[Hearing disorder. Mechanical management of hearing loss: conventional and implantable hearing aids].
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A series of 22 patients who presented with non-organic hearing disorder were examined for auditory perceptual problems. A test battery that examined eight areas of auditory perception revealed significant auditory perceptual problems in each of the 22 patients. The results indicate a view that differs from the traditional view of non-organic hearing disorder as either conscious feigning of a hearing disorder or an unconscious symptom of an unidentified emotional disorder. The information presented here indicates that specific auditory processing disorders could adversely affect patients' hearing and that auditory perceptual disorders could be diagnosed and treated.
The early fitting of a hearing aid to a child with a proven hearing disorder is decisive for the further development of the child. Depending upon the age of the child, the fitting of a hearing aid requires a variety of strategies, and differs considerably from aid fitting in adulthood. It is more difficult, requires more time, and needs a lot of patience and experience. The individual steps and procedures for fitting an aid are described, and the possibilities of rehabilitation of a child with such a hearing impairment are discussed. Apart from the optimal provision of a hearing aid, the child requires intensive early and long-term aftercare to ensure normal speech and normal development.
Hearing-aid provision is still partly a medical task, in spite of technological progress and a confusing variety of types and models of devices. The diagnosis of the underlying disease, the appreciation of the consequences of hearing loss and the counseling of the hearing impaired person are prerequisites of the treatment. The physician should try to determine the etiology of the hearing loss. Also, a state-of-the-art otologic examination should be performed and the physician should be sensitive to the psychological and social consequences of the patient's hearing loss. For the assessment of fitted hearing aids, basic knowledge of the technical background is required. To measure hearing-aid benefit in the ENT office, suprathreshold and speech audiometry are indispensable tools.
The aim of this study was to assess hearing and hearing disorders among rock/jazz musicians. One hundred and thirty-nine (43 women and 96 men) musicians participated. The results are based on pure-tone audiometry and questionnaire responses. According to our definition of hearing loss, tinnitus, hyperacusis, distortion and/or diplacusis as hearing disorders, we found disorders in 74%, of the rock/jazz musicians studied. Hearing loss, tinnitus and hyperacusis were most common, and the latter two were found significantly more frequently than in different reference populations. The women showed bilateral, significantly better hearing thresholds at 3-6 kHz than the men. Hyperacusis, and the combination of both hyperacusis and tinnitus, were found to be significantly more frequent among women than among men. Hearing loss and tinnitus were significantly more common among men than among women. It is important to evaluate all kinds of hearing problems (other than hearing loss) in musicians, since they represent an occupational group especially dependent on optimal, functional hearing. On the basis of our results, we suggest that hearing problems such as tinnitus, hyperacusis, distortion and/or diplacusis should, in addition to hearing loss, be defined as hearing disorders.
OBJECTIVE: To investigate the relationship between hypertension and hearing disorders of the elderly. DESIGN: Prospective case-control study. SETTING: Lin Yi People's Hospital, Shandong Province, the People's Republic of China, from October 1995 to June 1997. PARTICIPANTS: Fifty five hypertensive patients and fifty five normal elderly subjects (control group) between the ages of fifty five and eighty nine years old were enrolled in the study. All patients were male and were divided into three groups according to age as follows: Group A: 55-64; Group B: 65-74; Group C: 75-89. INTERVENTIONS AND MAIN OUTCOME MEASURES: SIMENS DA-64 audiometer and MEDSON 2250 auditory brainstem response audiometry (ABR) were utilised to determine the degree of hearing disorders. Serum triglycerides and cholesterol were also tested in our central laboratory. RESULTS: Auditory thresholds collected during audiometric tests increased gradually in proportion with age, especially in the hypertensive group (p < 0.05). The results of the ABR demonstrated that the latencies of wave V, I-VIPL and III-VIPL were also prolonged compared with normal elderly subjects. The patient's duration of illness and the complications of hypertension had an influence on hearing disorders in relation to the ageing process. CONCLUSION: Hypertension, with a high level of blood triglycerides and cholesterol, worsens the hearing of the elderly. The hearing disorder in elderly people is a result of long duration of illness and the complications of the hypertension.
Evaluation of central hearing disorders in neuropsychologic patients is handicapped by their insufficient ability to describe auditory deficits and by the lack of easily applicable audiological tests. A novel psychoacoustic discrimination test (PDT) was developed to determine ear asymmetries in the discrimination of changes in intensity, frequency, or temporal structure of regularly presented dichotic stimuli. In 19 of 21 patients with lesions of the auditory cortex or the acoustic radiation according to CT scan evaluation a higher error score was observed for target stimuli presented at the ear contralateral to the side of brain infarction (6 right, 15 left). In the remaining 2 and in 3 other patients with lesions sparing auditory structures no significant ear asymmetries were seen. This may indicate that auditory perception is reduced in patients with only one intact auditory cortex or one intact acoustic radiation, possibly because of a limitation in information processing capacity. Auditory evoked potential results are presented for a normal subject and two patients to illustrate electrophysiologic correlates of central hearing disorders. Using a transformation of scalp into dipole source activity (Scherg and von Cramon 1986), a unilateral loss of middle latency activity was found in case A, who had a lesion of the left acoustic radiation. The extended lesion of the right auditory cortex in case B resulted in a loss of both middle and late latency dipole source potentials of the right temporal lobe. In both cases a corresponding increase in the PDT error score on the contralateral ear was found.
