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Results for “PULMONARY HEART DISEASE”

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At least 379 records · Page 21Linked to original sources
PubMed1991

[Diagnosis of right ventricle hypertrophy during pulmonary heart disease in patients with chronic bronchitis].

The comparison of ECG, VCG and ECHO data performed in 78 patients with chronic bronchitis allowed us to distinguish 4 grades of right ventricular hypertrophy (RVH): 1) absence of RVH (QRS loop occupies up to 70% of the area in horizontal plane, right ventricular wall thickness (RVWT) up to 5 mm, ECG changes); 2) slightly marked RVH (QRS-loop area 70-89%, RVWT 6-9 mm, electric axis deviation to the right on ECG, and P.100/R + S greater than 20 in lead II); 3) marked RVH (QRS-loop area 90-100%, RVWT 6-10 mm, right ventricular end-diastolic dimension 2-1-3.0 cm, on ECG--the above changes plus R/SV1 greater than 1.0, RV1 greater than or equal to 5 mm, RV1 greater than or equal to 7 mm, and P-pulmonale); 4) marked RVH with significant dilatation and repolarization abnormalities (VCG and ECHO data as above plus right ventricular end-diastolic dimension on ECG more than 3.0 cm, S-T depression, and negative T-wave in leads aVF and V1-3). Apparent ECG signs of RVH in chronic bronchitis develop much later, after the development of secondary pulmonary hypertension.

Adult↗
PubMed1996

[Massive pulmonary artery embolism complicated by acute pulmonary heart disease].

43-year old woman, with considerable overweight had been admitted to Intensive Medical Care Unit with suspicion of pulmonary embolism (PE). The patient had the limb immobilized in gypsum for last several weeks. This episode was tangled with recurrent thrombosis of deep veins in the left limb, treated with heparin and oral anticoagulants irregularly without sufficient control. Taking into consideration the data of anamnesis, clinical picture and the results of ECG, chest X-ray, gasometric and echocardiographic examination we got much closer to the recognition of PE. Our suspicion of PE was confirmed by the result of pulmonary angiography. Indications for thrombolytic treatment (r-tPA) had been established. During the following hours considerable improvement of general state was observed. The therapy was continued with constant drip infusion of heparin. No prolongation of therapeutic PTT was observed. The deficit of AT III was diagnosed. In this situation the patient was given AT III to obtain normalization of its level and therapeutic extension of PTT. Therefore there were settled indications for the operation of uterus with myoma changes. As the rich thrombolytic material in the leg's vein was found the patient was implanted LGM Filter, with excellent prophylactic effect (no PE in perioperative period). The clinical course of our case enabled to present most of diagnostic, therapeutic and preventive methods applied in venous thromboembolism.

Administration, Oral↗
PubMed1977

Clinical and electrocardiographic correlations in pulmonary heart disease (cor pulmonale).

It is now possible to diagnose early cor pulmonale by utilizing electrocardiographic fluctuations which are the result of moderate pulmonary hypertension and the accompanying effect upon right ventricular function. This holds whether the ECG alterations are produced by chronic obstructive pulmonary disease or by the rarer forms of obliterative lung disease. The late stage of cor pulmonale, in which right ventricular hypertrophy is manifest, should now be rarely encountered in view of our improved understanding of cor pulmonale and more rational therapeutic approaches to the underlying disorders.

Coronary Circulation↗
PubMed1995

[Chronic pulmonary heart disease: therapeutic indications].

Chronic thromboembolic cor pulmonale is a serious condition and its frequency seems to be on the increase. The authors report their personal experience of 100 cases and review the literature to describe the natural history, diagnostic strategy and therapeutic indications of this condition. The mechanisms of this aberrant evolution of acute pulmonary embolism are unknown. They key symptom is effort dyspnea without a ventilatory deficit. The most useful diagnostic investigations are Doppler echocardiography and perfusion pulmonary scintigraphy. The diagnosis is confirmed by pulmonary angiography which, with the aid of a thoracic CT scan or endovascular techniques (ultrasonography or angioscopy), allow assessment of the feasibility of thromboendarteriectomy. Medical treatment is based on long-term anticoagulation, in some cases of lower limb thrombosis associated with interruption of the inferior vena cava. Two possible curative surgical techniques are available in selected patients; thromboendarteriectomy and pulmonary transplantation, the relative indications of which are listed. Of all the causes of pulmonary hypertension, chronic thromboembolic cor pulmonary should be systematically looked for because curative surgical treatment is possible in some cases.

Anticoagulants↗