[Considerations on the therapeutic action of phenylpropyldiphenylpropylamine (Segontin) in myocardial infarct].
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Humans↗
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[Clinical observations and electrocardiography at rest and during exercise in coronary disease patients subjected to treatment with phenyl-propyldiphenylpropyl-amine].
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Angina Pectoris↗
The effect of Segontin, Persantin and nitroglycerin on the coronary artery. Experimental studies with cinecoronarography.
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Coronary Vessels↗
[Clinical and electrocardiographic considerations on the use of a new synthesis (phenyl-propyl-diphenyl-propyl-amine) in the therapy of syndromes of coronary insufficiency in urologic patients].
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Biphenyl Compounds↗
[Comparison of phenylpropyl-diphenylpropyl-amine with other drugs for the treatment of coronary angina and influence on the consumption of nitroglycerin derivatives].
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Amines↗
[Treatment with phenyl-propyl-diphenyl-propyl-amine (Segontin Hoechst) in coronary disease].
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Amines↗
Recently introduced cardiovascular drugs under experimental test.
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Cardiovascular Agents↗
[Histochemical studies on the action of Segontin on the adrenaline and noradrenaline content of the adrenal cortex of the golden hamster (Mesocricetus auratus)].
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Adrenal Cortex↗
[On modern coronary dilators].
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Dipyridamole↗
[A double-blind experiment with the coronary therapeutic agent segontin].
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Anti-Arrhythmia Agents↗
[Clinical experiences with segontin].
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Anti-Arrhythmia Agents↗
[Control of 5-hydroxyindolacetic acid excretion by Segontin].
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Biological Transport↗
[Experiences with segontin with special reference to chronic pulmonary heart disease, intrapleural pneumothorax and pneumoperitoneum].
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Abdomen↗
[Experiences with the clinical use of Segontin in coronary disease].
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Anti-Arrhythmia Agents↗
[Clinical studies on monoamine metabolism under the influence of segontin].
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Biomedical Research↗
[Coronary drugs: facts and assumptions (author's transl)].
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Adrenergic beta-Antagonists↗
[Inhibition of intestinal absorption as a principle of the treatment of metabolic diseases].
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Animals↗
Calcium channel antagonists. Omega-conotoxin defines a new high affinity site.
The omega-conotoxins, a class of Ca2+ channel antagonists from fish-hunting marine snails, have recently been described (Olivera, B. M., McIntosh, J. M., Zeikus, R., Gray, W. R., Varga, J., Rivier, J., de Santos, V., and Cruz, L. J. (1985) Science, 230, 1338-1343). One of these peptide neurotoxins, omega-conotoxin GVIA, was radiolabeled with iodine, and the 125I-labeled toxin was shown to bind specifically to high affinity sites on chick brain synaptosomes. The toxin-receptor complex was extremely stable; addition of an excess of unlabeled toxin did not cause significant displacement of the labeled toxin after 2 h. Binding competition data suggest that omega-conotoxin defines a new high affinity receptor site affecting voltage-activated Ca2+ channels, distinct from both the verapamil and dihydropyridine target sites.
Animals↗