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PubMedDate not supplied

Measurement of safe sex behavior in adolescents and young adults.

The aim of this project was to develop an instrument to measure use of safe sex practices among adolescents and to conduct initial evaluation of the psychometric properties of the instrument. The Safe Sex Behavior Questionnaire (SSBQ) was designed to measure the frequency of use of safe sex practices and was assessed for content validity, reliability, and construct validity through a series of tests. The content validity index computed for the SSBQ was 98%. Initial reliability computed for sums of items of the total scale was .82 among 89 college freshmen. Using a second sample of 531 subjects, the SSBQ was factor-analyzed separately for males and females and five similar factors emerged for each gender. Reliability coefficients for sums of salient items for each factor ranged from .52 to .85. Using a third sample of 174 subjects, construct validity was assessed by correlating the SSBQ with measures of general assertiveness and general risk-taking. The resulting correlations were appreciable and in the predicted directions, thus providing support for the construct validity of the instrument.

Acquired Immunodeficiency Syndrome↗
PubMed1992

The relationship of assertiveness to college students' safer sex behaviors.

Educational programs designed to curtail the present sexually transmitted disease (STD) epidemic emphasize the role of assertiveness in reducing personal risk. This study investigated the relationship between assertiveness tendencies and safer sex behaviors in heterosexual college students. It was predicted that assertiveness would vary positively, and nonassertiveness negatively, with three types of precautionary behaviors: caution about engaging in sexual intimacy, inquiry about a potential partner's STD risk history, and frequency of condom use. A convenience sample of 253 undergraduates (159 females and 94 males) anonymously completed a sexual behavior/demographic questionnaire and the Intimate Relationships Questionnaire (IRQ), designed for this study and involving assertive, nonassertive, and aggressive responses to scenarios relevant to safer sex. The findings confirmed the hypothesized relationships between IRQ assertiveness tendencies and safer sex practices. Gender differences were discovered on the IRQ and in the health-protective behaviors of using caution and inquiring about a prospective partner's STD risk history. Implications for STD prevention strategies are discussed.

Adult↗
PubMedDate not supplied

Decreased postnatal testosterone and corticosterone concentrations in rats following acute intermittent prenatal hypoxia without alterations in adult male sex behavior.

The prenatal and postnatal testosterone surges in the male rat are associated with neurobehavioral sexual differentiation of the brain. Both surges can be attenuated by maternal stress or other environmental factors that activate the maternal and/or fetal hypothalamic/pituitary/adrenal (HPA) axis during the last week of gestation. Since hypoxia is known to activate the HPA axis, we studied its effect during gestation on sexual differentiation in the male rat. We examined the influence of intermittent hypoxic exposure during gestation with respect to the postnatal testosterone surge and corticosterone levels, and subsequent development of adult reproductive and nonreproductive sexually dimorphic behaviors. Plasma testosterone and corticosterone concentrations of male neonates were measured after maternal exposure to acute, intermittent, prenatal hypoxia (9% O2 6 h/day from Day 15 to 21 of gestation). Relative to normoxic controls, acute, intermittent, prenatal hypoxia significantly attenuated the postnatal testosterone surge. Postpartum plasma corticosterone levels in these animals were also suppressed. In adulthood, prenatally hypoxic animals exhibited normal masculine sex behavior. Lordosis behavior in response to estrogen and progesterone priming was not significantly different between treatment groups. Saccharin preference, a nonreproductive, sexually dimorphic behavior, was not significantly influenced by prenatal hypoxic exposure. These results demonstrate that in the male acute intermittent prenatal hypoxia attenuates the postnatal testosterone surge. However, this reduction failed to result in significant alterations in the expression of sex related behaviors in adulthood.

Animals↗
PubMed2004

Induction of PGE2 by estradiol mediates developmental masculinization of sex behavior.

