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Results for “Program Activities”

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PubMed1990

[Scientific basis and professional concept of instituting a medical program of active recreation].

Physical activity is essential to health and should be exercised in accordance with one's functional and health state. Among the most advanced forms of programmed physical activity is medically programmed active leisure (MPAL). The implementation of MPAL in practice relies on research and on scientifically based concepts and attitudes which control the selection of persons for and their inclusion in MPAL. In Croatia MPAL is a form of specific care for worker's health, which is conducted in compliance with the health care and health insurance acts.

Exercise↗
PubMedDate not supplied

Legal issues in the delivery of worksite health promotion.

Employee health promotion programs typically involve one or more of the following: (1) education programs, (2) cholesterol, blood pressure, diabetes, or other similar screening programs, (3) smoking cessation programs, (4) drug/alcohol testing and counseling programs, (5) exercise facility or activity programs, and (6) recreational activity programs. The authors examine potential legal implications of these activities. This chapter is intended for those attempting to define and establish these programs or to focus and operate them without undue conflict or legal system involvement.

Health Education↗
PubMed1991

Activation of programmed cell death by recombinant human tumor necrosis factor plus topoisomerase II-targeted drugs in L929 tumor cells.

The mechanism of death induced by recombinant human tumor necrosis factor (rHuTNF) in L929 tumor cells of C3H mice was investigated. Treatment with rHuTNF led to fragmentation of DNA into nucleosomal oligomers and to induction of the expression of TRPM-2, a programmed cell death-associated gene. Both events preceded cell death by several hours. Treatment with DNA topoisomerase II inhibitors accelerated both the rHuTNF-mediated DNA fragmentation and the elevation in TRPM-2 messenger RNA levels. These results suggest that rHuTNF exerts its cytotoxicity on L929 cells by activating programmed cell death, leading to apoptosis, and that topoisomerase II inhibitors enhance rHuTNF-mediated cytotoxicity by accelerating this process.

Animals↗