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[The effect of DL-methionine, N-hydroxymethyl-DL-methionine calcium salt (Mepron) and DL-methionine hydroxy analog calcium salt (MHA) on the N-deposits of calves].

Digestibility and N balance experiments with calves showed that the methionine requirement of the host animal is covered by microbial protein synthesis even in the already ruminant animal. A supplement of the ration with DL methionine is only effective when it is provided in milk replacer. The MHA substitution of the ration has only little influence on N retention, independent of its supply in milk replacer or concentrated feed. MEPRON proved to be the most effective methionine derivative. As supplement both with the fluid feed and with the concentrated feed this compound shows exclusively positive results even in already ruminant calves with regard to live weight gain, N retention and the content of free methionine in the blood serum of the test animals.

Animal Feed↗
PubMed1967

Effects of chronic excess salt ingestion. Genetic influence on the development of salt hypertension in parabiotic rats: evidence for a humoral factor.

Parabiosis has been found to modify the expected blood pressure response of rats from two strains with opposite genetic propensities for experimental hypertension. When a member from one strain was united in parabiosis with a member from the other and both were maintained on high NaCl diet, the rat from the strain ordinarily resistant to it rapidly developed hypertension, in contrast to appropriate controls from this strain. The development of hypertension in this resistant animal preceded that in its mate from the strain highly sensitive to hypertension. In the latter, both the level of hypertension and mortality were significantly less than in its control. It seems likely that the hypertension observed is the resistant parabiont was initiated in its partner from the sensitive strain. This modification in blood pressures was not observed in the absence of a high NaCl diet. Parabiosis between animals from the same strain did not alter their response. Thus, as in earlier experiences (1-4) the interaction of a nongenetic factor (NaCl) with the appropriate genetic substrate appeared to be necessary for the development of hypertension. The findings are interpreted as evidence that a transmittable humoral influence plays an important role in the pathogenesis of rat hypertension. The presence of this agent is genetically determined but, under the conditions of these experiments, it took the added stimulus of dietary NaCl to demonstrate its existence.

Animals↗