[Role of chlormadinone in the oxidation-reduction processes in the rat retina].
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The light-induced changes in the kinetics of the oxidation of photosystem I reaction center P700 in leaves of C3 and C4 plants, depending on the time of leaf adaptation to dark before the illumination were studied. The EPR 1 signal from oxidized centers P+700 was used to monitor the kinetics of P700 redox transients. Similar dependences of the light-induced changes in the kinetics of P700 versus the adaptation time were observed in leaves of three kinds of plants: bean, orange and maize. These data suggest that the deactivation of the Calvin cycle enzymes, which occurs 1-3 min after ceasing the illumination, is the main factor that causes the retardation of the light-induced oxidation of P700.
Prolonged contacts with the hazardous substances in phosphorus processing caused noticeable changes in the oxidation-restoration system and bioenergetics. Therapeutic and preventive hyperbaric oxidation procedures improved the processes of bioenergetic supply in the body, which was caused by more active oxidation processes and decreased tissue hypoxia.
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