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At least 469 records · Page 26Linked to original sources
PubMed1997

Posttraumatic anosmia in craniofacial trauma.

Although the clinical implications of anosmia can be significant, posttraumatic anosmia is generally given relatively little attention in the clinical setting. Patients who sustain craniofacial trauma are most at risk. The incidence of posttraumatic anosmia varies according to the severity of injury and has an overall estimated incidence of 7%. Factors that increase the risk of developing anosmia include anterior skull base fractures, bilateral subfrontal lobe injury, dural lacerations, and cerebrospinal fluid leakage. Recovery of function has been estimated to be approximately 10%. Time of recovery, if it occurs, varies between 8 weeks and 2 years. Presented herein are the clinical, radiographic, pathophysiologic, and anatomic substrata of posttraumatic anosmia.

Adolescent↗
PubMed1987

Plant poisoning and oto-rhino-laryngology. Poisonous plants, growing or used in Europe and the United States.

Poisoning often affects the ear, nose, mouth and throat. Sialorrhea, stomatitis, oropharyngeal dryness, a burning sensation in the mouth, rhinitis and nasal septal perforation, hearing loss and vestibular disturbances can be the result of intoxications by poisonous plants, growing or used in Europe and the United States. This article has not the intention to be exhaustive, but to give a synopsis of this topic.

Hearing Disorders↗
PubMedDate not supplied

Drug-related gustatory disorders.

A review is presented of the drug-induced gustatory disorders that have so far been identified. Metallic, bitter or salty phantogeusias and hypogeusias, often of a dissociated type, which may even develop into ageusia have been described as side effects of treatment with some drugs. Personal observations on six cases are reported in which the gustatory disorders are attributed to the ingestion of phenylbutazone, carbamazepine, chlormezanone and baclofen. After interruption of the treatments these disorders were reversible within weeks to months.

Adult↗
PubMedDate not supplied

CT of the brain in taste and smell dysfunction.

Three hundred fifty-four patients with taste and/or smell disorders were evaluated with computed tomography (CT). The largest group was characterized by head trauma (27%), followed by idiopathic causes (26%), postinfluenza-like hyposmia and hypogeusia (15%), and congenital etiologies (14%). Hyposmia and hypogeusia occurred concomitantly in 21%-45%, the percentage varying according to etiologic subgroup. CT abnormalities were found in 108 (31%) of the 354 patients. The most frequent pathologies were frontal encephalomalacia, subfrontal atrophy in the region of the olfactory bulbs, and anterior temporal lobe atrophy. These changes were found alone or in tandem. Some CT findings suggest common cerebral taste and smell centers and common neural pathways and association centers.

Ageusia↗