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At least 505 records · Page 28Linked to original sources
PubMed1997

Influence of vanadium on acclimatization of humans to high altitude.

The study was conducted on human volunteers as controls as well as after administration of vanadyl sulphate on induction to high altitude (HA) at 3500 m. The plasma vanadium contents were significantly reduced in the control group on abrupt induction to HA on days 3 and 10, indicating redistribution to other organs/tissues under the stressful situation. In the vanadium salt-treated group, plasma vanadium contents were similar to those obtained at sea-level. Administration of vanadyl sulphate did not act as a diuretic. Moreover the vanadium supplemented group drank more water and also excrete less urine than the control group.

Acclimatization↗
PubMed1991

Endogenous opiates and ventilatory acclimatization to chronic hypoxia in the cat.

The effects of the opiate antagonist naloxone (0.4 mg.kg-1, i.v.) on carotid chemoreceptor and ventilatory responses to graded steady-state levels of hypoxia and hypercapnia were investigated in two groups of cats: chronically normoxic and chronically hypoxic. The cats of the latter group were exposed to PIO2 of about 70 mm Hg at sea level for 3-4 weeks and showed an attenuated response to hypoxia. All cats were tested under alpha-chloralose anesthesia. Naloxone treatment did not increase appreciably carotid chemoreceptor activity or its responses to hypoxia and hypercapnia in either cat group. Naloxone caused a small ventilatory stimulation in the chronically hypoxic cats, so that the attenuated response to hypoxia was not relieved. By contrast, the chemoreflex ventilatory response to hypoxia was stimulated by naloxone in the chronically normoxic cats. The findings that the depressed ventilatory chemoreflexes in the chronically hypoxic cat were not ameliorated by the opiate antagonist indicate that an increased elaboration of endogenous opiates does not underlie ventilatory adaptation to chronic hypoxia.

Acclimatization↗
PubMed1988

Acclimatization of roach, Rutilus rutilus (L.), to toxic components of kraft pulp mill effluents.

Roach (Rutilus rutilus) were exposed, along with their appropriate controls, to a simulated bleached kraft mill effluent (KME-Sa + CP) first for 38 days at a concentration of 0.035 X LC50 and then for 14 days at 0.07 X LC50. During the experiment, their tolerances to KME-Sa + CP were tested five times by measuring the 48-hr LC50 values. In addition, the growth of roach was monitored. At the end of the exposure, accumulation of [14C]pentachlorophenol in various parts of the fish (total PCP in water 15.6 micrograms/liter) was measured. When the fish were preexposed to KME-Sa + CP, the acute tolerance of this mixture in roach increased by 30-39%, but the response was abolished in 31 days. Fish growth remained unchanged during the experiment. Measurement of PCP accumulation revealed no difference in the absorption rate, but under steady-state conditions the degree of bioconcentration was 16% lower (P less than 0.02) in preexposed roach than in their unexposed controls. This difference was entirely accounted for in the head and visceral parts of the fish. Even when no final changes were noted in tolerance and growth rate of the fish, the authors suggest that the significantly lowered body burden implied acclimatizatory compensation under subchronic exposure of this xenobiotic.

Acclimatization↗