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[Botulism; review and case report].
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Botulism accommodation paralysis.
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Fish-borne and type E botulism: two cases due to home-pickled herring.
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[Postmortem diagnosis of botulism].
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An unusual neuromuscular defect in a case of possible botulism.
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Neuromuscular physiology of wound botulism.
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Botulism. Ten-year experience.
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Acute infantile motor unit disorder. Infantile botulism?
Eight infants with an acute reversible motor unit disorder are described, including two infants from whom Clostridum botulinum type A was isolated from stool specimens. The clinical spectrum includes constipation, cranial nerve deficits, pupillary involvement, and generalized hypotonic weakness. There were no deaths, and all infants have had complete clinical recovery. A characteristic electromyographic (EMG) pattern was present in part until clinical recovery. This distinctive pattern consisted of brief, small, abundant for power exerted motor unit potentials. This EMG pattern in the context of the clinical syndrome may well be diagnostic for acute infantile motor unit disorder.
Ocular involvement in wound botulism.
A 7-year-old girl developed bilateral ptosis, total ophthalmoplegia, and fixed, dilated pupils associated with bulbar paralysis and generalized weakness six days after she sustained a compound supracondylar fracture of the right humerus. Nerve conduction studies showed a facilitated muscle action potential after repetitive nerve stimulation. Blood cultures were negative. Although the wound site appeared noninfected, the wound was explored. Clostridium botulinum, type B, grew from cultures taken from the depths of the wound. The patient recovered fully with supportive care, and EEG abnormalities present during the acute phase of the illness disappeared.
Infant botulism in 1931. Discovery of a misclassified case.
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Diagnosis and management of infant botulism.
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Type F infant botulism.
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