Search PubMed⌕ Search

SEARCH · Search PubMed

Results for “TREMATODE INFECTIONS”

Search indexed PubMed citations on genomics, clinical trials, systematic reviews and public health. Explore titles, authors and supplied subject terms, then open the PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 379 records · Page 21Linked to original sources
PubMed1995

[Karyometric analysis of selected neurohormonal cells of the snail Lymnaea stagnalis (L.) under conditions of natural infection with digenetic trematode parthenites].

To supply more documentary evidence for the effect of parasitic infection on activity of neurosecretory centres in the cerebral ganglia of the snail host, caudo-dorsal and light green cells of adult individuals of Lymnaea stagnalis naturally infected with digenean parthenites were subjected to karyometric analysis. In infected animals significant enlargement of nuclear volumes of both examined types of neurosecretory cells was ascertained. Mean nuclear volume of the light green cells in snails infected with Furocercariae was significantly lower in comparison with the individuals infected with parthenites from groups Echinostomata and Xiphidiocercariae. In remaining cases differences in nuclear volumes among subpopulations distinguished on the ground of an infection factor were not statistically significant.

Animals↗
PubMed1986

Enzyme activity in livers from chickens (Gallus domesticus) experimentally infected with the digenetic trematode, Amphimerus elongatus (Trematoda: Opisthorchiidae).

Day old Leghorn chickens were infected with the digenetic trematode Amphimerus elongatus. After different periods of growth, birds were sacrificed and liver enzyme activity determined for six enzymes. Specific activities of adenylate kinase, lactate dehydrogenase, malate dehydrogenase and phosphoglucomutase were significantly lower in infected chickens than in controls. Creatine kinase and glucosephosphate isomerase activities were unaffected by the parasite.

Adenylate Kinase↗
PubMed1997

Removal of the submaxillary salivary glands and infection with the trematode Schistosoma mansoni alters exploratory behavior and pain thresholds in female mice.

In this study, CD-1 female mice, deprived of the submaxillary salivary glands, were infected with S. mansoni and their behavior was observed 15 weeks after infection, when the eggs of the parasite are present in the brain. Sialectomized infected mice showed changes in exploratory activity, sniffing, and wall-rearing in the open-field and in the black/white box, but no differences in pain sensitivity were observed on the hot plate. The present results suggest that the modifications in the behavior of sialectomized infected mice might be associated with the inability of the animals to cope with the aversive effects of the infection and, most probably, with modifications in the levels of polypeptides released into the bloodstream by the salivary glands, affecting the NGF-responsive cells of the nervous, endocrine, and immune systems.

Animals↗
PubMed1990

Comparative glucose tolerance studies in the freshwater snail Biomphalaria glabrata: influence of starvation and infection with the trematode Schistosoma mansoni.

The dynamic reactions of B. glabrata on intravasal loads of 20-1000 micrograms glucose.g live fresh wt-1 were studied in standard fed snails (SFS). Starved snails (SS) and snails infected with S. mansoni (IS) were given 100-500 micrograms glucose.g fresh live wt-1. Mean hemolymph glucose level was 12.1 mg.100 ml-1 in SFS. It was not significantly lower in SS (10.7 mg.100 ml-1), but significantly reduced in IS (8.5 mg.100 ml-1). Since hemolymph volumes were significantly increased in SS, the amount of circulating glucose (pool) did not change (75 micrograms.g body weight-1) compared to SFS (62 micrograms.g-1). It was, however, reduced to 41 micrograms.g-1 in IS. In SFS the circulating glucose pool had to be doubled to induce significantly elimination of the injected glucose. Tripled pools were eliminated with half-times of 45 min, whereas lower and higher glucose loads were eliminated significantly slower (half-times: 80-105 min). Glucose tolerance of SS was reduced: half-times were doubled, and metabolization of injected glucose was reduced. Since tissue fresh weights were lowered by 40%, absolute incorporation of 14C from labeled glucose was lowered, but specific incorporation (per mg) was higher than in SFS and IS. Glucose tolerance of IS was increased: metabolic clearance rates rose by 70% and half-times were shortened by 30%, though absolute and specific rates of 14C incorporation were lowered. However, IS lost 25% of the label to the water, whereas SFS lost 12% and SS only lost 8%. Using the antimetabolite 2-deoxyglucose, 80% of the losses proved to be glucose in IS, and 50% in SFS. The present results suggest the existence of a glucostatic regulation in B. glabrata with lower sensitivity and capacity than in mammals. As to glucose tolerance, the often reported parallelism in metabolic shifts induced by starvation and parasitic infection was not confirmed.

Animals↗