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[Messenger RNA and ribosomal RNA synthesis in leukemia cells during cell differentiation].
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Transcription and processing of viral messenger RNA in mammalian cells.
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Activation of preexisting messenger RNA in dry pea embryo axes.
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[Messenger RNA resulting from the expression of "early" and "late" functions of the lambda prophage].
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[Comparison of the biosynthesis of ribosomal and messenger RNA in a mammalian cell in the exponential growth phase and in the stationary phase].
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[A DNA complex of membrane lambda fractions in Escherichia coli elaborating lambda messenger RNA in the presence of DNAase].
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[Cellular messenger RNA in the initiation of influenza virus transcription].
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[Messenger RNA synthesis: supposition for cellular differentiation in higher plants].
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Some properties of bacteriophage messenger RNA's.
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[Messenger RNA synthesized after lambda prophage induction].
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[Polysomes and messenger RNA of cells of avian viral leukemia].
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[Identification of Newcastle disease virus messenger RNA].
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Messenger RNA in tumor cells induced by adenoviruses.
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Messenger RNA dependent synthesis of peptides by purified bacterial enzymes.
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[Nature of a virus-specific protein detected in the composition of a ribonucleoprotein containing cellular messenger RNA during viral infection].
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Increased adenosine deaminase synthesis and messenger RNA activity in deoxycoformycin-resistant cells.
The basis for the increased adenosine deaminase activity in deoxycoformycin-resistant rat hepatoma cells was investigated. Three variant cell lines with different levels of adenosine deaminase activity showed increases in the relative rate of synthesis of the enzyme in vivo. No difference in the rate of degradation of the enzyme was seen between the parental cell line and one variant cell line which exhibits a 180-fold increase in adenosine deaminase activity. Polysomal RNA isolated from this variant exhibited a 175-fold increase in the ability to direct the synthesis of adenosine deaminase in vitro.
In vitro translational activity of messenger RNA following treatment of human colon carcinoma cells with sangivamycin.
Total mRNA from human colon carcinoma cell line HT-29 treated with the pyrrolopyrimidine antibiotic, sangivamycin(7-deaza-7-carboxamidoadenosine), was assessed in vitro using a reticulocyte lysate translation system. Under conditions of known drug-induced cell lethality, sangivamycin-modified mRNA showed a diminished translational capacity. The decreased activity of drug-modified mRNA increased in proportion to the time-dependent cytocidal effects of sangivamycin in this cell line. These data suggest that the lethal effects of this drug may be associated, in part, with a reduced ability of drug-modified mRNA to sustain translation in situ.