Search PubMed⌕ Search

SEARCH · Search PubMed

Results for “DECOMPRESSION SICKNESS”

Search indexed PubMed citations on genomics, clinical trials, systematic reviews and public health. Explore titles, authors and supplied subject terms, then open the PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 415 records · Page 23Linked to original sources

Inner ear decompression sickness and mal de debarquement.

OBJECTIVE: To present a case series of vestibular symptoms appearing after combined sailing and diving activity, and to discuss the differential diagnosis and the workup algorithm. STUDY DESIGN: Case series. SETTING: Tertiary referral center. PATIENTS: Three patients aged 25 to 31 years suffering from unsteadiness and movement sensations after sailing and scuba diving. INTERVENTIONS: Neurotologic evaluation and recompression therapy in a hyperbaric chamber. MAIN OUTCOME MEASURES: The increasing popularity of marine sports and leisure activities has resulted in the exposure of a growing number of people to unique abnormalities not encountered under terrestrial conditions. The otolaryngologist who is involved in the care of these patients is required to diagnose and treat diving-related sinus and ear injuries such as barotrauma and decompression sickness, and also to be familiar with sailing-related disorientation syndromes such as seasickness and mal de debarquement. Treatment modalities for the various abnormalities differ significantly, and early commencement of treatment is often crucial for a successful outcome. CONCLUSION: Whenever doubt exists, recompression treatment must be instituted as soon as possible because of the potential for severe sequelae if the patient is left untreated, and because the risks involved in this therapy are minimal.

Adult↗

Latency in onset of decompression sickness on direct ascent from air saturation.

Twenty-four human subjects were exposed to compressed air at simulated depths ranging from 45 ft seawater gauge (fsw) (2.36 ATA) to 75 fsw (3.27 ATA) for periods of time sufficient to allow saturation of most tissues with inert gas. From each of four depths (45, 55, 65 and 75 fsw), subjects ascended directly to 1 ATA, where they remained for 30, 17, 13, and 10 min, respectively. During this time the onset of Doppler detected venous gas emboli (VGE), pruritus, and any other symptomatology was monitored and recorded. In the ascending excursion from 45 fsw (n = 18), the mean appearance times of pruritus and VGE were 19.3 +/- 7.5 (mean +/- SD) and 21.9 +/- 7.4 min, respectively, with one case of pain-only decompression sickness (DCS). The excursion from 55 fsw (n = 17) shortened these times to 9.2 +/- 3.3 and 14.4 +/- 3.2 min, respectively, with two cases of DCS, one each of pain-only and serious types. In the excursion from 65 fsw (n = 23), mean pruritus and VGE appearance times were 6.3 +/- 1.4 and 10.8 +/- 1.9 min, respectively, with no cases of DCS. The excursion from 75 fsw (n = 6) resulted in appearance times of 4.8 +/- 0.8 and 8.2 +/- 1.2, respectively, with one case of pain-only DCS. Sufficient cases of DCS did not occur in any of the excursions to allow determination of mean appearance times. The relationship between pressure reduction and appearance times of pruritus and VGE, and the threshold time of DCS, can be described mathematically. We conclude that latency in the development of DCS on direct ascent from air saturation exists and is of sufficient magnitude to permit practical application in emergency decompressions or unpressurized transfers between compressed air or N2-O2 environments.

Adult↗