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PubMed1979

Comparison of skin tests and RAST for the diagnosis of bee sting allergy.

Skin tests and estimation of specific IgE-antibodies by the RAST are evaluated as diagnostic procedures in bee sting hypersensitivity with the followiny extracts. (2) With bee venom results of skin tests and RAST correspond in 82%, with bee wholebody extracts in 68%. (3) Both with skin tests and the RAST with bee venom a good discrimination between patients with bee sting hypersensitivity and non-allergic controls is observed. Skin tests with bee venom are somewhat more sensitive than bee venom-RAST. (4) With the careful skin test procedure chosen, no adverse reactions were observed.

Allergens↗
PubMed1995

Clinical symptoms and immunologic reactivity to bee and wasp stings in beekeepers.

We compared the history of sting reactions with venom skin prick test (SPT) and CAP RAST reactions in beekeepers in order to assess the value of structured questions and symptom backgrounds. The study population consisted of 102 beekeepers, who were 25-75 years of age. Bee and wasp venom SPT was performed with concentrations of 10, 100, and 300 micrograms/ml. The CAP Phadiatop test was used in the screening of IgE antibodies against common inhalant allergens. Forty-two beekeepers had never experienced large local or systemic reactions after a bee sting. Of the 31 subjects with a history of systemic reactions, 13 had experienced these during the previous year. A significant difference (P < 0.01) between systemic reactors and nonreactors was found in bee venom CAP and SPT (300 micrograms/ml). However, due to considerable overlap, these tests are unable to discriminate between allergic and nonallergic beekeepers. Both bee venom SPT (300 micrograms/ml) and CAP tests were positive in 65% of systemic reactors and in 34% of nonreactors (P = 0.008). Venom SPT (300 micrograms/ml) correlated significantly with CAP for both venoms. No correlation was observed between venom allergy and atopy. Clinically, the most practical concentration for evaluating bee and wasp venom sensitization by SPT proved to be 300 micrograms/ml.

Adult↗
PubMed1984

Reversible parasympathetic dysautonomia following stinging attributed to the box jelly fish (Chironex fleckeri).

Following a box jelly fish sting, a 52 year old Chinese fisherman developed acute abdominal distension, inability to pass urine and failure of erection. Examination revealed gaseous abdominal distension and a distended urinary bladder. Absence of lachrimation and absence of changes in the R-R interval in the ECG during breathing and carotid sinus massage gave further evidence of parasympathetic dysautonomia. The patient made a complete recovery. The case highlights the occurrence of reversible parasympathetic dysautonomia following box jelly fish sting.

Autonomic Nervous System Diseases↗
PubMed1992

Imported fire ant stings: clinical manifestations and treatment.

Stings by imported fire ants result in local reactions to the insect's venom; less frequently, they induce systemic allergic reactions. A 19-month-old child developed a characteristic localized cutaneous reaction at the sites of numerous stings after disturbing an ant mound. He experienced no secondary complications, and his pustular lesions resolved with conservative treatment.

Animals↗
PubMed1992

Case report. Bee sting brachial block.

A case of brachial plexus block is presented, following a bee sting in the posterior triangle of the neck. The onset of neurological symptoms was rapid as was their subsequent resolution. Delayed peripheral neurological symptoms believed to have an immunological basis have been reported in response to stings from bees and other Hymenoptera both in the central and peripheral nervous systems (Goldstein et al., 1960; Means et al., 1973; Bachman et al., 1982; Weatherall et al., 1987; Van Antwerpen et al., 1988), but to the authors' knowledge no similar case of immediate peripheral block has been reported.

Adult↗
PubMed1995

Bilateral pallidostriatal necrosis caused by a wasp sting: a clinical and pathological study.

A previously healthy man developed an acute encephalopathy with coma after a single wasp sting on his chin. Brain CT showed bilateral pallidostriatal radio-lucencies. He died 72 hours after the sting with no evidence of primary cardiorespiratory failure or allergic reaction. Pathological findings were bilateral pallidostriatal necrosis and diffuse neuronal damage in the frontal, temporal, and parietal cortex. The neurotoxic effect of the poison, together with a hypersensitivity are the most likely explanations for this unusual encephalopathy.

