Psychological component in the etiology of geographic tongue.
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Irradiation injury of the mouse tongue was studied by electron and light microscopy. A specific lesion was found to be the result of edema. Epithelial compensation was seen in the form of proliferation of desmosomes and hemidesmosomes, elongation of rete ridges, and hypertrophy of cytoplasmic projections from basal cells.
The case of an 18-year-old patient who developed critical upper airway compromise after central tongue piercing is presented. Otolaryngologists must be aware of the many potential complications of tongue piercing and their management.
The advent of the human immunodeficiency virus and the increasing prevalence of immunocompromised individuals in the community have resulted in a resurgence of opportunistic infections, including oral candidoses. Despite the availability of a number of effective antimycotics for the management of oral candidoses, therapeutic failure is not uncommon. Further, the presence of many clinical variants of oral candidosis, both new and old, may confound the unwary clinician and complicate its management. These problems have been partly circumvented by the introduction of the triazole group of antimycotics, which initially appeared to be highly effective. However, an alarming increase in organisms resistant to triazoles has been reported recently. In this paper we provide an overview of clinical variants of oral candidosis. A second paper will discuss recent advances in the usage of antimycotics in the management of this condition.
We have presented a case of BMG with concomitant fissured tongue, a not uncommon association for two rather common oral diseases. BMG is relatively simple to recognize, and the diagnosis most commonly is based on clinical appearance and history of the presence of the lesions. Clinicians should be aware, however, of the similarity, clinically and histologically, of lesions of BMG with other, more serious diseases such as psoriasis and Reiter's syndrome. Patients with characteristic lesions of BMG should be evaluated closely for signs and symptoms of these other diseases.
Regardless of what name is used to identify the lesion and its nature of origin, clinicians should be aware of the clinical features and biologic activity of this relatively common oral abnormality and be prepared to reassure patients of the innocuous nature of the lesion.
Stomatitis areata migrans is an uncommon oral disease that may affect mucous membranes other than the tongue or be concomitant with geographic tongue. The clinical appearance emulates geographic tongue at an ectopic site, and the lesions rarely are symptomatic.
Five new cases of geographic stomatitis have been presented along with data from 24 previously reported patients. For the first time, basic parameters of this disorder have been tabulated from this population. Although the earliest documented case appeared in the literature in 1955, the majority of articles has been published during the last 15 years. This increased frequency of reporting indicates a growing awareness of this rather innocuous lesion. Thus, the general practitioner is advised to become familiar with the salient features of geographic stomatitis. Prompt recognition and diagnosis of this lesion, based on the clinical findings and history, will usually mitigate the need for biopsy.
Benign migratory stomatitis is a relatively rare entity, but because of its benign nature and clinical characteristics, it should be easily recognized by the general practitioner. Biopsy is usually not indicated and treatment consists of reassuring the patient that the lesions are benign even though they may disappear, reappear, and change location; the patient should report back for periodic follow-up.
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This case report discusses a 42-year-old male patient who presented with migratory stomatitis located on the labial and buccal mucosa and the lateral tongue border. The lesions were circumscribed, flat, smooth, and red in color with a slightly raised white border varying in size from 3 mm to over 1 cm. Duration was between 7 and 14 days and healing transpired without scarring. Follow-up continued for approximately 1 year and at each visit several lesions were seen. The possibility of stress and heredity as positive factors was considered, but with so few reported cases conclusions would be purely speculative. The absence of dermatologic pathology does not aid in establishing a relationship with psoriasis, however there is a microscopic similarity. An almost total lack of clinical symptoms may contribute to this sparse documentation, therefore dental practitioners should be articularly observant when examining oral soft tissues. Further recognition and investigation is necessary before a cause can be discovered.
A fluorescent antibody investigation was conducted to determine first the difference, if any, in the presence of tissue-bound antibodies in normal gingiva and atypical gingivostomatitis gingiva, and second to determine if the serum of atypical gingivostomatitis patients had auto-antibodies directed against any specific structures of normal gingiva. The immunofluorescent tests produced two signficant results: 1. Most of the mononuclear inflammatory cells present in AGS gingiva had an antibody halo on the cell membrane surface. This could indicate that AGS is the result of hypersensitivity reaction. 2. The serum of AGS patients did not contain detectable auto-antibodies for normal gingiva which would be one indication that AGS is not an autoimmune disease.
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Explore the source record for details and available documents.