary sodium depletion in normal man and secondly that P113 does have agonist as well as antagonist activity in normal man, the effect depending on the level of angiotension II and sodium intake. When looking for angiotensin II mediated hypertension it may ne important to use an incremental rate of infusion of P113 as the agonist activity of larger doses may mask its hypotensive action.

Angiotensin II↗
PubMedDate not supplied

Less salt intake or more salt excretion: is hypertension preventable?

Hypertension is preventable by modification of the common source (i.e., sodium chloride) and by prevention of weight gain and increased consumption of "good" nutrients. Unfortunately, the necessary societal changes are unlikely to occur. Individual behavioral approaches are weak. A pharmacologic approach as a "crutch" for our unsuccessful control of a "common source" epidemic may work and have a major impact on morbidity and mortality due to hypertensive diseases. Once-a-day diuretics may be a better health dose than once-a-day vitamins for our nutrient-rich society. (c)2001 by Le Jacq Communications, Inc.

Dietary Supplements↗
PubMed1981

A comparison of the effects of prostaglandins E2 and I2 on renal function and renin release in salt-loaded and salt-depleted anaesthetized dogs.

Prostaglandin E2 (PGE2) and prostaglandins I2 (PGI2) were infused into the renal artery of either sodium-loaded or sodium-depleted dogs. Systemic blood pressure was decreased and arterial plasma renin concentration increased in the sodium-loaded dogs by infusion of PGI2 (1200 ng/min). Both compounds increased renal blood flow but PGE2 had a much more potent effect than PGI2 on increasing urine flow and sodium excretion. In the sodium-depleted dogs systemic blood pressure was significantly decreased and arterial plasma renin concentration increased by infusions of PGI2 (300 and 1200 ng/min) and PGE2 (300 ng/min). Both compounds stimulated urine flow and renal blood flow but PGE2 induced a much larger increase in sodium excretion than PGI2. PGI2 exerts its actions on renal function by inducing vasodilatation whereas PGE2 also has a direct action on tubular function.

Animals↗