Adult male sexual behavior in mammals requires the neuronal organizing effects of gonadal steroids during a sensitive perinatal period. During development, estradiol differentiates the rat preoptic area (POA), an essential brain region in the male copulatory circuit. Here we report that increases in prostaglandin-E(2) (PGE(2)), resulting from changes in cyclooxygenase-2 (COX-2) regulation induced by perinatal exposure to estradiol, are necessary and sufficient to organize the crucial neural substrate that mediates male sexual behavior. Briefly preventing prostaglandin synthesis in newborn males with the COX inhibitor indomethacin permanently downregulates markers of dendritic spines in the POA and severely impairs male sexual behavior. Developmental exposure to the COX inhibitor aspirin results in mild impairment of sexual behavior. Conversely, administration of PGE(2) to newborn females masculinizes the POA and leads to male sex behavior in adults, thereby highlighting the pathway of steroid-independent brain masculinization. Our findings show that PGE(2) functions as a downstream effector of estradiol to permanently masculinize the brain.

Animals↗
PubMedDate not supplied

Beyond stereotypes: promoting safer sex behaviors among Latino adolescents.

Latino youth are at increasing risk for consequences of risky sexual behavior including pregnancy and sexually transmitted diseases, as well as HIV infection. The diversity among Latinos and the prevalence of stereotypes about Latino sexual altitudes, beliefs, and behaviors present barriers in effective prevention and treatment. Strategies for promoting safe sex among Latino adolescents include recognizing the diversity among Latino adolescents and assessing attitudes, beliefs, and values that can be used to support safer sex behaviors.

Adolescent↗
PubMed2000

The two thyroid hormone receptor genes have opposite effects on estrogen-stimulated sex behaviors.

The two genes coding for thyroid hormone receptors (TR) alpha 1 and beta have opposite effects on female sex behaviors. Deletion of TRalpha 1 reduced them, whereas deletion of TRbeta actually increased them. These results could not be attributed to altered levels of hormones in the blood, general alterations in estrogen responsiveness or altered general activity. Instead, they indicate a previously unknown molecular mechanism upon which the two TR genes exert opposite influences.

Animals↗
PubMed1994

Sexually dimorphic cell groups in the medial preoptic area that are essential for male sex behavior and the neural pathways needed for their effects.

The research summarized here shows that the two major cell groups of the sexually dimorphic area (SDA) of the gerbil hypothalamus are essential for male sex behavior. Bilateral cell-body lesions of either the medial or lateral SDA virtually eliminate mating in sexually experienced male gerbils given exogenous testosterone. Similar deficits occur when the SDA is bilaterally disconnected from the retrorubral field (RRF) as a result of unilateral cell-body lesions in the SDA and contralateral RRF. The A8 cells of the RRF do not account for this effect. Bilaterally disconnecting the SDA from the caudomedial part of the bed nucleus of the stria terminalis (caudal BSTm) also eliminates sexual activity. Disconnecting the SDA from the medial amygdala does not mimic this effect. Neither does disconnecting the medial amygdala from the caudal BSTm. Thus, caudal BSTm neurons that are essential for mating via connections with the SDA do not simply relay information from the medial amygdala.

Amygdala↗
PubMed1994

Partial demasculinization and feminization of sex behavior in male rats by in utero and lactational exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin is not associated with alterations in estrogen receptor binding or volumes of sexually differentiated brain nuclei.

