[Experimental studies on the influence of intense sound on the ear-labyrinth].
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Langerhans' cell histiocytosis is a rare pathology that implies an abnormal proliferation of these kind of cells associated with granular infiltration that affects different structures of the human body, including the temporal bone. The middle ear and the mastoid are mostly involved (61%). The inner ear is more resistant to the destruction caused by the granulation tissue, but when occurs, an irreversible neurosensorial hearing loss appears. The early diagnose and the choice of the therapeutical sequences are essential to avoid these lesions.
We report the case of a 44-year-old woman suffering, since 5 years, from repetitive vertigo seizures, lasting less than one hour, together with light neuro-sensorial deafness, fullness of the left ear and left ear tinnitus. After analysis of audiologic and imagery tests a Meniere's syndrome secondary to Paget disease--confirm histologically through cortical cranial biopsy--was established.
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The case of a 61-year-old woman who presented with herpes labialis, subclinical meningitis, and sudden onset of bilateral sensorineural hearing loss is presented. Contrast-enhanced MR imaging showed marked bilateral enhancement of the intracanalicular portion of the eighth cranial nerve, right cochlea, and left vestibule. Polymerase chain reaction was positive for human herpesvirus 1 obtained from the cerebral spinal fluid, which suggested the diagnosis of viral neuritis.
The clinical picture of Positional Paroxysmal Vertigo (PPV) induced by vertical canal labyrintholithiasis has been clearly described, eliminating previous interpretations of pathogenesis of this disorder. The diagnosis of PPV is based on the well-known picture of positional paroxysmal Nystagmus. The Authors report a clinical sign which has not previously been reported in the literature: torsional Nystagmus induced by the Head-Shaking Test (HST). The Authors encoutered this sign in 30% of the cases of vertical canalolithiasis and in 50% of the cases diagnosed as vertical cupulolithiasis. This sign was also found in patients with a history of prior positional vertigo and in patients who, after treatment with release maneuver, no longer show clinical signs of positional vertigo. Such Nystagmus was not, however, found in the control group (normal subjects and patients suffering from other vestibular pathologies). In the present study the possible pathogenesis of this sign is discussed and some practical implications are considered.
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The microvasculature of the inner ear in guinea pigs and humans was observed with a scanning electron microscope using corrosion casting method. Alterations in the inner ear vasculature which occurred in association with experimental endolymphatic hydrops were also investigated. The results thus obtained are summarized as follows: 1. In the cochlea and vestibule, the arteries, coiled arterioles, and the veins are endowed with their respective characteristic morphologic features and play a role in the regulatory mechanisms of circulation. 2. The point in humans which is most different from guinea pigs was that coiled arterioles in the cochlea and the coil-like traveling of the anterior vestibular artery is not outstanding. 3. Arteriovenous anastomoses were demonstrated to exist in lateral wall of cochlea and utricular macula, a finding suggesting the existence of a regulatory mechanism for local blood flow. 4. Endolymphatic hydrops was noted to be preferentially associated with vascular abnormalities in the lateral wall of the cochlear duct and in the saccular macula, among other vestibular structures.
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