Aged↗
PubMed1991

A "scorpion fish" (Trachinus vipera) sting: fishermen's hazard.

"Scorpion fish" is a nickname given by fishermen to members of the Trachinidae family as a result of their unusual stinging mechanism. These fish are found throughout the eastern Atlantic region from the North Sea through the Mediterranean and Black Seas and along the western coast of Northern and Central Africa. They are characterised by poisonous glands located at the base and sides of the spines of their anterior back fin and at the base of a spine located on the gill cover. Because of the unusual location of the glands, fishermen handling these fish frequently suffer local injuries. A case of necrosis of the tip of the middle finger after a "scorpion fish" sting is described.

Accidents, Occupational↗
PubMed1990

Localized neuropathy following jellyfish sting.

This report describes the case of an 18 year old female who sustained a jellyfish sting on her right wrist. She subsequently developed complete radial, ulnar and median nerve palsies distal to the site of the sting, which recovered fully over the next 10 months. We believe this to be due to a direct neurotoxic effect of the jellyfish venom.

Adolescent↗
PubMed1988

Acute renal failure following multiple hornet stings.

Five patients who developed acute renal failure due to acute tubular necrosis following multiple hornet (Vespa orientalis) stings are described. All of them had intravascular hemolysis. Evidence for rhabdomyolysis was present in 2 patients. Two patients had elevated transaminase and alkaline phosphatase levels and in 1 of these, liver biopsy showed centrilobular necrosis. Two patients had thrombocytopenia in the absence of disseminated intravascular coagulation. Two patients died of infections while the remaining 3 recovered completely. Acute renal failure following multiple hornet stings appears to result mainly from intravascular hemolysis or rhabdomyolysis although a direct nephrotoxic effect of venom cannot be excluded.

Acute Kidney Injury↗
PubMed1985

Insect sting allergy in children: what is the real cost of the disease?

The impact of insect sting allergies on the quality of life of 118 children and their parents is assessed using attitudinal and psychometric questionnaires. Children, ranging in age from 7-15 years, manifested more anxiety in the clinical setting (state anxiety) than usual (trait anxiety), whereas for parents the trend was reversed. Most children believed that they could control being stung, and restrictions imposed by two-thirds of the parents assisted in preventing stinging episodes. Parents perceived their child's academic achievement, social abilities and extracurricular involvement as superior to that of their peers and closest aged siblings.

Adolescent↗
PubMed1985

Scorpion sting in children. A review of 51 cases.

Scorpion sting in children is a hazardous and potentially lethal condition. Fifty-one infants and children were admitted to the Pediatric Departments at the Hadassah-Hebrew University Hospitals in Jerusalem, during a 5-year period, following scorpion sting. Fifteen (29.4%) had severe systemic signs of envenomation and two (3.9%) died. Analysis of our data showed that patients with severe toxicity were brought to the hospital after a significantly longer time lapse than were the patients with mild-to-moderate symptoms. The current management of children with scorpion envenomation consists of administration of specific antivenom and close surveillance in an intensive care unit, where vital signs and continuous cardiac monitoring enable early initiation of therapy for life-threatening complications, such as cardiac and respiratory failure, convulsions, or hypertension.

Antivenins↗
PubMed2004

Transient claw hand owing to a bee sting. A report of two cases.

We describe two patients with claw hand as a result of a bee sting. It is likely that this was caused by the apamin in the sting which has an effect on the upper limb, at the spinal cord and on the peripheral nerves. It is important to recognise that the claw hand is not owing to compartment syndrome. Both patients were treated conservatively with full resolution within 48 hours, without any lasting effects.

Adult↗
PubMed1980

Oral inflammatory response to a sting from a Portuguese man-of-war.