In utero and lactational 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) exposure partially demasculinizes and feminizes sexual behavior in adult male rats, presumably by causing incomplete sexual differentiation of the central nervous system (CNS). Our objective was to determine if TCDD exposure affects other aspects of sexual differentiation of the CNS. Because sex differences in the estrogen receptor system are thought to play a role in sexually dimorphic estrogen-mediated responses, and because estrogen is an important activator of both male and female sex behavior, the possible effect of TCDD exposure on estrogen binding in specific brain nuclei was examined. In addition, we investigated effects of in utero and lactational TCDD exposure on sex differences in the volumes of brain nuclei which are dependent on steroid hormone stimulation during the period of CNS sexual differentiation. Pregnant Holtzman rats were given TCDD (0.7 microgram/kg, po) or vehicle (control) on gestation Day 15. Offspring were exposed to TCDD in utero and via lactation and then assessed in adulthood. Demasculinized sexual behavior was evidenced in the TCDD-exposed males by increased intromission latencies and a greater number of intromissions prior to ejaculation. These males were then castrated, primed with ovarian steroids, and tested for feminine sexual behavior. In utero and lactational TCDD exposure increased both the frequency and intensity of lordotic behavior, indicating that the males were partially feminized. To determine if TCDD exposure had a generalized effect on estrogen receptor concentrations the arcuate nucleus, cortical and medical amygdala and the bed nucleus of the stria terminalis, previously found to have equivalent numbers of estrogen receptors in males and females, were evaluated in littermates of the rats whose sexual behavior had been assessed. TCDD had no effect in either sex. To determine if TCDD exposure had an effect specific to sexual differentiation of the brain, estrogen receptor concentrations in the medial preoptic nucleus (MPO), ventrolateral aspect of the ventro-medial nucleus, and periventricular preoptic area were assessed. As expected, females had higher estrogen receptor concentrations in these nuclei than did males, but TCDD exposure did not affect estrogen receptor concentrations in any of these sexually dimorphic brain nuclei. The volumes of sexually dimorphic brain nuclei were examined in additional littermates. In control rats, the volume of the sexually dimorphic nucleus of the preoptic area (SDN-POA) is larger in males than in females whereas the MPO is larger in females than in males. TCDD exposure had no effect on the volume of either the SDN-POA or MPO in either males or females.(ABSTRACT TRUNCATED AT 400 WORDS)

Animals↗
PubMed1984

Immunoreactive-beta-endorphin and LHRH levels in the brains of aged male rats with impaired sex behavior.

Levels of immunoreactive beta-endorphin and luteinizing hormone releasing hormone (LHRH) were measured in brain tissue of aged male Long-Evans rats. The animals were tested for sex behavior twice in one week at bimonthly intervals between the 7th and 27th month of life and were sacrificed along with a group of young (5-month old) sexually active rats. Thirty-one of the 89 rats which began the study remained healthy and tumor-free. By month 27, 21 of these had completely ceased to mate and 10 continued to show adequate sexual behavior. Diminished levels of beta-endorphin-like immunoreactivity were measured in the hypothalami and hindbrain of the old animals grouped together as compared to young animals and this reduction was shown to be significantly greater in hypothalamic tissue from the behaviorally inactive subgroup. Hypothalamic LHRH levels were not significantly altered by age in these animals. However, a marked reduction of LHRH content in the septal and midbrain regions of the aged-behaviorally inactive subgroup was evident when compared with the behaviorally active group. The data suggest that altered function of beta-endorphin and LHRH neurons of the aged brain may be involved in the behavioral deterioration observed in aged animals.

Aging↗
PubMed1993

The medial and lateral cell groups of the sexually dimorphic area of the gerbil hypothalamus are essential for male sex behavior and act via separate pathways.

Male reptiles, birds and mammals do not copulate if the medial preoptic area (MPOA) is destroyed but the MPOA cell groups necessary for male sexual behavior were not known. Here, two cell groups essential for copulation are identified in the sexually dimorphic area (SDA) of the gerbil (Meriones unguiculatus) MPOA. Bilateral cell-body lesions of either the medial or lateral SDA eliminated mating in sexually experienced male gerbils given testosterone. Nearby MPOA lesions did not. The medial and lateral SDA affect sex behavior via separate pathways since lesioning the medial SDA on one side of the brain and the lateral SDA on the other did not stop sexual behavior.

Animals↗
PubMed1975

Stereotyped sex role behavior and sex guilt.

A study of self-reported sex guilt and sex behavior in college students closely replicated previous findings. In addition, it was found that high guilt subjects were more likely to act in conformity with social expectations regarding sexual behavior. That is, high guilt males were more likely to be assertive in their sexual behaviors than low guilt males, and high guilt females were more likely to engage in passive sexual behaviors than low guilt females. Assertion was defined as acting upon another, and passivity was defined as being acted upon.

Aggression↗