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Results for “Aphasia, Conduction”

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PubMed1990

[Conduction aphasia. Neuropsychologic study of a patient].

We report a patient with clinical features consistent with conduction aphasia. The patient had dilated cardiomyopathy, and as a complication, had cerebral infarction one year before neuropsychiatric evaluation. His language was fluid, paraphasic, with a "phonetic approximation behavior" in an attempt to pronounce the adequate word. Comprenssion was normal and repetition difficult, with numerous paraphasias. Number repetition was particularly affected. Reading and writing had the same features as spontaneous language. In association with the language disorder, he had "aphasic" acalculia, but other signs of left parietal involvement were absent. Magnetic resonance images showed a low parietal small chronic ischemic area, which also involved the left infrasilvian region, both in the cortex and in the white matter, presumably in the arcuate bundle. The differential diagnostic difficulties of this condition are discussed from a behavioral standpoint.

Aphasia↗
PubMed1987

Deep left parietal lobe syndrome: conduction aphasia and other neurobehavioural disorders due to a small subcortical lesion.

A patient with sudden onset of conduction aphasia in the context of an ischaemic stroke is reported. Other neurological and neuropsychological findings included bilateral ideomotor apraxia, right hemisensory defect and paradoxical left ear extinction on a dichotic listening test. Lesion location, as inferred from magnetic resonance imaging, involved a restricted subcortical area in the left parietal lobe, near the lateral wall of the cerebral ventricle. The anatomical correlate for each of the clinical findings is discussed in the light of classical anatomo-clinical correlations. It is concluded that this tetrad constitutes a specific syndrome which may be easily recognised and ascribed to a single lesion in the deep white matter of the left parietal lobe.

Aged↗
PubMed1977

[Nature of repetition disorders in conduction aphasia].

The skeptic over the existence of this important clinical entity, are now very few. Anatomo-clinical studies and the analysis of altered cognitive procedures suggest, however, that we are dealing with a heterogeneous group. Two models of conduction apahasias are presented, in which the mechanism of the altered repetition, as well as the probable localization of the lesion, differ considerably. One has a lesion in the posterior parietal region of the left hemisphere and it exemplifies an acoustic verbal memory deficit (short term memory). The other points at a functional disconnection between the decodification and verbal processes and those of expression. In this case, we are dealing with a left temporal lesion in a left-handed individual. The analysis of our material and of the clinical literature upholds the idea of a plurality of the altered mechanisms in the verbal repetition and suggest that the conduction aphasia is a syndrome.

Adolescent↗
PubMed2005

Dysgraphia in two forms of conduction aphasia.

Recent clinical observations, in the absence of experimental data, appear to suggest that written expression in conduction aphasics parallels their speech (Goodglass, 1992). The current study undertakes an analysis of word level writing in two conduction aphasics, and attempts to explore the posited 'parallel' relationship between speech production deficits and deficits in written expression. JL, a 66-year-old female with left posterior parietal lobe lesion and PP, a 65-year-old female with a left posterior temporo-parietal lobe lesion served as subjects of this study. Their response patterns on Boston Naming Test (BNT) and written naming task (John Hopkins Dysgraphia Battery) were utilized to verify the parallel hypothesis. Although both cases have exhibited phonological and semantic paraphasias on BNT, PP's overall performance was far superior to that of JL. JL produced numerous multiple responses to stimuli compared to PP's occasional multiple responses. PP's performance on the written naming task was far inferior to that of JL. JL's predominant error pattern in writing was the production of phonologically similar words to the target words. This paper argues that such seemingly contradictory, unpredicted patterns can be parsimoniously better explained, not by the parallel hypothesis but by current cognitive-neuropsychological models of writing.

Aged↗
PubMed1988

Reeducation strategies in conduction aphasia.

This article proposes a reeducation program for conduction aphasics with reproductive difficulties. The characteristics of the program are analysis and manipulation of visual stimuli (written words and syllables); suppression of the compensation effect of the spared lexical-semantic system; and progressive increase in length and complexity of phonological and syntactic construction of words and sentences. The aim of this program is to teach patients to control phonemic production. Results of the reeducation of three conduction aphasics are presented.

Aphasia↗
PubMed1988

Comprehension of sentence structure in anomic and conduction aphasia.

Picture-pointing auditory and reading comprehension tests were administered to anomic and conduction aphasics. Subjects responded to active sentences of the present progressive form. The possible errors which a subject could make on these experimental tasks included failure to correctly interpret noun order, number, or lexical meaning. Both groups made significantly more correct responses than error responses. Of their error responses, noun-order errors significantly exceeded number and lexical errors for which no differences were observed. When compared with results previously obtained for agrammatic Broca's aphasics, no differences in the pattern of errors were identified. These results are discussed relative to current theories of syntactic processing and for the mechanisms which account for these syntactic comprehension deficits following aphasia.

Adolescent↗