[Progressive septic granulomatosis (PSG)--a new clinical syndrome in pediatrics].
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Anemia↗
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Results for “Phagocyte Bactericidal Dysfunction”
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Chronic granulomatous disease: correlation between pathogenesis and clinical findings.
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Autopsy↗
[The nitroblue tetrazolium test in the newborn].
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Humans↗
[Lung disease in progressive septic granulomatosis in children].
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Chronic Disease↗
[Septic granulomatosis, a functional disorder of segmentated leukocytes].
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Child, Preschool↗
[The importance of tests using nitroblue tetrazolium].
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Humans↗
[Progressive septic granulomatosis (chronic granulomatous disease) in infancy].
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Antibodies↗
Studies on the pathogenesis of chronic granulomatous disease. 2. Impaired activation of glucose-6-phosphate dehydrogenase in leukocytes of a patient with chronic granulomatous disease during phagocytosis.
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Adult↗
[Changes in oral mucosa in general diseases. 3. (conclusion)].
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Endocrine System Diseases↗
Chorioretinal lesions in familial chronic granulomatous disease of childhood.
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Adolescent↗
Instrinsic defect of the polymorphonuclear leucocyte resulting in impaired chemotaxis and phagocytosis.
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Agammaglobulinemia↗
Leprosy--a model for the understanding of chronic granulomatous disease.
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Chronic Disease↗
[Assessment of the metabolic changes of granulocytes during phagocytosis (author's transl)].
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Granulocytes↗
[The lysosome in pathology].
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Age Factors↗
[Isolated chronic granulomatous gastritis].
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Chronic Disease↗
Editorial: Microbial killing by neutrophils.
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Cell Membrane↗
[The biochemical and clinical aspects of granulocyte functions in the process of phagocytosis (author's transl)].
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Adenosine Diphosphate↗
[Membranes properties of granulocytes in Job's-Syndrome with E. coli-septicemia (author's transl)].
Since the age of nine weeks a red haired girl suffered from purulent dermatitis and recurrent, systemic E. coli infections. She had an excessive hyperimmunoglobulinemia E, as well as impaired granulocyte adherence and chemotaxis. Though a sepsis was evident, the granulocytes exhibited a random FITC-Concanavalin A fluorescence. In spite of intensive treatment with various antibiotics and several granulocyte transfusions the child died at the age of 2 years and 11 months. As shown by the FITC-Concanavalin A distribution, the hyperimmunoglobulinemia E may have caused a decreased membrane fluidity causing the impaired adherence and chemotaxis. This could explain the pathophysiology of the Job's Syndrome.
Cell Membrane↗