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PubMed1990

[The local use of clofelin in the combined treatment of patients with periodontosis and hypertension].

Patients suffering of parodontosis and hypertensive disease (stage II) revealed an increase of the microcirculatory blood vessels of the periodontal tissues and correspondingly a deterioration of circulation in the region of the microcirculatory bed, a reduction of the stability of microcirculatory blood vessels of the periodontal tissues. After a single use of a 0.25% clophelin solution in the form of applications the pathologically elevated arterial pressure decreased and the tone of the periodontal tissue vessels reduced as well, the rheographic index increased, the stability of blood vessels of the microcirculatory bed increased. The author recommends the use of hypotensive agents in the complex treatment of changes of the periodontal tissues in patients with hypertensive disease.

Administration, Topical↗
PubMed1994

Genetic considerations of risk in human periodontal disease.

Increasingly, evidence suggests that underlying host susceptibility factors play a significant role in periodontitis in high-risk patients and that this risk may be partly under genetic control. Support for a heritable component to periodontitis risk comes from the consistent association of periodontitis with certain monogenic traits, as well as from formal genetic studies of both twins and early onset periodontitis. It is significant that not all genetic predispositions to periodontitis involve defects of the immune system. Structural or developmental defects in collagen, cementum, and epithelium also appear to be significant risk factors for periodontitis. Dissection of a genetic model system for periodontitis may prove valuable in fully understanding disease pathogenesis in periodontitis, particularly for understanding the role of host susceptibility factors.

Aggressive Periodontitis↗
PubMed1997

Can the location of tooth agenesis and the location of initial bone loss seen in juvenile periodontitis be explained by neural developmental fields in the jaws?

Recent studies on prenatal innervation of the jaws have shown that three separate main innervation paths, constituting three bilateral neural developmental fields (incisor field, canine/premolar field, molar field) exist in each jaw. In this communication the sequences in which the fields are innervated are indicated. These correspond to the sequences of formation of teeth and jawbone. The normal pattern of tooth agenesis is closely related to the neural fields, as the region within a single field were innervation occurs last is always the area most often affected by tooth agenesis. The initial manifestations of juvenile periodontitis also appear at the sites within the different fields where innervation occurs last. It is suggested that the pubertal growth of the alveolar process does not occur in these regions due to deficient innervation, and that the infection in juvenile periodontitis might be secondary to this regional lack of bone apposition.

Aggressive Periodontitis↗