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A quantitative detector-tube method for breath-alcohol estimation.
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ABC of oral health: periodontal disease.
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Chlamydial isolates from Reiter's syndrome.
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Differentiation of mouth versus gut as site of origin of odoriferous breath gases after garlic ingestion.
Utilizing the sulfur-containing gases of garlic as probes, we investigated the gut versus mouth origin of odoriferous breath gases. Five individuals ingested 6 g of garlic, and sulfur gases in mouth, alveolar air, and urine samples were measured. The mouth normally contained low concentrations of hydrogen sulfide, methanethiol, and dimethyl sulfide. Immediately after garlic ingestion, transient high concentrations of methanethiol and allyl mercaptan and lesser concentrations of allyl methyl sulfide (AMS), allyl methyl disulfide, and allyl disulfide were observed. With the exception of AMS, all gases were present in far greater concentrations in mouth than alveolar air, indicating an oral origin. Only AMS was of gut origin as evidenced by similar partial pressures in mouth, alveolar air, and urine. After 3 h, AMS was the predominant breath sulfur gas. The unique derivation of AMS from the gut is attributable to the lack of gut and liver metabolism of this gas versus the rapid metabolism of the other gases. Breath odor after garlic ingestion initially originates from the mouth and subsequently from the gut.
"Bad breath": presenting manifestation of anaerobic pulmonary infection.
Three patients are described in whom a putrid breath odor was the only or fist manifestation of anaerobic infection of the lung. Fever, productive cough, and pleuritic pain, common findings in anaerobic pulmonary infection, were not initally present. "Bad breath" may be an early due to the presence of anaerobic pulmonary infection.
Progression of olfactory reference syndrome to mania: a case report.
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Olfactory reference syndrome and depression.
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Fetor ex ore--a rare presenting symptom of a complete second branchial fistula.
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Morning breath odor: influence of treatments on sulfur gases.
We assessed the effects of several treatments on the concentrations of oral sulfur-containing gases, compounds thought to be responsible for morning breath. Upon awakening in the morning, healthy volunteers collected oral gas samples before and for eight hours after the following treatments: no treatment, brushing the teeth with toothpaste, brushing the tongue, rinsing with 5 mL of 3% hydrogen peroxide, breakfast ingestion, or swallowing two BreathAsure capsules. The gas samples were analyzed for sulfur-containing volatiles via gas chromatography. Baseline collections usually contained three sulfur gases: hydrogen sulfide, methanethiol, and dimethylsulfide. The effectiveness of a treatment was determined via comparison of the areas under gas concentrations-time curves with and without treatment. Brushing the teeth or ingestion of BreathAsure had no apparent influence on the sulfur gases. Ingestion of breakfast and tongue brushing resulted in strong trends toward decreased sulfur gases. Hydrogen peroxide significantly reduced the sulfur gas concentrations for eight hours.
Correlation of some oral hygiene variables with age, sex, and incidence of oral protozoa.
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Effects of an antiseptic mouthwash on odorigenic microbes in the human gingival crevice.
Odorigenic gram-negative anaerobes in human gingival crevices were significantly decreased for up to 120 minutes after rinsing with an antiseptic mouthwash. This observation parallels clinical measurements of the antiseptic effect on oral malodor and suggests that its odor-decreasing properties are related to its antimicrobial action in the mouth.
The in vivo effects of an antiseptic mouthwash on odor-producing microorganisms.
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Mechanism of action of an antiseptic, anti-odor mouthwash.
Inter-related determinants of oral malodor were measured over a three-hour period in 30 human subjects after mouthwash treatments. Re-odoration was important to mouthwash activity for 30 min. At post-treatment times of 60-180 min, the anti-odor activity of the product is due solely to its anti-microbial action.