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Repression and self-presentation: when audiences interfere with self-deceptive strategies.

To defend against threatening feedback, one may avoid and ignore it, or one may dwell on it and think of refutations. Repressors who received threatening feedback privately spent the least amount of time reading it, whereas repressors who received the same feedback publicly spent a long time reading it. Thus, the audience prevented repressors from ignoring threatening feedback; instead, they thought and worried about the partner's (bad) impression of them. Nonrepressors were unaffected by the favorability of the evaluation or the public nature of the situation. Repressors showed superior recall for the few bits of threatening information embedded in a generally favorable evaluation, suggesting that they are especially sensitive when their defenses are down.

Adult↗

Pathogenic trickery: deception of host cell processes.

Microbial pathogens cause a spectrum of diseases in humans. Although the disease mechanisms vary considerably, most pathogens have developed virulence factors that interact with host molecules, often usurping normal cellular processes, including cytoskeletal dynamics and vesicle targeting. These virulence factors often mimic host molecules, and mediate events as diverse as bacterial invasion, antiphagocytosis, and intracellular parastism.

Animals↗

Seeing through the face of deception.

We have developed a high-definition thermal-imaging technique that can detect attempted deceit by recording the thermal patterns from people's faces. This technique has an accuracy comparable to that of polygraph examination by experts and has potential for application in remote and rapid security screening, without the need for skilled staff or physical contact.

Body Temperature↗

Masters of deception: a review of herpesvirus immune evasion strategies.

Herpesviruses have acquired a variety of different mechanisms to avoid the damaging effects of host immunity. Frequently, these viruses subvert normal immune regulatory functions utilized by the host. The focus of this review is upon herpesvirus genes encoding known or potential immunomodulatory proteins. Areas covered include inhibition of complement and antibody function, herpesvirus-encoded homologues of cytokines and chemokine receptors, and potential disruption of cellular recognition of virally infected targets.

Amino Acid Sequence↗