A careful history was taken in an attempt to document an unusual delayed inflammatory reaction involving the ears and oral cavity. Severe oral and auricular inflammations have been described as apparent reactions to stings on the patient's foot by a Portuguese man-of-war (P physalia). Toxin was injected into the right foot and resulted in a delayed type of severe swellings and inflammation. The soft palate, cheek, periodontal tissues, and lower lip were primarily involved. Oral inflammations coincided with massive, acute, inflammatory lesions behind the ears. Delayed oral and ear inflammations suggest an unusual reaction to the toxin of the sting of P physalia; it was possibly hypersensitive in nature. The described pathology seems unique to medical reports concerning the Portuguese man-of-war. The foot responded rapidly to topical application of ammonia solution. Antibiotic therapy taken orally apparently helped resolve the oral and ear lesions in approximately 11 days.

Bites and Stings↗
PubMedDate not supplied

Acute renal failure after massive honeybee stings.

Two clinical cases of patients who survived after numerous attacks of Africanized bees (600 and 1500 bee stings, respectively) are reported. Clinical manifestation was characterized by diffuse and widespread edema, a burning sensation in the skin, headache, weakness, dizziness, generalized paresthesia, somnolence and hypotension. Acute renal failure developed and was attributed to hypotension, intravascular hemolysis, myoglobinuria due to rhabdomyolysis and probably to direct toxic effect of the massive quantity of injected venom. They were treated with antihistaminic, corticosteroids and fluid infusion. One of them had severe acute renal failure and dialysis was required. No clinical complication was observed during hospital stay and complete renal function recovery was observed in both patients. In conclusion, acute renal failure after bee stings is probably due to pigment nephropathy associated with hypovolemia. Early recognition of this syndrome is crucial to the successful management of these patients.

Acute Kidney Injury↗
PubMedDate not supplied

[Recording of venomous bites and stings by National Information Systems in Brazil].

This paper highlights the epidemiological surveillance of venomous bites and stings according to four national information systems: SINAN (National Databank of Major Causes of Morbidity), SINITOX (National Information System on Poisoning), SIH-SUS (Hospital Information System of the Unified Health System), and SIM (Mortality Information System). The authors conclude that each information system has specific characteristics and addresses different demands. Although they contain large amounts of data, even if combined they fail to reflect the real magnitude of disorders caused by venomous bites and stings in the country.

Animals↗
PubMedDate not supplied

Reactions to imported fire ant stings.

Reactions to the sting of the imported fire ant are an increasing problem for residents of the Gulf South. Three recent evolutionary changes suggest that the habitat of the ants will continue to expand. This article gives a brief review of the problem, including unresolved questions related to management of sting reactions.

Allergens↗
PubMed2000

Optic neuritis after bee sting.

The purpose of this report is to document an unusual case of fully recovered vision after optic neuritis caused by bee sting. A 46-year-old man presented with sudden visual loss after being stung by a bee on the left conjunctiva. He developed optic disc swelling and there was a delay in the P100 wave of the pattern visual evoked potential (VEP). The patient received acute treatment, with intravenous methylprednisolone followed by oral prednisolone. Two days later, visual acuity in the left eye was recovered to 20/20 and P100 latency in pattern VEP was also normalized. Furthermore, visual field and color vision tests revealed no remaining abnormalities. This case suggests that early corticosteroid treatment is effective in optic neuritis caused by bee sting.

Animals↗
PubMedDate not supplied

Management of bee-sting anaphylaxis.

A retrospective case analysis of 101 adverse reactions to bee-stings and a prospective questionnaire analysis of the proposed management by local medical practitioners and resident hospital staff members of three hypothetical bee-sting reactions has revealed that understanding of the use of adrenaline in patients with reactions to bee envenomation is confused with regard to the indications for its use, dosage and route; that corticosteroid agents are used or are recommended too frequently, sometimes as the sole therapeutic agent; and that there is a lack of awareness of the need for volume replacement in hypotensive shocked patients. These conclusions highlight the urgent need for a greater understanding of the optimal forms of management for patients with acute anaphylactic reactions to bee envenomation.

Adrenal Cortex Hormones↗