Tinnitus and hearing loss, both reversible and irreversible, are associated both with acute intoxication and long term administration of a large range of drugs. The mechanism causing drug-induced ototoxicity is unclear, but may involve biochemical and consequent electrophysiological changes in the inner ear and eighth cranial nerve impulse transmission. Over 130 drugs and chemicals have been reported to be potentially ototoxic. The major classes are the aminoglycosides and other antimicrobials, anti-inflammatory agents, diuretics, antimalarial drugs, antineoplastic agents and some topically administered agents. Prevention of drug-induced ototoxicity is generally based upon consideration and avoidance of appropriate risk factors, as well as on monitoring of renal function, serum drug concentrations, and cochlear and auditory functions before and during drug therapy. Ototoxicity, although not life-threatening, may cause considerable discomfort to patients taking ototoxic drugs, and in some cases drug discontinuation may be necessary to prevent permanent damage. Much research has been performed to investigate the causes and mechanisms of ototoxicity, to try to prevent this complication. Despite these efforts, ototoxicity still occurs, and there is much work to be done in order to understand the mechanism of ototoxicity of different drugs and to prevent hearing loss and tinnitus in the future.
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Among 4947 children in an outpatients unit for hearing disorders 102 children with Down's syndrome were seen and checked for hearing disorders. 57 had hearing deficiencies, 50 (88 %) conductive hearing loss, 4 (7 %) combined and 3 (5 %) a sensory neural hearing loss. Compared to other publications the number of very young children was very high. 32 patients under two years of age had a hearing disorder. The results underline the necessity of early diagnosis and follow up also in children with normal reactions during the first presentation. Early diagnosis enables early treatment, conservative, surgical or fitting with hearing aids, especially important in the rehabilitation of these children. Hearing aids may be given temporarily imploring communication during the development of the child.
In a short survey the physiologic basis of hearing mechanisms, main etiological factors and histopathological findings in hearing disorders are reviewed. The main cause of iatrogenic sensoneural hearing disorders are aminoglycosides. Some hints are given to prevent these ototoxic side effects. The so-called "sociacusis" is a special part of environmental hearing disorders, due to non-occupational noise trauma. Presbyacusis is therefore the end result of various exogenous and also endogenous factors impairing the hearing ability during the individual course of life.
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Hearing losses of different etiology can impair the individual development during the entire childhood. Especially moderate hearing losses are difficult to diagnose because they rather present as speech or learning disorders. From the paedaudiologist's view methods are printed out for an early detection of hearing loss during childhood.
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Hearing impairment is not often considered as a potential complication of general anesthesia, despite several reports of post-operative sensorineural hearing loss. These disorders have occurred after otological as well as cardiobypass surgery. We experienced two patients both of whom had undergone orthopedic surgery. In both cases the patients experienced bilateral reversible hearing impairment after general anesthesia with nitrous oxide. It is likely that a change in the middle ear pressure as a result of Eustachian tube dysfunction may have caused transient conductive hearing loss added to sensorineural hearing disorder. After these cases we interviewed a series of 115 patients who had undergone general anesthesia to assess the extent of this problem. Contrary to our expectation, 7 patients complained of ear fullness or autophony after inhalation of nitrous oxide, although these symptons diminished within 24 hours. It is important to be aware of the possibility of hearing impairment when nitrous oxide is used especially if the patient has a history of a previous middle ear disease.
Fourteen children with the fetal alcohol syndrome were evaluated by standard audiologic procedures. Thirteen of the 14 children had childhood histories of hearing disorders. All 13 of these children (93%) had clinically significant histories of bilateral recurrent serous otitis media (ie, they were otitis prone), and at least four children (29%) had bilateral sensorineural hearing losses in addition to being otitis prone. Many of the children with recurrent serous otitis media required repeated myringotomies with placement of ventilation tubes, and those with sensorineural hearing losses required sound amplification during childhood. Recurrent respiratory infections (secondary to immune deficiencies) and eustachian tube dysfunction (secondary to embryonic malformations of the first and second branchial arches) are discussed as possible etiologic factors in the presence of the recurrent serous otitis media. An alcohol-induced neuroectoderm syndrome and alcohol ototoxicity are discussed as possible etiologic factors in the occurrence of sensorineural hearing loss. The findings suggest that hearing disorders are a heretofore unrecognized characteristic of the fetal alcohol syndrome. Such hearing disorders may contribute to the speech and language and learning difficulties seen in children with fetal alcohol syndrome. The present study also provided confirmatory evidence of visual, health, and speech and language disorders in children with this syndrome.