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Results for “Brain Infarction”

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At least 559 records · Page 31Linked to original sources
PubMed1991

[Young patient with left brain infarct and transient right sided hemiparesis in cardiac papillary fibroelastoma].

This is a case report of a 25-year-old female with recurrent episodes of transient cerebral ischemia accompanied by transient hemiparesis. Cardiac ultrasound examination revealed a small left-ventricular tumor located between the chordae tendineae. The echocardiographic finding was subsequently confirmed by surgery. The entire tumor was removed and the mitral valve remained intact. Due to the histological and immunohistochemical findings a papillary fibroelastoma was diagnosed. The early and late postoperative course was without event.

Adult↗
PubMed1990

[Menetrier's disease: brain infarction as an unusual first symptom].

A 43-year-old patient was admitted to hospital with an apoplectic stroke caused by an angiographically confirmed partial stenosis of the arteria cerebri media branch. We diagnosed Ménétrier's disease, documented by gastroscopy and histology, after having eliminated other causes for the apoplexy. We suggest a direct causal relationship between the stroke and Ménétrier's disease, since high incidences of thromboembolic complications have been reported in association with this rare disease. After conservative therapy with H2-blockers, laboratory values returned to normal. In the two years following diagnosis, the patient reported no recurrence of neurologic complications.

Adult↗
PubMed1989

[Protein-S deficiency in young patients with thrombotic brain infarction].

The observation of protein S deficiency in a family six members of which had recurrent cerebral strokes prompted a prospective study so far including 33 patients (23 women, 10 men) with cerebral arterial thrombosis at the age of 19 to 57 years (mean 31 years) admitted to the neurological departments of several hospitals. Diseases associated with cerebral arterial occlusions, such as arterial embolism or hematological, infectious and immunological disorders were not found. The following parameters were tested: antithrombin III, protein C, plasminogen, fibrinolytic capacity after infusion of DDAVP, and free and total protein S. Prothrombin times of all patients were within normal ranges; no woman had taken oral contraceptives for at least 3 months before examination. In 9 patients (8 women, 1 man) a decrease of free and total protein S was found; in 4 additional patients (2 women, 2 men) only the free protein S was reduced. Protein S deficiency was confirmed as an inherited defect in 5 cases by family studies, but excluded in one (both parents normal). Protein S deficiency was combined with protein C deficiency in 3 cases, plasminogen deficiency in 2 cases and reduced fibrinolytic capacity in two. Venous thromboembolism occurred only in one of the 13 patients with protein S deficiency and cerebral arterial thrombosis. Protein S deficiency may be found-mainly as a familial trait-in a substantial percentage of younger patients with "idiopathic" cerebral arterial thrombosis.

Adult↗
PubMed1989

[An unusual sequela of a frequently occurring neurologic disorder: delirium caused by brain infarct].

Six patients are described with delirium after cerebral infarction. Five had a right-sided parietal infarction with involvement of the inferior parietal lobule. One patient presented with a right-sided medial temporo-occipital infarction. The mild neurological signs were dominated by the delirium. The outcome was good in all patients. Delirium after cerebral infarction is probably caused by injury to one of the convergence sites for integration of sensory information with disturbance of the directed attention to relevant stimuli. These sites are localized in the right parietal cortex and the medial temporo-occipital regions. This cause of delirium is probably compatible with the supposition that the right hemisphere is mainly responsible for the distribution of directed attention within extrapersonal space. The importance of an accurate neurological examination in delirious patients is emphasized.

Aged↗
PubMed1986

[Hemodynamic risk factors of secondary brain infarcts in patients with occlusion of the internal carotid artery].

Study of the cerebral hemodynamics in 52 patients with occlusion of the internal carotid artery made it possible to ascertain the factors responsible for a bilateral decrease in the cerebral blood flow. The identification of the causes of a decreased cerebral blood supply in this pathology may contribute to timely rational treatment of patients and to reducing the risk of repeated cerebral infarction.

Adult↗
PubMedDate not supplied

MRI of brain infarction.

MRI of twenty patients with cerebral infarction were reported with their X-CT. MRI with long SE mode clearly showed the ischemic lesion at 18 hours after insult, whereas X-CT performed immediately after MRI scanning showed no abnormality. The signal contrast of the lesion with long SE mode seemed to increase slightly during the patient's course, for a period of several months. The MRI images with long SE at the chronic stage were varied: small lesions appeared as hot areas, whereas large lesions appeared as cold areas and were accompanied with signal enhancement in the surrounding areas. A phantom study was also performed and it was determined that MRI was superior to X-CT in its ability to detect tissue water. One of the reasons for the high diagnostic capability of MRI for acute stroke was, therefore, attributed to this experimental result.

Aged↗