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PubMed1978

[Frostbite].

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Frostbite↗
PubMed1989

Treatment of frostbites of the upper extremities with prolonged blockade of axillary plexus.

Encouraged by the beneficial effect in the treatment of cold injury of the lower extremities, the authors applied permanent plexus anesthesia for treatment of the cold injury of the upper extremities too. Of the total of 10 in-patients treated with this method, amputation had to be made only in 2 cases due to the lack of compliance of the patients retarding the commencement of therapy. 7 patients with the same severity of cold injury admitted from other hospitals where they had been treated with traditional methods have undergone multiple phalangectomies. The method had beneficial effect only when applied within the first 8 h after trauma.

Anesthesia, Epidural↗
PubMed1989

[Pathophysiology, diagnosis and therapy of localized frostbite].

Pathophysiology, clinical feature, diagnosis and partially adverse recommendations for therapy are presented. Widening of diagnostic procedures for valuation of the late results by introduction of DSA, fluorescence-video-microscopy and digit phlethysmography are emphasized.

Combined Modality Therapy↗
PubMed1989

[Hemostatic changes after local exposure to low temperatures (frostbite)].

Hemostatic blood potential was studied in experiments on 400 rats, 25 dogs and in 120 patients, during changes of CNS reactivity after intraventricular injection of reserpine, adrenaline, and irritation of the thalamus ventromedial nucleus (VMN) or its destruction. CNS reactivity in dogs changed after intraarterial injections of acetylcholine, reserpine, heparin and fibrinolysin solutions. It was shown that activation of adrenergic structures of the hypothalamus by intraventricular adrenaline injections, or the hypothalamus VMN irritation, stimulated coagulation and depressed anticoagulation mechanisms of the blood, and increased hemostatic blood potential, that led to rejection of frostbitten tissues. Activation of the hypothalamus cholinergic structures by intraventricular injections of reserpine, destruction of the hypothalamus VMN, and intraarterial injections of acetylcholine, reserpine, heparin, fibrilysin and contrycal mixtures stimulated the blood anticoagulation mechanisms, decreased the hemostatic potential, led to retention of cryodestructed tissues.

Animals↗